Effectiveness of 10 and 13-valent pneumococcal conjugate vaccines against invasive pneumococcal disease in European children: SpIDnet observational multicentre study.

10-valent pneumococcal vaccine 13-valent pneumococcal vaccine Invasive pneumococcal disease Pneumococcal Infections Serotype Streptococcus pneumoniae

Journal

Vaccine
ISSN: 1873-2518
Titre abrégé: Vaccine
Pays: Netherlands
ID NLM: 8406899

Informations de publication

Date de publication:
23 06 2022
Historique:
received: 03 11 2021
revised: 19 04 2022
accepted: 04 05 2022
pubmed: 1 6 2022
medline: 22 6 2022
entrez: 31 5 2022
Statut: ppublish

Résumé

Pneumococcal conjugate vaccines covering 10 (PCV10) and 13 (PCV13) serotypes have been introduced in the infant immunization schedule of most European countries in 2010-11. To provide additional real-life data, we measured the effectiveness of PCV10 and PCV13 against invasive pneumococcal disease (IPD) in children of 12 European sites (SpIDnet). We compared the vaccination status of PCV10 and PCV13 serotype IPD (cases) to that of nonPCV13 serotype IPD (controls) reported in 2012-2018. We calculated pooled effectiveness as (1-vaccination odds ratio)*100, and measured effectiveness over time since booster dose. The PCV13 and PCV10 studies included 2522 IPD cases from ten sites and 486 cases from four sites, respectively. The effectiveness of ≥ 1 PCV13 dose was 84.2% (95 %CI: 79.0-88.1) against PCV13 serotypes (n = 2353) and decreased from 93.1% (87.8-96.1) < 12 months to 85.1% (72.0-92.1) ≥ 24 months after booster dose. PCV13 effectiveness of ≥ 1 dose was 84.7% (55.7-94.7) against fatal PCV13 IPD, 64.5% (43.7-77.6), 83.2% (73.7-89.3) and 85.1% (67.6-93.1) against top serotypes 3, 19A and 1, respectively, and 85.4% (62.3-94.4) against 6C. Serotype 3 and 19A effectiveness declined more rapidly. PCV10 effectiveness of ≥ 1 dose was 84.8% (69.4-92.5) against PCV10 serotypes (n = 370), 27.2% (-187.6 to 81.6) and 85.3% (35.2-96.7) against top serotypes 1 and 7F, 32.5% (-28.3 to 64.5) and -14.4% (-526.5 to 79.1) against vaccine-related serotypes 19A and 6C, respectively. PCV10 and PCV13 provide similar protection against IPD due to the respective vaccine serotype groups but serotype-specific effectiveness varies by serotype and vaccine. PCV13 provided individual protection against serotype 3 and vaccine-related serotype 6C IPD. PCV10 effectiveness was not significant against vaccine-related serotypes 19A and 6C. PCV13 effectiveness declined with time after booster vaccination. This multinational study enabled measuring serotype-specific vaccine effectiveness with a precision rarely possible at the national level. Such large networks are crucial for the post-licensure evaluation of vaccines.

Sections du résumé

BACKGROUND
Pneumococcal conjugate vaccines covering 10 (PCV10) and 13 (PCV13) serotypes have been introduced in the infant immunization schedule of most European countries in 2010-11. To provide additional real-life data, we measured the effectiveness of PCV10 and PCV13 against invasive pneumococcal disease (IPD) in children of 12 European sites (SpIDnet).
METHODS
We compared the vaccination status of PCV10 and PCV13 serotype IPD (cases) to that of nonPCV13 serotype IPD (controls) reported in 2012-2018. We calculated pooled effectiveness as (1-vaccination odds ratio)*100, and measured effectiveness over time since booster dose.
RESULTS
The PCV13 and PCV10 studies included 2522 IPD cases from ten sites and 486 cases from four sites, respectively. The effectiveness of ≥ 1 PCV13 dose was 84.2% (95 %CI: 79.0-88.1) against PCV13 serotypes (n = 2353) and decreased from 93.1% (87.8-96.1) < 12 months to 85.1% (72.0-92.1) ≥ 24 months after booster dose. PCV13 effectiveness of ≥ 1 dose was 84.7% (55.7-94.7) against fatal PCV13 IPD, 64.5% (43.7-77.6), 83.2% (73.7-89.3) and 85.1% (67.6-93.1) against top serotypes 3, 19A and 1, respectively, and 85.4% (62.3-94.4) against 6C. Serotype 3 and 19A effectiveness declined more rapidly. PCV10 effectiveness of ≥ 1 dose was 84.8% (69.4-92.5) against PCV10 serotypes (n = 370), 27.2% (-187.6 to 81.6) and 85.3% (35.2-96.7) against top serotypes 1 and 7F, 32.5% (-28.3 to 64.5) and -14.4% (-526.5 to 79.1) against vaccine-related serotypes 19A and 6C, respectively.
CONCLUSIONS
PCV10 and PCV13 provide similar protection against IPD due to the respective vaccine serotype groups but serotype-specific effectiveness varies by serotype and vaccine. PCV13 provided individual protection against serotype 3 and vaccine-related serotype 6C IPD. PCV10 effectiveness was not significant against vaccine-related serotypes 19A and 6C. PCV13 effectiveness declined with time after booster vaccination. This multinational study enabled measuring serotype-specific vaccine effectiveness with a precision rarely possible at the national level. Such large networks are crucial for the post-licensure evaluation of vaccines.

Identifiants

pubmed: 35637067
pii: S0264-410X(22)00581-3
doi: 10.1016/j.vaccine.2022.05.011
pii:
doi:

Substances chimiques

Pneumococcal Vaccines 0
Vaccines, Conjugate 0

Types de publication

Journal Article Multicenter Study Observational Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3963-3974

Informations de copyright

Copyright © 2022 Elsevier Ltd. All rights reserved.

Auteurs

Camelia Savulescu (C)

Epiconcept, Paris, France.

Pavla Krizova (P)

National Institute of Public Health, Prague, Czech Republic.

Palle Valentiner-Branth (P)

Statens Serum Institut, Copenhagen, Denmark.

Shamez Ladhani (S)

Public Health England, London, United Kingdom.

Hanna Rinta-Kokko (H)

National Institute for Health and Welfare, Helsinki, Finland.

Corinne Levy (C)

ACTIV, Créteil, France.

Jolita Mereckiene (J)

Health Protection Surveillance Centre Dublin, Ireland.

Mirjam Knol (M)

National Institute for Public Health and the Environment, Bilthoven, the Netherlands.

Brita A Winje (BA)

Norwegian Institute of Public Health, Oslo, Norway.

Pilar Ciruela (P)

Health Agency of Catalunya, Barcelona, Spain; CIBER Epidemiología y Salud Pública, Madrid, Spain.

Sara de Miguel (S)

General Directorate of Public Health Madrid, Spain.

Marcela Guevara (M)

Public Health Institute of Navarra - IdiSNA, Pamplona, Spain; CIBER Epidemiología y Salud Pública, Madrid, Spain.

Laura MacDonald (L)

Public Health Scotland, Glasgow, Scotland, UK.

Jana Kozakova (J)

National Institute of Public Health, Prague, Czech Republic.

Hans-Christian Slotved (HC)

Statens Serum Institut, Copenhagen, Denmark.

Norman K Fry (NK)

Public Health England, London, United Kingdom.

J Pekka Nuorti (J)

National Institute for Health and Welfare, Helsinki, Finland; Tampere University, Tampere, Finland.

Kostas Danis (K)

Santé publique France, the National Public Health Institute, Saint-Maurice, France.

Mary Corcoran (M)

Temple Street Children's University Hospital, Irish Pneumococcal Reference Laboratory, Dublin, Ireland.

Arie van der Ende (A)

Academic Medical Centre, National Reference Laboratory for Bacterial Meningitis, Amsterdam, the Netherlands.

Didrik F Vestrheim (DF)

Norwegian Institute of Public Health, Oslo, Norway.

Carmen Munoz-Almagro (C)

Hospital Sant Joan de Déu, and International University of Catalunya, Barcelona, Spain; CIBER Epidemiología y Salud Pública, Madrid, Spain.

Juan-Carlos Sanz (JC)

General Directorate of Public Health Madrid, Spain.

Jesus Castilla (J)

Public Health Institute of Navarra - IdiSNA, Pamplona, Spain; CIBER Epidemiología y Salud Pública, Madrid, Spain.

Andrew Smith (A)

Bacterial Respiratory Infection Service, Scottish Microbiology Reference Laboratory, Glasgow Royal Infirmary & MVLS, University of Glasgow, Glasgow, Scotland, UK.

Edoardo Colzani (E)

European Centre for Disease Prevention and Control, Stockholm, Sweden.

Lucia Pastore Celentano (L)

European Centre for Disease Prevention and Control, Stockholm, Sweden.

Germaine Hanquet (G)

Epiconcept, Paris, France; Antwerp university, Antwerp, Belgium. Electronic address: ghanquet@skynet.be.

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