Recombinant von Willebrand factor prophylaxis in patients with severe von Willebrand disease: phase 3 study results.


Journal

Blood
ISSN: 1528-0020
Titre abrégé: Blood
Pays: United States
ID NLM: 7603509

Informations de publication

Date de publication:
14 07 2022
Historique:
received: 16 11 2021
accepted: 05 04 2022
pubmed: 20 4 2022
medline: 19 7 2022
entrez: 19 4 2022
Statut: ppublish

Résumé

International guidelines conditionally recommend long-term prophylaxis in patients with von Willebrand disease (VWD) and severe and frequent bleeding. As recombinant von Willebrand factor (rVWF; vonicog alfa) may reduce the frequency of treated spontaneous bleeding events (BEs), we investigated the efficacy and safety of rVWF prophylaxis in adults with severe VWD. Patients with BEs requiring VWF therapy in the past year (on-demand VWF therapy [prior on-demand group] or plasma-derived VWF prophylaxis [pdVWF; switch group]) were enrolled in a prospective, open-label, nonrandomized, phase 3 study. The planned duration of rVWF prophylaxis was 12 months; starting rVWF dose was 50 ± 10 VWF: ristocetin cofactor (VWF:RCo) IU/kg twice weekly (prior on-demand group) or based on prior pdVWF weekly dose/dosing frequency (switch group). The primary endpoint was annualized bleeding rate (ABR) of treated spontaneous BEs (sABR) during rVWF prophylaxis. Over the 12-month study period, treated sABR decreased by 91.5% on-study vs historical sABR in 13 patients in the prior on-demand group, and by 45.0% in 10 patients in the switch group (model-based analysis ratio, 0.085; 95% confidence interval [CI], 0.021-0.346 and 0.550; 95% CI, 0.086-3.523, respectively). No treated spontaneous BEs were recorded in 84.6% (11/13) and 70.0% (7/10) of patients, respectively. The safety profile of rVWF was consistent with the previously established profile, with no new adverse drug reactions identified. Findings suggest that rVWF prophylaxis can reduce treated spontaneous BEs in patients previously receiving on-demand VWF therapy and maintains at least the same level of hemostatic control in patients who switch from prophylaxis with pdVWF to rVWF, with a favorable safety profile. This trial was registered at www.clinicaltrials.gov (#NCT02973087) and www.clinicaltrialsregister.eu (#EudraCT 2016-001478-14).

Identifiants

pubmed: 35439298
pii: S0006-4971(22)00545-6
doi: 10.1182/blood.2021014810
pmc: PMC9283967
doi:

Substances chimiques

Hemostatics 0
Recombinant Proteins 0
von Willebrand Factor 0

Banques de données

ClinicalTrials.gov
['NCT02973087']

Types de publication

Clinical Trial, Phase III Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

89-98

Informations de copyright

© 2022 by The American Society of Hematology.

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Auteurs

Frank W G Leebeek (FWG)

Department of Hematology, University Medical Center, Erasmus MC, Rotterdam, The Netherlands.

Flora Peyvandi (F)

Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico, Angelo Bianchi Bonomi Hemophilia and Thrombosis Center, Milan, Italy.
Department of Pathophysiology and Transplantation, Università degli Studi di Milano, Milan, Italy.

Miguel Escobar (M)

University of Texas Health Science Center at Houston, Houston, TX.

Andreas Tiede (A)

Department of Hematology, Hemostasis, Oncology, and Stem Cell Transplantation, Hannover Medical School, Hannover, Germany.

Giancarlo Castaman (G)

Center for Bleeding Disorders and Coagulation, Careggi University Hospital, Florence, Italy.

Michael Wang (M)

Hemophilia and Thrombosis Center, University of Colorado Anschutz Medical Campus, Aurora, CO.

Tung Wynn (T)

Department of Pediatrics, University of Florida, Gainesville, FL.

Jovanna Baptista (J)

Takeda Development Center Americas, Inc., Lexington, MA; and.

Yi Wang (Y)

Takeda Development Center Americas, Inc, Cambridge, MA.

Jingmei Zhang (J)

Takeda Development Center Americas, Inc, Cambridge, MA.

Björn Mellgård (B)

Takeda Development Center Americas, Inc, Cambridge, MA.

Gülden Özen (G)

Takeda Development Center Americas, Inc, Cambridge, MA.

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