Peroxynitrous acid-modified extracellular matrix alters gene and protein expression in human coronary artery smooth muscle cells and induces a pro-inflammatory phenotype.


Journal

Free radical biology & medicine
ISSN: 1873-4596
Titre abrégé: Free Radic Biol Med
Pays: United States
ID NLM: 8709159

Informations de publication

Date de publication:
06 2022
Historique:
received: 17 11 2021
revised: 29 03 2022
accepted: 03 05 2022
pubmed: 9 5 2022
medline: 3 6 2022
entrez: 8 5 2022
Statut: ppublish

Résumé

Leukocytes produce oxidants at inflammatory sites, including within the artery wall during the development of atherosclerosis. Developing lesions contain high numbers of activated leukocytes that generate reactive nitrogen species, including peroxynitrite/peroxynitrous acid (ONOO

Identifiants

pubmed: 35526806
pii: S0891-5849(22)00176-9
doi: 10.1016/j.freeradbiomed.2022.05.001
pii:
doi:

Substances chimiques

Oxidants 0
Vascular Cell Adhesion Molecule-1 0
Peroxynitrous Acid 14691-52-2

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

43-52

Informations de copyright

Copyright © 2022 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Sara M Jørgensen (SM)

Department of Biomedical Sciences, Panum Institute, University of Copenhagen, Copenhagen, 2200, Denmark.

Lasse G Lorentzen (LG)

Department of Biomedical Sciences, Panum Institute, University of Copenhagen, Copenhagen, 2200, Denmark.

Christine Y Chuang (CY)

Department of Biomedical Sciences, Panum Institute, University of Copenhagen, Copenhagen, 2200, Denmark.

Michael J Davies (MJ)

Department of Biomedical Sciences, Panum Institute, University of Copenhagen, Copenhagen, 2200, Denmark. Electronic address: davies@sund.ku.dk.

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Classifications MeSH