B cells and tertiary lymphoid structures as determinants of tumour immune contexture and clinical outcome.


Journal

Nature reviews. Clinical oncology
ISSN: 1759-4782
Titre abrégé: Nat Rev Clin Oncol
Pays: England
ID NLM: 101500077

Informations de publication

Date de publication:
07 2022
Historique:
accepted: 04 03 2022
pubmed: 3 4 2022
medline: 28 6 2022
entrez: 2 4 2022
Statut: ppublish

Résumé

B cells are a major component of the tumour microenvironment, where they are predominantly associated with tertiary lymphoid structures (TLS). In germinal centres within mature TLS, B cell clones are selectively activated and amplified, and undergo antibody class switching and somatic hypermutation. Subsequently, these B cell clones differentiate into plasma cells that can produce IgG or IgA antibodies targeting tumour-associated antigens. In tumours without mature TLS, B cells are either scarce or differentiate into regulatory cells that produce immunosuppressive cytokines. Indeed, different tumours vary considerably in their TLS and B cell content. Notably, tumours with mature TLS, a high density of B cells and plasma cells, as well as the presence of antibodies to tumour-associated antigens are typically associated with favourable clinical outcomes and responses to immunotherapy compared with those lacking these characteristics. However, polyclonal B cell activation can also result in the formation of immune complexes that trigger the production of pro-inflammatory cytokines by macrophages and neutrophils. In complement-rich tumours, IgG antibodies can also activate the complement cascade, resulting in the production of anaphylatoxins that sustain tumour-promoting inflammation and angiogenesis. Herein, we review the phenotypic heterogeneity of intratumoural B cells and the importance of TLS in their generation as well as the potential of B cells and TLS as prognostic and predictive biomarkers. We also discuss novel therapeutic approaches that are being explored with the aim of increasing mature TLS formation, B cell differentiation and anti-tumour antibody production within tumours.

Identifiants

pubmed: 35365796
doi: 10.1038/s41571-022-00619-z
pii: 10.1038/s41571-022-00619-z
doi:

Substances chimiques

Antigens, Neoplasm 0
Cytokines 0
Immunoglobulin G 0

Types de publication

Journal Article Review Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

441-457

Informations de copyright

© 2022. Springer Nature Limited.

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Auteurs

Wolf H Fridman (WH)

Centre de Recherche des Cordeliers, INSERM, Sorbonne Université, Université Paris-Cité, Equipe inflammation, complément et cancer, Paris, France. herve.fridman@crc.jussieu.fr.
Equipe labellisée Ligue contre le Cancer, Paris, France. herve.fridman@crc.jussieu.fr.

Maxime Meylan (M)

Centre de Recherche des Cordeliers, INSERM, Sorbonne Université, Université Paris-Cité, Equipe inflammation, complément et cancer, Paris, France.
Equipe labellisée Ligue contre le Cancer, Paris, France.

Florent Petitprez (F)

MRC Centre for Reproductive Health, Queen's Medical Research Institute, The University of Edinburgh, Edinburgh, UK.

Cheng-Ming Sun (CM)

Centre de Recherche des Cordeliers, INSERM, Sorbonne Université, Université Paris-Cité, Equipe inflammation, complément et cancer, Paris, France.
Equipe labellisée Ligue contre le Cancer, Paris, France.

Antoine Italiano (A)

Faculty of Medicine, University of Bordeaux, Bordeaux, France.
Department of Medicine, Institute Bergonié, Bordeaux, France.

Catherine Sautès-Fridman (C)

Centre de Recherche des Cordeliers, INSERM, Sorbonne Université, Université Paris-Cité, Equipe inflammation, complément et cancer, Paris, France.
Equipe labellisée Ligue contre le Cancer, Paris, France.

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