A pilot clinical study to Evaluate Liraglutide-mediated Anti-platelet activity in patients with type-2 Diabetes (ELAID study).


Journal

Journal of diabetes and its complications
ISSN: 1873-460X
Titre abrégé: J Diabetes Complications
Pays: United States
ID NLM: 9204583

Informations de publication

Date de publication:
05 2022
Historique:
received: 27 01 2022
revised: 24 03 2022
accepted: 26 03 2022
pubmed: 7 4 2022
medline: 20 4 2022
entrez: 6 4 2022
Statut: ppublish

Résumé

Liraglutide is an effective treatment for the management of type 2 diabetes mellitus (T2DM). In addition to glycemic control and potential cardioprotective effects, recent studies suggest a possible role for liraglutide in the inhibition of platelet reactivity, further attenuating atherothrombotic risk in patients with T2DM. We evaluated the in-vivo antiplatelet effect of liraglutide in T2DM patients without macrovascular disease or concurrent anti-platelet therapy. A double-blind, placebo-controlled pilot study of 16 T2DM patients, 51-69 y/o, (mean age 60.4 y/o, 63.0% male) randomised to receive liraglutide (1.8 mg/day) or placebo (saline) for 6 months was conducted. Platelet aggregation studies at baseline and after initiation of the study intervention: days 1, 7, and 14 and months 1, 3 and 6 were performed. Liraglutide (n = 7) and placebo (n = 9) treated patients demonstrated normal platelet aggregation responses although transient and significant attenuation in maximum slope of platelet aggregation in response to collagen (p ≤ 0.05), arachidonic acid (p ≤ 0.05) and ADP (p ≤ 0.02) was observed in liraglutide compared to placebo treated patients in the first week. In this pilot study of patients with T2DM liraglutide treatment was associated with a significant, early and transient decrease in maximum slope of platelet aggregation. The clinical significance of this effect is currently unknown and may warrant further investigation. UTN 1111-1181-9567.

Sections du résumé

BACKGROUND
Liraglutide is an effective treatment for the management of type 2 diabetes mellitus (T2DM). In addition to glycemic control and potential cardioprotective effects, recent studies suggest a possible role for liraglutide in the inhibition of platelet reactivity, further attenuating atherothrombotic risk in patients with T2DM. We evaluated the in-vivo antiplatelet effect of liraglutide in T2DM patients without macrovascular disease or concurrent anti-platelet therapy.
METHODS
A double-blind, placebo-controlled pilot study of 16 T2DM patients, 51-69 y/o, (mean age 60.4 y/o, 63.0% male) randomised to receive liraglutide (1.8 mg/day) or placebo (saline) for 6 months was conducted. Platelet aggregation studies at baseline and after initiation of the study intervention: days 1, 7, and 14 and months 1, 3 and 6 were performed.
RESULTS
Liraglutide (n = 7) and placebo (n = 9) treated patients demonstrated normal platelet aggregation responses although transient and significant attenuation in maximum slope of platelet aggregation in response to collagen (p ≤ 0.05), arachidonic acid (p ≤ 0.05) and ADP (p ≤ 0.02) was observed in liraglutide compared to placebo treated patients in the first week.
CONCLUSIONS
In this pilot study of patients with T2DM liraglutide treatment was associated with a significant, early and transient decrease in maximum slope of platelet aggregation. The clinical significance of this effect is currently unknown and may warrant further investigation.
CLINICAL TRIAL REGISTRATION NUMBER
UTN 1111-1181-9567.

Identifiants

pubmed: 35382966
pii: S1056-8727(22)00082-4
doi: 10.1016/j.jdiacomp.2022.108188
pii:
doi:

Substances chimiques

Hypoglycemic Agents 0
Liraglutide 839I73S42A

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

108188

Informations de copyright

Copyright © 2022 Elsevier Inc. All rights reserved.

Auteurs

Jayasree Loganathan (J)

Eastern Health Clinical School, Monash University, Melbourne, Victoria, Australia.

Adam C Cohen (AC)

Eastern Health Clinical School, Monash University, Melbourne, Victoria, Australia.

Georgia M Kaloupis (GM)

Eastern Health Clinical School, Monash University, Melbourne, Victoria, Australia.

Carolyn Harris (C)

Eastern Health Clinical School, Monash University, Melbourne, Victoria, Australia.

Andriana Chronopoulos (A)

Eastern Health Clinical School, Monash University, Melbourne, Victoria, Australia.

Vanessa James (V)

Eastern Health Clinical School, Monash University, Melbourne, Victoria, Australia.

Justin Hamilton (J)

Australian Centre for Blood Diseases, Monash University, Prahran, Melbourne, Victoria, Australia.

Sarah Green (S)

Alfred Pathology Service, Alfred Health, Alfred Hospital, Melbourne, Victoria, Australia.

Andrew Wallis (A)

Alfred Pathology Service, Alfred Health, Alfred Hospital, Melbourne, Victoria, Australia.

Susan Morgan (S)

Alfred Pathology Service, Alfred Health, Alfred Hospital, Melbourne, Victoria, Australia.

Raymond Dauer (R)

Department of Pathology, Eastern Health, Box Hill Hospital, Melbourne, Australia.

Christopher Gilfillan (C)

Eastern Health Clinical School, Monash University, Melbourne, Victoria, Australia; Department of Endocrinology, Eastern Health, Box Hill Hospital, Melbourne, Victoria, Australia.

Anthony E Dear (AE)

Eastern Health Clinical School, Monash University, Melbourne, Victoria, Australia. Electronic address: Anthony.dear@monash.edu.

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Classifications MeSH