Biomarker changes as surrogate endpoints in early-phase trials in heart failure with reduced ejection fraction.

Biomarkers Heart failure with reduced ejection fraction Phase 2 Randomized trial Surrogate endpoint Surrogate outcome

Journal

ESC heart failure
ISSN: 2055-5822
Titre abrégé: ESC Heart Fail
Pays: England
ID NLM: 101669191

Informations de publication

Date de publication:
08 2022
Historique:
revised: 04 03 2022
received: 20 12 2021
accepted: 14 03 2022
pubmed: 8 4 2022
medline: 20 7 2022
entrez: 7 4 2022
Statut: ppublish

Résumé

No biomarker has achieved widespread acceptance as a surrogate endpoint for early-phase heart failure (HF) trials. We assessed whether changes over time in a panel of plasma biomarkers were associated with subsequent morbidity/mortality in HF with reduced ejection fraction (HFrEF). In 1040 patients with HFrEF from the BIOSTAT-CHF cohort, we investigated the associations between changes in the plasma concentrations of 30 biomarkers, before (baseline) and after (9 months) attempted optimization of guideline-recommended therapy, on top of the BIOSTAT risk score and the subsequent risk of HF hospitalization/all-cause mortality using Cox regression models. C-statistics were calculated to assess discriminatory power of biomarker changes/month-nine assessment. Changes in N-terminal pro-B-type natriuretic peptide (NT-proBNP) and WAP four-disulphide core domain protein HE4 (WAP-4C) were the only independent predictors of the outcome after adjusting for their baseline plasma concentration, 28 other biomarkers (both baseline and changes), and BIOSTAT risk score at baseline. When adjusting for month-nine rather than baseline biomarkers concentrations, only changes in NT-proBNP were independently associated with the outcome. The C-statistic of the model including the BIOSTAT risk score and NT-proBNP increased by 4% when changes were considered on top of baseline concentrations and by 1% when changes in NT-proBNP were considered on top of its month-nine concentrations and the BIOSTAT risk score. Among 30 relevant biomarkers, a change over time was significantly and independently associated with HF hospitalization/all-cause death only for NT-proBNP. Changes over time were modestly more prognostic than baseline or end-values alone. Changes in biomarkers should be further explored as potential surrogate endpoints in early phase HF trials.

Identifiants

pubmed: 35388650
doi: 10.1002/ehf2.13917
pmc: PMC9288797
doi:

Substances chimiques

Biomarkers 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2107-2118

Informations de copyright

© 2022 The Authors. ESC Heart Failure published by John Wiley & Sons Ltd on behalf of European Society of Cardiology.

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Auteurs

Gianluigi Savarese (G)

Division of Cardiology, Department of Medicine, Karolinska Institutet, Stockholm, Sweden.

Alicia Uijl (A)

Division of Cardiology, Department of Medicine, Karolinska Institutet, Stockholm, Sweden.
Julius Center for Health Sciences and Primary Care, University Medical Center Utrecht, Utrecht University, Utrecht, The Netherlands.

Wouter Ouwerkerk (W)

National Heart Centre Singapore, Singapore.
Department of Dermatology, Amsterdam UMC, University of Amsterdam, Amsterdam Infection & Immunity Institute, Amsterdam, The Netherlands.

Jasper Tromp (J)

National Heart Centre Singapore, Singapore.
Saw Swee Hock School of Public Health, National University of Singapore, Singapore.
Duke-NUS Medical School, Singapore.
Department of Cardiology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.

Stefan D Anker (SD)

Department of Cardiology (CVK); and Berlin Institute of Health Center for Regenerative Therapies (BCRT); German Centre for Cardiovascular Research (DZHK) partner site Berlin, Charité Universitätsmedizin Berlin, Berlin, Germany.

Kenneth Dickstein (K)

Stavanger University Hospital, Stavanger, Norway.
University of Bergen, Bergen, Norway.

Camilla Hage (C)

Division of Cardiology, Department of Medicine, Karolinska Institutet, Stockholm, Sweden.

Carolyn S P Lam (CSP)

National Heart Centre Singapore, Singapore.
Duke-NUS Medical School, Singapore.

Chim C Lang (CC)

Division of Molecular and Clinical Medicine, University of Dundee, Dundee, UK.

Marco Metra (M)

Cardiology Unit, ASST Spedali Civili and Department of Medical and Surgical Specialties, Radiological Sciences and Public Health, University of Brescia, Brescia, Italy.

Leong L Ng (LL)

Department of Cardiovascular Sciences, University of Leicester, Glenfield Hospital, and NIHR Leicester Biomedical Research Centre, Leicester, UK.

Nicola Orsini (N)

Department of Global Public Health, Karolinska Institutet, Stockholm, Sweden.

Nilesh J Samani (NJ)

Department of Cardiovascular Sciences, University of Leicester, Glenfield Hospital, and NIHR Leicester Biomedical Research Centre, Leicester, UK.

Dirk J van Veldhuisen (DJ)

Department of Cardiology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.

John G F Cleland (JGF)

Robertson Centre for Biostatistics, Institute of Health and Wellbeing, University of Glasgow, Glasgow and National Heart & Lung Institute, Imperial College, London, UK.

Adriaan A Voors (AA)

Department of Cardiology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.

Lars H Lund (LH)

Division of Cardiology, Department of Medicine, Karolinska Institutet, Stockholm, Sweden.

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