Nanomolar affinity of EF-hands in neuronal calcium sensor 1 for bivalent cations Pb2+, Mn2+, and Hg2.

EF-hands calcium fluorescence metal binding neuronal calcium sensor 1 non-physiological metals

Journal

Metallomics : integrated biometal science
ISSN: 1756-591X
Titre abrégé: Metallomics
Pays: England
ID NLM: 101478346

Informations de publication

Date de publication:
08 07 2022
Historique:
received: 04 01 2022
accepted: 31 05 2022
pubmed: 4 6 2022
medline: 12 7 2022
entrez: 3 6 2022
Statut: ppublish

Résumé

Abiogenic metals Pb and Hg are highly toxic since chronic and/or acute exposure often leads to severe neuropathologies. Mn2+ is an essential metal ion but in excess can impair neuronal function. In this study, we address in vitro the interactions between neuronal calcium sensor 1 (NCS1) and divalent cations. Results showed that non-physiological ions (Pb2+ and Mn2+) bind to EF-hands in NCS1 with nanomolar affinity and lower equilibrium dissociation constant than the physiological Ca2+ ion. (Kd, Pb2+ = 7.0 ± 1.0 nM; Kd, Mn2+ = 34.0 ± 6.0 nM; K). Native ultra-high resolution mass spectrometry (FT-ICR MS) and trapped ion mobility spectrometry-mass spectrometry (nESI-TIMS-MS) studies provided the NCS1-metal complex compositions-up to four Ca2+ or Mn2+ ions and three Pb2+ ions (M⋅Pb1-3Ca1-3, M⋅Mn1-4Ca1-2, and M⋅Ca1-4) were observed in complex-and similarity across the mobility profiles suggests that the overall native structure is preserved regardless of the number and type of cations. However, the non-physiological metal ions (Pb2+, Mn2+, and Hg2+) binding to NCS1 leads to more efficient quenching of Trp emission and a decrease in W30 and W103 solvent exposure compared to the apo and Ca2+ bound form, although the secondary structural rearrangement and exposure of hydrophobic sites are analogous to those for Ca2+ bound protein. Only Pb2+ and Hg2+ binding to EF-hands leads to the NCS1 dimerization whereas Mn2+ bound NCS1 remains in the monomeric form, suggesting that other factors in addition to metal ion coordination, are required for protein dimerization.

Identifiants

pubmed: 35657675
pii: 6601456
doi: 10.1093/mtomcs/mfac039
pmc: PMC9611292
pii:
doi:

Substances chimiques

Cations, Divalent 0
Neuronal Calcium-Sensor Proteins 0
Neuropeptides 0
frequenin calcium sensor proteins 0
Lead 2P299V784P
Manganese 42Z2K6ZL8P
Calcium SY7Q814VUP

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : NIGMS NIH HHS
ID : R01 GM134247
Pays : United States

Informations de copyright

© The Author(s) 2022. Published by Oxford University Press.

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Auteurs

Md Shofiul Alam (MS)

Department of Chemistry and Biochemistry, Florida International University, Miami, FL 33199, USA.

Samiol Azam (S)

Department of Chemistry and Biochemistry, Florida International University, Miami, FL 33199, USA.

Khoa Pham (K)

Department of Chemistry and Biochemistry, Florida International University, Miami, FL 33199, USA.

Dennys Leyva (D)

Department of Chemistry and Biochemistry, Florida International University, Miami, FL 33199, USA.

Kevin Jeanne Dit Fouque (KJD)

Department of Chemistry and Biochemistry, Florida International University, Miami, FL 33199, USA.
Biomolecular Sciences Institute, Florida International University, Miami 33199, USA.

Francisco Fernandez-Lima (F)

Department of Chemistry and Biochemistry, Florida International University, Miami, FL 33199, USA.
Biomolecular Sciences Institute, Florida International University, Miami 33199, USA.

Jaroslava Miksovska (J)

Department of Chemistry and Biochemistry, Florida International University, Miami, FL 33199, USA.
Biomolecular Sciences Institute, Florida International University, Miami 33199, USA.

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