Differential prognostic burden of cardiovascular disease and lower-limb amputation on the risk of all-cause death in people with long-standing type 1 diabetes.


Journal

Cardiovascular diabetology
ISSN: 1475-2840
Titre abrégé: Cardiovasc Diabetol
Pays: England
ID NLM: 101147637

Informations de publication

Date de publication:
09 05 2022
Historique:
received: 03 02 2022
accepted: 28 02 2022
entrez: 9 5 2022
pubmed: 10 5 2022
medline: 12 5 2022
Statut: epublish

Résumé

Cardiovascular disease (CVD) and nontraumatic lower-limb amputation (LLA) each results in reduced life expectancy in patients with type 1 diabetes, but the differential burden between these conditions is unknown. We compared the effects of CVD and LLA on the risk of mortality in people with type 1 diabetes. We used pooled data from the SURGENE, GENEDIAB, and GENESIS prospective cohorts. Data were divided into: 1/absence of CVD (myocardial infarction and/or stroke) nor LLA, 2/history of CVD alone without LLA, 3/LLA alone without CVD or 4/both conditions at baseline. Participants with baseline history of peripheral artery disease were excluded from groups 1 and 2. The study endpoint was any death occurring during follow-up, regardless of the causes. Among 1169 participants (male 55%, age 40 ± 13 years, diabetes duration 23 ± 11 years), CVD, LLA or both were present at baseline in 49 (4.2%), 62 (5.3%) and 20 (1.7%) subjects, respectively. All-cause death occurred in 304 (26%) participants during 17-year follow-up, corresponding to 18,426 person-years and an incidence rate of 16 (95%CI, 15-18) per 1000 person-years. The risk of death increased in individuals with baseline history of CVD (adjusted HR 2.00 [95% CI 1.34-3.01], p = 0.0008) or LLA (2.26 [1.56-3.28], p < 0.0001), versus no condition, with an additive effect in people with both conditions (5.32 [3.14-9.00], p < 0.0001). No incremental risk of death was observed in people with CVD versus LLA (0.87 [0.54-1.41]). Compared with no condition, CVD and LLA were similarly associated with reduced life expectancy during follow-up: 2.79 (95% CI 1.26-4.32) and 3.38 (1.87-4.88) years, respectively. Combined conditions expose to 7.04 (4.76-9.31) less years of life expectancy (all p < 0.0001). CVD and LLA conferred a similar burden regarding mortality in type 1 diabetes population. Our findings encourage a careful consideration of people with type 1 diabetes and LLA as usually recommended for those with CVD, in terms of management of risk factors, treatments and prevention.

Sections du résumé

BACKGROUND
Cardiovascular disease (CVD) and nontraumatic lower-limb amputation (LLA) each results in reduced life expectancy in patients with type 1 diabetes, but the differential burden between these conditions is unknown. We compared the effects of CVD and LLA on the risk of mortality in people with type 1 diabetes.
METHODS
We used pooled data from the SURGENE, GENEDIAB, and GENESIS prospective cohorts. Data were divided into: 1/absence of CVD (myocardial infarction and/or stroke) nor LLA, 2/history of CVD alone without LLA, 3/LLA alone without CVD or 4/both conditions at baseline. Participants with baseline history of peripheral artery disease were excluded from groups 1 and 2. The study endpoint was any death occurring during follow-up, regardless of the causes.
RESULTS
Among 1169 participants (male 55%, age 40 ± 13 years, diabetes duration 23 ± 11 years), CVD, LLA or both were present at baseline in 49 (4.2%), 62 (5.3%) and 20 (1.7%) subjects, respectively. All-cause death occurred in 304 (26%) participants during 17-year follow-up, corresponding to 18,426 person-years and an incidence rate of 16 (95%CI, 15-18) per 1000 person-years. The risk of death increased in individuals with baseline history of CVD (adjusted HR 2.00 [95% CI 1.34-3.01], p = 0.0008) or LLA (2.26 [1.56-3.28], p < 0.0001), versus no condition, with an additive effect in people with both conditions (5.32 [3.14-9.00], p < 0.0001). No incremental risk of death was observed in people with CVD versus LLA (0.87 [0.54-1.41]). Compared with no condition, CVD and LLA were similarly associated with reduced life expectancy during follow-up: 2.79 (95% CI 1.26-4.32) and 3.38 (1.87-4.88) years, respectively. Combined conditions expose to 7.04 (4.76-9.31) less years of life expectancy (all p < 0.0001).
CONCLUSIONS
CVD and LLA conferred a similar burden regarding mortality in type 1 diabetes population. Our findings encourage a careful consideration of people with type 1 diabetes and LLA as usually recommended for those with CVD, in terms of management of risk factors, treatments and prevention.

Identifiants

pubmed: 35534880
doi: 10.1186/s12933-022-01487-8
pii: 10.1186/s12933-022-01487-8
pmc: PMC9088124
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

71

Informations de copyright

© 2022. The Author(s).

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Auteurs

Marion Camoin (M)

Department of Endocrinology, Diabetes and Nutrition, CEDEX, Bordeaux University Hospital, Hôpital Haut-Lévêque, Avenue de Magellan, 33604, Pessac, France.
Service d'Endocrinologie Diabétologie Nutrition, Hôpital Bichat, Fédération de Diabétologie de Paris, AP-HP, Université de Paris, Paris, France.

Gilberto Velho (G)

INEM, INSERM, Université de Paris, Paris, France.

Pierre-Jean Saulnier (PJ)

UFR de Médecine et Pharmacie, Université de Poitiers, Poitiers, France.
Centre d'Investigation Clinique, CHU de Poitiers, Poitiers, France.
Inserm, CIC 1402, Poitiers, France.

Louis Potier (L)

Service d'Endocrinologie Diabétologie Nutrition, Hôpital Bichat, Fédération de Diabétologie de Paris, AP-HP, Université de Paris, Paris, France.
INEM, INSERM, Université de Paris, Paris, France.

Yawa Abouleka (Y)

Service d'Endocrinologie Diabétologie Nutrition, Hôpital Bichat, Fédération de Diabétologie de Paris, AP-HP, Université de Paris, Paris, France.

Charlyne Carpentier (C)

Service d'Endocrinologie Diabétologie Nutrition, CHU d'Angers, Angers, France.

Severine Dubois (S)

Service d'Endocrinologie Diabétologie Nutrition, CHU d'Angers, Angers, France.

Alice Larroumet (A)

Department of Endocrinology, Diabetes and Nutrition, CEDEX, Bordeaux University Hospital, Hôpital Haut-Lévêque, Avenue de Magellan, 33604, Pessac, France.

Vincent Rigalleau (V)

Department of Endocrinology, Diabetes and Nutrition, CEDEX, Bordeaux University Hospital, Hôpital Haut-Lévêque, Avenue de Magellan, 33604, Pessac, France.
Faculty of Medicine, University of Bordeaux, Bordeaux, France.
INSERM U1219, Bordeaux Population Health Research Center, Bordeaux, France.

Elise Gand (E)

Centre d'Investigation Clinique, CHU de Poitiers, Poitiers, France.

Olivier Bourron (O)

Service de Diabétologie et Métabolisme, APHP, Groupe Hospitalier La Pitié-Salpêtrière, Sorbonne Université, Paris, France.
INSERM, UMRS 1138, Centre de Recherche des Cordeliers, Sorbonne Université, Paris, France.

Lyse Bordier (L)

Service d'Endocrinologie, Hôpital Bégin, Saint Mandé, France.

André Scheen (A)

CHU Liège, Liège Université, Liège, Belgique.

Samy Hadjadj (S)

Institut du Thorax, INSERM, CNRS, UNIV Nantes, CHU Nantes, Nantes, France.

Ronan Roussel (R)

Service d'Endocrinologie Diabétologie Nutrition, Hôpital Bichat, Fédération de Diabétologie de Paris, AP-HP, Université de Paris, Paris, France.
INEM, INSERM, Université de Paris, Paris, France.

Michel Marre (M)

INEM, INSERM, Université de Paris, Paris, France.
Clinique Ambroise Paré, Neuilly-sur-Seine, France.

Kamel Mohammedi (K)

Department of Endocrinology, Diabetes and Nutrition, CEDEX, Bordeaux University Hospital, Hôpital Haut-Lévêque, Avenue de Magellan, 33604, Pessac, France. km.mmohammedi@gmail.com.
Faculty of Medicine, University of Bordeaux, Bordeaux, France. km.mmohammedi@gmail.com.
Biology of Cardiovascular Diseases, INSERM Unit 1034, Pessac, France. km.mmohammedi@gmail.com.

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