Quantitative Blood Volume Analysis and Hemodynamic Measures of Vascular Compliance in Patients With Worsening Heart Failure.


Journal

Journal of cardiac failure
ISSN: 1532-8414
Titre abrégé: J Card Fail
Pays: United States
ID NLM: 9442138

Informations de publication

Date de publication:
09 2022
Historique:
received: 07 03 2022
revised: 25 03 2022
accepted: 09 04 2022
pubmed: 29 4 2022
medline: 21 9 2022
entrez: 28 4 2022
Statut: ppublish

Résumé

The role of blood volume (BV) expansion vs a change in vascular compliance in worsening heart failure (HF) remains under debate. We aimed to assess the relationship between BV and resting and stress hemodynamics in worsening HF and to further elucidate the significance of BV in cardiac decompensation. Patients with worsening HF underwent radiolabeled indicator-dilution BV analysis and cardiac catheterization. Intravascular volumes and resting/stress hemodynamics were recorded. Provocative stress maneuvers included change in systolic blood pressure (ΔSBP) from lying to standing and Valsalva and intracardiac pressure changes with leg raise. Correlation between BV and invasive hemodynamics were assessed by linear regression. Of 27 patients with worsening HF, patients' characteristics included mean age 61 ± 12 years, 70% male, 19% Black, and mean ejection fraction 29% ± 15%. Of the patients, 13 (48%) had hypervolemia as measured by total BV, which weakly correlated with ΔSBP by position (R In patients with worsening HF, BV mildly correlated with intracardiac pressures at rest. Provocative maneuvers intended to test vascular compliance did not correlate with BV, indicating that compliance may serve as a stand-alone metric in HF.

Sections du résumé

BACKGROUND
The role of blood volume (BV) expansion vs a change in vascular compliance in worsening heart failure (HF) remains under debate. We aimed to assess the relationship between BV and resting and stress hemodynamics in worsening HF and to further elucidate the significance of BV in cardiac decompensation.
METHODS AND RESULTS
Patients with worsening HF underwent radiolabeled indicator-dilution BV analysis and cardiac catheterization. Intravascular volumes and resting/stress hemodynamics were recorded. Provocative stress maneuvers included change in systolic blood pressure (ΔSBP) from lying to standing and Valsalva and intracardiac pressure changes with leg raise. Correlation between BV and invasive hemodynamics were assessed by linear regression. Of 27 patients with worsening HF, patients' characteristics included mean age 61 ± 12 years, 70% male, 19% Black, and mean ejection fraction 29% ± 15%. Of the patients, 13 (48%) had hypervolemia as measured by total BV, which weakly correlated with ΔSBP by position (R
CONCLUSIONS
In patients with worsening HF, BV mildly correlated with intracardiac pressures at rest. Provocative maneuvers intended to test vascular compliance did not correlate with BV, indicating that compliance may serve as a stand-alone metric in HF.

Identifiants

pubmed: 35483537
pii: S1071-9164(22)00494-8
doi: 10.1016/j.cardfail.2022.04.004
pmc: PMC10311606
mid: NIHMS1906043
pii:
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1469-1474

Subventions

Organisme : NHLBI NIH HHS
ID : K23 HL151744
Pays : United States
Organisme : NHLBI NIH HHS
ID : T32 HL069749
Pays : United States

Informations de copyright

Copyright © 2022 Elsevier Inc. All rights reserved.

Références

J Card Fail. 2021 Apr;27(4):445-452
pubmed: 33347996
J Am Coll Cardiol. 2019 Mar 5;73(8):935-944
pubmed: 30819362
Am J Cardiol. 2004 May 15;93(10):1254-9
pubmed: 15135699
J Appl Physiol (1985). 1991 Oct;71(4):1563-7
pubmed: 1757382
J Am Heart Assoc. 2018 Jun 12;7(12):
pubmed: 29895590
Circ Heart Fail. 2016 Aug;9(8):e002922
pubmed: 27436837
J Card Fail. 2022 May;28(5):870-872
pubmed: 35158025

Auteurs

Vishal N Rao (VN)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina; Duke Clinical Research Institute, Duke University School of Medicine, Durham, North Carolina.

Jennifer Andrews (J)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina.

Willard N Applefeld (WN)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina; Critical Care Medicine Department, National Institutes of Health, Bethesda, Maryland.

James M Gray (JM)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina.

Jeroen Molinger (J)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina.

G Michael Felker (GM)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina; Duke Clinical Research Institute, Duke University School of Medicine, Durham, North Carolina.

Wayne L Miller (WL)

Mayo Clinic, Rochester, Minnesota.

Manesh R Patel (MR)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina; Duke Clinical Research Institute, Duke University School of Medicine, Durham, North Carolina.

Adrian F Hernandez (AF)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina; Duke Clinical Research Institute, Duke University School of Medicine, Durham, North Carolina.

Marat Fudim (M)

Division of Cardiology, Duke University Medical Center, Durham, North Carolina; Duke Clinical Research Institute, Duke University School of Medicine, Durham, North Carolina. Electronic address: marat.fudim@duke.edu.

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