Target occupancy study and whole-body dosimetry with a MAGL PET ligand [

MAGL Non-human primate Occupancy PET Radiation dose

Journal

EJNMMI research
ISSN: 2191-219X
Titre abrégé: EJNMMI Res
Pays: Germany
ID NLM: 101560946

Informations de publication

Date de publication:
04 Mar 2022
Historique:
received: 13 07 2021
accepted: 25 01 2022
entrez: 4 3 2022
pubmed: 5 3 2022
medline: 5 3 2022
Statut: epublish

Résumé

Monoacylglycerol lipase (MAGL) is a key serine hydrolase which terminates endocannabinoid signaling and regulates arachidonic acid driven inflammatory responses within the central nervous system. To develop [ Seven cynomolgus monkeys were enrolled for brain PET measurements. Two PET measurements along with arterial blood sampling were performed in each NHP: one baseline and one pretreatment condition with intravenous administration of PF-06818883, a pro-drug of a selective MAGL inhibitor (total of seven doses between 0.01 and 1.27 mg/kg). Kinetic parameters K Radioactivity retention was decreased in all brain regions following pretreatment with PF-06818883. Occupancy was measured as 25.4-100.5% in a dose dependent manner. Whole-body PET showed high radioactivity uptake values in the liver, small intestine, kidney, and brain. The effective dose of [ [

Sections du résumé

BACKGROUND BACKGROUND
Monoacylglycerol lipase (MAGL) is a key serine hydrolase which terminates endocannabinoid signaling and regulates arachidonic acid driven inflammatory responses within the central nervous system. To develop [
METHODS METHODS
Seven cynomolgus monkeys were enrolled for brain PET measurements. Two PET measurements along with arterial blood sampling were performed in each NHP: one baseline and one pretreatment condition with intravenous administration of PF-06818883, a pro-drug of a selective MAGL inhibitor (total of seven doses between 0.01 and 1.27 mg/kg). Kinetic parameters K
RESULTS RESULTS
Radioactivity retention was decreased in all brain regions following pretreatment with PF-06818883. Occupancy was measured as 25.4-100.5% in a dose dependent manner. Whole-body PET showed high radioactivity uptake values in the liver, small intestine, kidney, and brain. The effective dose of [
CONCLUSIONS CONCLUSIONS
[

Identifiants

pubmed: 35244788
doi: 10.1186/s13550-022-00882-2
pii: 10.1186/s13550-022-00882-2
pmc: PMC8897535
doi:

Types de publication

Journal Article

Langues

eng

Pagination

13

Informations de copyright

© 2022. The Author(s).

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Auteurs

Ryosuke Arakawa (R)

Centre for Psychiatry Research, Department of Clinical Neuroscience, Karolinska Institutet, Karolinska University Hospital Solna, R5:02, 17176, Stockholm, Sweden. ryosuke.arakawa@ki.se.
Stockholm Health Care Services, Stockholm County Council, Stockholm, Sweden. ryosuke.arakawa@ki.se.

Akihiro Takano (A)

Centre for Psychiatry Research, Department of Clinical Neuroscience, Karolinska Institutet, Karolinska University Hospital Solna, R5:02, 17176, Stockholm, Sweden.
Stockholm Health Care Services, Stockholm County Council, Stockholm, Sweden.

Sangram Nag (S)

Centre for Psychiatry Research, Department of Clinical Neuroscience, Karolinska Institutet, Karolinska University Hospital Solna, R5:02, 17176, Stockholm, Sweden.
Stockholm Health Care Services, Stockholm County Council, Stockholm, Sweden.

Zhisheng Jia (Z)

Centre for Psychiatry Research, Department of Clinical Neuroscience, Karolinska Institutet, Karolinska University Hospital Solna, R5:02, 17176, Stockholm, Sweden.
Stockholm Health Care Services, Stockholm County Council, Stockholm, Sweden.

Nahid Amini (N)

Centre for Psychiatry Research, Department of Clinical Neuroscience, Karolinska Institutet, Karolinska University Hospital Solna, R5:02, 17176, Stockholm, Sweden.
Stockholm Health Care Services, Stockholm County Council, Stockholm, Sweden.

Kevin P Maresca (KP)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Lei Zhang (L)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Edmund J Keliher (EJ)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Christopher R Butler (CR)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Justin R Piro (JR)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Tarek A Samad (TA)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Deborah Smith (D)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Deane Nason (D)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Steve O'Neil (S)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Patrick Trapa (P)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Kari R Fonseca (KR)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

John Litchfield (J)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Timothy McCarthy (T)

Worldwide Research and Development, Pfizer Inc., Cambridge, MA, USA.

Richard E Carson (RE)

Department of Radiology and Biomedical Imaging, Yale University School of Medicine, New Haven, CT, USA.

Christer Halldin (C)

Centre for Psychiatry Research, Department of Clinical Neuroscience, Karolinska Institutet, Karolinska University Hospital Solna, R5:02, 17176, Stockholm, Sweden.
Stockholm Health Care Services, Stockholm County Council, Stockholm, Sweden.

Classifications MeSH