Macrophages use apoptotic cell-derived methionine and DNMT3A during efferocytosis to promote tissue resolution.


Journal

Nature metabolism
ISSN: 2522-5812
Titre abrégé: Nat Metab
Pays: Germany
ID NLM: 101736592

Informations de publication

Date de publication:
04 2022
Historique:
received: 02 08 2021
accepted: 11 02 2022
pubmed: 2 4 2022
medline: 30 4 2022
entrez: 1 4 2022
Statut: ppublish

Résumé

Efferocytosis, the clearance of apoptotic cells (ACs) by macrophages, is critical for tissue resolution, with defects driving many diseases. Mechanisms of efferocytosis-mediated resolution are incompletely understood. Here, we show that AC-derived methionine regulates resolution through epigenetic repression of the extracellular signal-regulated kinase 1/2 (ERK1/2) phosphatase Dusp4. We focus on two key efferocytosis-induced pro-resolving mediators, prostaglandin E

Identifiants

pubmed: 35361955
doi: 10.1038/s42255-022-00551-7
pii: 10.1038/s42255-022-00551-7
pmc: PMC9050866
mid: NIHMS1780101
doi:

Substances chimiques

Dnmt3a protein, mouse 0
Prostaglandins E 0
Transforming Growth Factor beta1 0
Methionine AE28F7PNPL
Cyclooxygenase 2 EC 1.14.99.1
DNA Methyltransferase 3A EC 2.1.1.37

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

444-457

Subventions

Organisme : NHLBI NIH HHS
ID : R35 HL145228
Pays : United States
Organisme : NHLBI NIH HHS
ID : R00 HL145131
Pays : United States
Organisme : NIH HHS
ID : S10 OD030286
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL155431
Pays : United States
Organisme : NHLBI NIH HHS
ID : P01 HL087123
Pays : United States

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2022. The Author(s), under exclusive licence to Springer Nature Limited.

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Auteurs

Patrick B Ampomah (PB)

Department of Medicine, Columbia University Irving Medical Center, New York, NY, USA. pbampomah1@gmail.com.
Novartis Institutes for BioMedical Research, Cambridge, MA, USA. pbampomah1@gmail.com.

Bishuang Cai (B)

Division of Liver Diseases, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Santosh R Sukka (SR)

Department of Medicine, Columbia University Irving Medical Center, New York, NY, USA.

Brennan D Gerlach (BD)

Novartis Institutes for BioMedical Research, Cambridge, MA, USA.

Arif Yurdagul (A)

Department of Molecular and Cellular Physiology, LSU Health Shreveport, Shreveport, LA, USA.

Xiaobo Wang (X)

Department of Medicine, Columbia University Irving Medical Center, New York, NY, USA.

George Kuriakose (G)

Department of Medicine, Columbia University Irving Medical Center, New York, NY, USA.

Lancia N F Darville (LNF)

Proteomics and Metabolomics Core, H. Lee Moffitt Cancer Center and Research Institute, Tampa, FL, USA.

Yan Sun (Y)

Department of Biochemistry, Albert Einstein College of Medicine, New York, NY, USA.

Simone Sidoli (S)

Department of Biochemistry, Albert Einstein College of Medicine, New York, NY, USA.

John M Koomen (JM)

Proteomics and Metabolomics Core, H. Lee Moffitt Cancer Center and Research Institute, Tampa, FL, USA.

Alan R Tall (AR)

Department of Medicine, Columbia University Irving Medical Center, New York, NY, USA.

Ira Tabas (I)

Department of Medicine, Columbia University Irving Medical Center, New York, NY, USA. iat1@columbia.edu.
Department of Pathology and Cell Biology, Columbia University Irving Medical Center, New York, NY, USA. iat1@columbia.edu.
Department of Physiology, Columbia University Irving Medical Center, New York, NY, USA. iat1@columbia.edu.

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