ACE2 expression in adipose tissue is associated with cardio-metabolic risk factors and cell type composition-implications for COVID-19.


Journal

International journal of obesity (2005)
ISSN: 1476-5497
Titre abrégé: Int J Obes (Lond)
Pays: England
ID NLM: 101256108

Informations de publication

Date de publication:
08 2022
Historique:
received: 24 06 2021
accepted: 28 04 2022
revised: 21 04 2022
pubmed: 20 5 2022
medline: 28 7 2022
entrez: 19 5 2022
Statut: ppublish

Résumé

COVID-19 severity varies widely. Although some demographic and cardio-metabolic factors, including age and obesity, are associated with increasing risk of severe illness, the underlying mechanism(s) are uncertain. In a meta-analysis of three independent studies of 1471 participants in total, we investigated phenotypic and genetic factors associated with subcutaneous adipose tissue expression of Angiotensin I Converting Enzyme 2 (ACE2), measured by RNA-Seq, which acts as a receptor for SARS-CoV-2 cellular entry. Lower adipose tissue ACE2 expression was associated with multiple adverse cardio-metabolic health indices, including type 2 diabetes (T2D) (P = 9.14 × 10 Our results demonstrate that individuals with cardio-metabolic features known to increase risk of severe COVID-19 have lower background ACE2 levels in this highly relevant tissue. Reduced adipose tissue ACE2 expression may contribute to the pathophysiology of cardio-metabolic diseases, as well as the associated increased risk of severe COVID-19.

Sections du résumé

BACKGROUND
COVID-19 severity varies widely. Although some demographic and cardio-metabolic factors, including age and obesity, are associated with increasing risk of severe illness, the underlying mechanism(s) are uncertain.
SUBJECTS/METHODS
In a meta-analysis of three independent studies of 1471 participants in total, we investigated phenotypic and genetic factors associated with subcutaneous adipose tissue expression of Angiotensin I Converting Enzyme 2 (ACE2), measured by RNA-Seq, which acts as a receptor for SARS-CoV-2 cellular entry.
RESULTS
Lower adipose tissue ACE2 expression was associated with multiple adverse cardio-metabolic health indices, including type 2 diabetes (T2D) (P = 9.14 × 10
CONCLUSIONS
Our results demonstrate that individuals with cardio-metabolic features known to increase risk of severe COVID-19 have lower background ACE2 levels in this highly relevant tissue. Reduced adipose tissue ACE2 expression may contribute to the pathophysiology of cardio-metabolic diseases, as well as the associated increased risk of severe COVID-19.

Identifiants

pubmed: 35589964
doi: 10.1038/s41366-022-01136-w
pii: 10.1038/s41366-022-01136-w
pmc: PMC9119844
mid: EMS144632
doi:

Substances chimiques

Angiotensin-Converting Enzyme 2 EC 3.4.17.23

Types de publication

Journal Article Meta-Analysis Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1478-1486

Subventions

Organisme : Medical Research Council
ID : MR/R023131/1
Pays : United Kingdom
Organisme : NIDDK NIH HHS
ID : U01 DK062370
Pays : United States
Organisme : NIDDK NIH HHS
ID : P30 DK020572
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK093757
Pays : United States
Organisme : Wellcome Trust
ID : 221574
Pays : United Kingdom
Organisme : NIDDK NIH HHS
ID : R01 DK062370
Pays : United States
Organisme : Medical Research Council
ID : MR/M004422/1
Pays : United Kingdom

Commentaires et corrections

Type : UpdateOf

Informations de copyright

© 2022. The Author(s).

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Auteurs

Julia S El-Sayed Moustafa (JS)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK. julia.el-sayed_moustafa@kcl.ac.uk.

Anne U Jackson (AU)

Department of Biostatistics and Center for Statistical Genetics, School of Public Health, University of Michigan, Ann Arbor, MI, USA.

Sarah M Brotman (SM)

Department of Genetics, University of North Carolina, Chapel Hill, NC, USA.

Li Guan (L)

Department of Computational Medicine & Bioinformatics, University of Michigan, Ann Arbor, MI, USA.

Sergio Villicaña (S)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK.

Amy L Roberts (AL)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK.

Antonino Zito (A)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK.
Department of Molecular Biology, Massachusetts General Hospital, Boston, MA, 02114, USA.
Department of Genetics, Harvard Medical School, Boston, MA, 02114, USA.

Lori Bonnycastle (L)

National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, USA.

Michael R Erdos (MR)

National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, USA.

Narisu Narisu (N)

National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, USA.

Heather M Stringham (HM)

Department of Biostatistics and Center for Statistical Genetics, School of Public Health, University of Michigan, Ann Arbor, MI, USA.

Ryan Welch (R)

Department of Biostatistics and Center for Statistical Genetics, School of Public Health, University of Michigan, Ann Arbor, MI, USA.

Tingfen Yan (T)

National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, USA.

Timo Lakka (T)

Institute of Biomedicine/Physiology, University of Eastern Finland, Kuopio, Finland.
Kuopio Research Institute of Exercise Medicine, Kuopio, Finland.
Department of Clinical Physiology and Nuclear Medicine, Kuopio University Hospital, University of Eastern Finland, Kuopio, Finland.

Stephen Parker (S)

Department of Computational Medicine & Bioinformatics, University of Michigan, Ann Arbor, MI, USA.

Jaakko Tuomilehto (J)

University of Helsinki and Department of Medicine, Helsinki University Hospital, Helsinki, Finland.
Department of Public Health, University of Helsinki, Helsinki, Finland.
Diabetes Research Group, King Abdulaziz University, Jeddah, Saudi Arabia.

Jeffrey Seow (J)

Department of Infectious Diseases, School of Immunology & Microbial Sciences, King's College London, London, UK.

Carl Graham (C)

Department of Infectious Diseases, School of Immunology & Microbial Sciences, King's College London, London, UK.

Isabella Huettner (I)

Department of Infectious Diseases, School of Immunology & Microbial Sciences, King's College London, London, UK.

Sam Acors (S)

Department of Infectious Diseases, School of Immunology & Microbial Sciences, King's College London, London, UK.

Neophytos Kouphou (N)

Department of Infectious Diseases, School of Immunology & Microbial Sciences, King's College London, London, UK.

Samuel Wadge (S)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK.

Emma L Duncan (EL)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK.

Claire J Steves (CJ)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK.

Katie J Doores (KJ)

Department of Infectious Diseases, School of Immunology & Microbial Sciences, King's College London, London, UK.

Michael H Malim (MH)

Department of Infectious Diseases, School of Immunology & Microbial Sciences, King's College London, London, UK.

Francis S Collins (FS)

National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, USA.

Päivi Pajukanta (P)

Department of Human Genetics and Institute for Precision Health, David Geffen School of Medicine at UCLA, Los Angeles, CA, USA.

Michael Boehnke (M)

Department of Biostatistics and Center for Statistical Genetics, School of Public Health, University of Michigan, Ann Arbor, MI, USA.

Heikki A Koistinen (HA)

University of Helsinki and Department of Medicine, Helsinki University Hospital, Helsinki, Finland.
Department of Public Health and Welfare, Finnish Institute for Health and Welfare, Helsinki, Finland.
Minerva Foundation Institute for Medical Research, Helsinki, Finland.

Markku Laakso (M)

Department of Medicine, University of Eastern Finland, Kuopio, Finland.
Kuopio University Hospital, Kuopio, Finland.

Mario Falchi (M)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK.

Jordana T Bell (JT)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK.

Laura J Scott (LJ)

Department of Biostatistics and Center for Statistical Genetics, School of Public Health, University of Michigan, Ann Arbor, MI, USA.

Karen L Mohlke (KL)

Department of Genetics, University of North Carolina, Chapel Hill, NC, USA.

Kerrin S Small (KS)

Department of Twin Research and Genetic Epidemiology, King's College London, London, UK. kerrin.small@kcl.ac.uk.

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