Natural History of Mitral Annular Calcification and Calcific Mitral Valve Disease.


Journal

Journal of the American Society of Echocardiography : official publication of the American Society of Echocardiography
ISSN: 1097-6795
Titre abrégé: J Am Soc Echocardiogr
Pays: United States
ID NLM: 8801388

Informations de publication

Date de publication:
09 2022
Historique:
received: 26 08 2021
revised: 05 03 2022
accepted: 10 05 2022
pubmed: 27 5 2022
medline: 9 9 2022
entrez: 26 5 2022
Statut: ppublish

Résumé

The natural history of mitral annular calcification (MAC) and risk for developing calcific mitral valve disease (CMVD) have been poorly defined. The aim of this study was to evaluate the progression rate of MAC and of the development of CMVD. Patients with MAC and paired echocardiograms ≥1 year apart between 2005 and 2019 were included. Progression rates from mild or moderate to severe MAC and to CMVD (defined as severe MAC and significant mitral stenosis and/or regurgitation) were assessed, along with potential association with sex. A total of 11,605 patients (mean age, 73 ± 10 years; 51% men) with MAC (78% mild, 17% moderate, 5% severe) were included and underwent follow-up echocardiography at 4.2 ± 2.7 years. Among patients with mild or moderate MAC, 33% presented with severe MAC at 10 years. The rate of severe MAC was higher in women than in men (41% vs 24% [P < .001]; hazard ratio, 1.3; P < .001) and in patients with moderate versus mild MAC (71% vs 22% [P < .001]; hazard ratio, 6.1; P < .001). At 10 years, 10% presented with CMVD (4%, 23%, and 60% in patients with mild, moderate, and severe MAC, respectively), which was predicted by female sex (15% vs 5%; P < .0001), even after adjustment for MAC severity (hazard ratio, 1.9; P < .001). In this large cohort of patients with MAC, progression to severe MAC was common and frequently resulted in CMVD. Female sex was associated with higher progression rates. MAC and CMVD are expected to dramatically increase as the population ages, highlighting the importance of a better understanding of the pathophysiology of MAC to develop effective preventive medical therapies.

Sections du résumé

BACKGROUND
The natural history of mitral annular calcification (MAC) and risk for developing calcific mitral valve disease (CMVD) have been poorly defined. The aim of this study was to evaluate the progression rate of MAC and of the development of CMVD.
METHODS
Patients with MAC and paired echocardiograms ≥1 year apart between 2005 and 2019 were included. Progression rates from mild or moderate to severe MAC and to CMVD (defined as severe MAC and significant mitral stenosis and/or regurgitation) were assessed, along with potential association with sex.
RESULTS
A total of 11,605 patients (mean age, 73 ± 10 years; 51% men) with MAC (78% mild, 17% moderate, 5% severe) were included and underwent follow-up echocardiography at 4.2 ± 2.7 years. Among patients with mild or moderate MAC, 33% presented with severe MAC at 10 years. The rate of severe MAC was higher in women than in men (41% vs 24% [P < .001]; hazard ratio, 1.3; P < .001) and in patients with moderate versus mild MAC (71% vs 22% [P < .001]; hazard ratio, 6.1; P < .001). At 10 years, 10% presented with CMVD (4%, 23%, and 60% in patients with mild, moderate, and severe MAC, respectively), which was predicted by female sex (15% vs 5%; P < .0001), even after adjustment for MAC severity (hazard ratio, 1.9; P < .001).
CONCLUSION
In this large cohort of patients with MAC, progression to severe MAC was common and frequently resulted in CMVD. Female sex was associated with higher progression rates. MAC and CMVD are expected to dramatically increase as the population ages, highlighting the importance of a better understanding of the pathophysiology of MAC to develop effective preventive medical therapies.

Identifiants

pubmed: 35618253
pii: S0894-7317(22)00255-3
doi: 10.1016/j.echo.2022.05.007
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

925-932

Informations de copyright

Copyright © 2022 American Society of Echocardiography. Published by Elsevier Inc. All rights reserved.

Auteurs

Nadav Willner (N)

Department of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Ian G Burwash (IG)

Department of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Luc Beauchesne (L)

Department of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Vince Chan (V)

Department of Cardiac Surgery, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Branka Vulesevic (B)

Department of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Kathy Ascah (K)

Department of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Thais Coutinho (T)

Department of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Steve Promislow (S)

Department of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Ellamae Stadnick (E)

Department of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Kwan L Chan (KL)

Department of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

Thierry Mesana (T)

Department of Cardiac Surgery, University of Ottawa Heart Institute, Ottawa, Ontario, Canada.

David Messika-Zeitoun (D)

Department of Cardiology, University of Ottawa Heart Institute, Ottawa, Ontario, Canada. Electronic address: dmessika-zeitoun@ottawaheart.ca.

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