A pan-tissue DNA methylation atlas enables in silico decomposition of human tissue methylomes at cell-type resolution.


Journal

Nature methods
ISSN: 1548-7105
Titre abrégé: Nat Methods
Pays: United States
ID NLM: 101215604

Informations de publication

Date de publication:
03 2022
Historique:
received: 11 07 2021
accepted: 28 01 2022
entrez: 12 3 2022
pubmed: 13 3 2022
medline: 28 4 2022
Statut: ppublish

Résumé

Bulk-tissue DNA methylomes represent an average over many different cell types, hampering our understanding of cell-type-specific contributions to disease development. As single-cell methylomics is not scalable to large cohorts of individuals, cost-effective computational solutions are needed, yet current methods are limited to tissues such as blood. Here we leverage the high-resolution nature of tissue-specific single-cell RNA-sequencing datasets to construct a DNA methylation atlas defined for 13 solid tissue types and 40 cell types. We comprehensively validate this atlas in independent bulk and single-nucleus DNA methylation datasets. We demonstrate that it correctly predicts the cell of origin of diverse cancer types and discovers new prognostic associations in olfactory neuroblastoma and stage 2 melanoma. In brain, the atlas predicts a neuronal origin for schizophrenia, with neuron-specific differential DNA methylation enriched for corresponding genome-wide association study risk loci. In summary, the DNA methylation atlas enables the decomposition of 13 different human tissue types at a high cellular resolution, paving the way for an improved interpretation of epigenetic data.

Identifiants

pubmed: 35277705
doi: 10.1038/s41592-022-01412-7
pii: 10.1038/s41592-022-01412-7
pmc: PMC8916958
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

296-306

Informations de copyright

© 2022. The Author(s).

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Auteurs

Tianyu Zhu (T)

CAS Key Laboratory of Computational Biology, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China.

Jacklyn Liu (J)

University College London, London, UK.

Stephan Beck (S)

UCL Cancer Institute, Paul O'Gorman Building, University College London, London, UK.

Sun Pan (S)

Department of Cardiac Surgery, Zhongshan Hospital, Fudan University, Shanghai, China.

David Capper (D)

Institut für Neuropathologie, Charité Universitätsmedizin, Berlin, Germany.
Charité ‑ Universitätsmedizin Berlin, Corporate Member of Freie Universitat Berlin and Humboldt‑Universitat zu Berlin, Department of Neuropathology, Berlin, Germany.
German Cancer Consortium (DKTK), Partner Site Berlin, German Cancer Research Center (DKFZ), Heidelberg, Germany.

Matt Lechner (M)

University College London, London, UK.
Department of ENT, Barts Health NHS Trust, London, UK.

Chrissie Thirlwell (C)

University of Exeter Medical School, University of Exeter, Exeter, UK.

Charles E Breeze (CE)

UCL Cancer Institute, Paul O'Gorman Building, University College London, London, UK. c.breeze@ucl.ac.uk.
Altius Institute for Biomedical Sciences, Seattle, WA, USA. c.breeze@ucl.ac.uk.

Andrew E Teschendorff (AE)

CAS Key Laboratory of Computational Biology, Shanghai Institute of Nutrition and Health, University of Chinese Academy of Sciences, Chinese Academy of Sciences, Shanghai, China. andrew@picb.ac.cn.
UCL Cancer Institute, Paul O'Gorman Building, University College London, London, UK. andrew@picb.ac.cn.

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