Actin assembly at sites of contact between the cortical ER and endocytic pits promotes ER autophagy.

Actin assembly End3-Pan1 Myo3/Myo5 Osh2/Osh3 Scs2/Scs22 contact sites endocytic pits endoplasmic reticulum reticulophagy

Journal

Autophagy
ISSN: 1554-8635
Titre abrégé: Autophagy
Pays: United States
ID NLM: 101265188

Informations de publication

Date de publication:
01 2023
Historique:
pubmed: 10 5 2022
medline: 30 12 2022
entrez: 9 5 2022
Statut: ppublish

Résumé

A recent screen of the

Identifiants

pubmed: 35532158
doi: 10.1080/15548627.2022.2074614
pmc: PMC9809928
doi:

Substances chimiques

Actins 0
Saccharomyces cerevisiae Proteins 0

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

358-359

Subventions

Organisme : NIGMS NIH HHS
ID : R01 GM035370
Pays : United States
Organisme : NIGMS NIH HHS
ID : R35 GM131681
Pays : United States
Organisme : NIGMS NIH HHS
ID : R37 GM035370
Pays : United States

Références

Proc Natl Acad Sci U S A. 2022 Feb 8;119(6):
pubmed: 35101986

Auteurs

Dongmei Liu (D)

Department of Cellular and Molecular Medicine, University of California San Diego, La Jolla, California, USA.

Susan Ferro-Novick (S)

Department of Cellular and Molecular Medicine, University of California San Diego, La Jolla, California, USA.

Peter Novick (P)

Department of Cellular and Molecular Medicine, University of California San Diego, La Jolla, California, USA.

Articles similaires

Animals Dogs Dog Diseases Autophagy Immunohistochemistry
Cerebrospinal Fluid Animals Liver Glymphatic System Spinal Cord
Adenosine Triphosphate Adenosine Diphosphate Mitochondrial ADP, ATP Translocases Binding Sites Mitochondria
Animals Humans Mice Neoplasms Tumor Microenvironment

Classifications MeSH