Necroptosis-driving genes


Journal

Journal for immunotherapy of cancer
ISSN: 2051-1426
Titre abrégé: J Immunother Cancer
Pays: England
ID NLM: 101620585

Informations de publication

Date de publication:
03 2022
Historique:
accepted: 21 01 2022
entrez: 10 3 2022
pubmed: 11 3 2022
medline: 11 5 2022
Statut: ppublish

Résumé

Hepatocellular carcinoma (HCC) is a highly lethal cancer and the second leading cause of cancer-related deaths worldwide. As demonstrated in other solid neoplasms and HCC, infiltrating CD8 We investigated the association between the main necroptosis-related genes, that is, Our findings highlighted the immunogenetic role of necroptosis and its potential prognostic role in HCC: RIPK1, RIPK3 and MLKL-p were found significantly associated with intratumoral CD3 Our results confirmed the immunogenetic properties of necroptosis (NCP) in human HCC, showing that tumor-infiltrating lymphocytes (TILs) and, specifically, CD8+ T cells accumulate in tumors with higher expression of the necroptosis-related genes. These results suggest the importance of further studies to better assess the specific composition, as well as the functional features of the immune environment associated with a necroptotic signature in order to explore new possible diagnostic and immunotherapeutic scenarios.

Sections du résumé

BACKGROUND
Hepatocellular carcinoma (HCC) is a highly lethal cancer and the second leading cause of cancer-related deaths worldwide. As demonstrated in other solid neoplasms and HCC, infiltrating CD8
METHODS
We investigated the association between the main necroptosis-related genes, that is,
RESULTS
Our findings highlighted the immunogenetic role of necroptosis and its potential prognostic role in HCC: RIPK1, RIPK3 and MLKL-p were found significantly associated with intratumoral CD3
CONCLUSIONS
Our results confirmed the immunogenetic properties of necroptosis (NCP) in human HCC, showing that tumor-infiltrating lymphocytes (TILs) and, specifically, CD8+ T cells accumulate in tumors with higher expression of the necroptosis-related genes. These results suggest the importance of further studies to better assess the specific composition, as well as the functional features of the immune environment associated with a necroptotic signature in order to explore new possible diagnostic and immunotherapeutic scenarios.

Identifiants

pubmed: 35264437
pii: jitc-2021-004031
doi: 10.1136/jitc-2021-004031
pmc: PMC8915343
pii:
doi:

Substances chimiques

MLKL protein, human EC 2.7.-
Protein Kinases EC 2.7.-
RIPK1 protein, human EC 2.7.11.1
RIPK3 protein, human EC 2.7.11.1
Receptor-Interacting Protein Serine-Threonine Kinases EC 2.7.11.1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© Author(s) (or their employer(s)) 2022. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

Déclaration de conflit d'intérêts

Competing interests: None declared.

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Auteurs

Lorenzo Nicolè (L)

Department of Medicine (DIMED), University of Padova, Padova, Italy.
Department of Pathology, Angelo Hospital, Mestre, Italy.

Tiziana Sanavia (T)

Department of Medical Sciences, University of Torino, Torino, Italy.

Rocco Cappellesso (R)

Department of Pathology, Padova University Hospital, Padova, Italy.

Valeria Maffeis (V)

Department of Pathology, Azienda ULSS2 Marca Trevigiana, Treviso, Italy.

Jun Akiba (J)

Department of Diagnostic Pathology, Kurume University Hospital, Kurume, Japan.

Akihiko Kawahara (A)

Department of Diagnostic Pathology, Kurume University Hospital, Kurume, Japan.

Yoshiki Naito (Y)

Department of Diagnostic Pathology, Kurume University Hospital, Kurume, Japan.

Claudia Maria Radu (CM)

Department of Medicine, General Internal Medicine and Thrombotic and Hemorrhagic Diseases Unit, University of Padova, Padova, Italy.

Paolo Simioni (P)

Department of Medicine, General Internal Medicine and Thrombotic and Hemorrhagic Diseases Unit, University of Padova, Padova, Italy.

Davide Serafin (D)

Department of Statistical Sciences, University of Padova, Padova, Italy.

Giuliana Cortese (G)

Department of Statistical Sciences, University of Padova, Padova, Italy.

Maria Guido (M)

Department of Medicine (DIMED), University of Padova, Padova, Italy.
Department of Pathology, Azienda ULSS2 Marca Trevigiana, Treviso, Italy.

Giacomo Zanus (G)

Department of Surgery, Oncology and Gastroenterology, University of Padova, Padova, Italy.
II Surgery Unit, Regional Hospital Treviso, Treviso, Italy.

Hirohisa Yano (H)

Department of Pathology, Kurume University School of Medicine, Kurume, Japan.

Ambrogio Fassina (A)

Department of Medicine (DIMED), University of Padova, Padova, Italy ambrogio.fassina@unipd.it.

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Classifications MeSH