Effects of Short-term Fasting on Ghrelin/GH/IGF-1 Axis in Healthy Humans: The Role of Ghrelin in the Thrifty Phenotype.


Journal

The Journal of clinical endocrinology and metabolism
ISSN: 1945-7197
Titre abrégé: J Clin Endocrinol Metab
Pays: United States
ID NLM: 0375362

Informations de publication

Date de publication:
18 08 2022
Historique:
received: 14 02 2022
pubmed: 10 6 2022
medline: 23 8 2022
entrez: 9 6 2022
Statut: ppublish

Résumé

A greater decrease in 24-hour energy expenditure (24hEE) during short-term fasting is indicative of a thrifty phenotype. As ghrelin and the growth hormone (GH)/insulin-like growth factor 1 (IGF-1) axis are implicated in the regulation of energy intake and metabolism, we investigated whether ghrelin, GH, and IGF-1 concentrations mediate the fasting-induced decrease in 24hEE that characterizes thriftiness. In 47 healthy individuals, 24hEE was measured in a whole-room indirect calorimeter both during 24-hour eucaloric and fasting conditions. Plasma total ghrelin, GH, and IGF-1 concentrations were measured by enzyme-linked immunosorbent assay after an overnight fast the morning before and after each 24-hour session. During 24-hour fasting, on average 24hEE decreased by 8.0% (P < .001), GH increased by ~5-fold (P < .001), whereas ghrelin (mean +23 pg/mL) and IGF-1 were unchanged (both P ≥ .19) despite a large interindividual variability in ghrelin change (SD 150 pg/mL). Greater fasting-induced increase in ghrelin was associated with a greater decrease in 24hEE during 24-hour fasting (r = -0.42, P = .003), such that individuals who increased ghrelin by 200 pg/mL showed an average decrease in 24hEE by 55 kcal/day. Short-term fasting induced selective changes in the ghrelin/GH/IGF-1 axis, specifically a ghrelin-independent GH hypersecretion that did not translate into increased IGF-1 concentrations. Greater increase in ghrelin after 24-hour fasting was associated with greater decrease in 24hEE, indicating ghrelin as a novel biomarker of increased energy efficiency of the thrifty phenotype.

Identifiants

pubmed: 35678263
pii: 6604640
doi: 10.1210/clinem/dgac353
pmc: PMC9387714
doi:

Substances chimiques

Ghrelin 0
IGF1 protein, human 0
Human Growth Hormone 12629-01-5
Insulin-Like Growth Factor I 67763-96-6
Growth Hormone 9002-72-6

Banques de données

ClinicalTrials.gov
['NCT00523627']

Types de publication

Journal Article Research Support, Non-U.S. Gov't Research Support, N.I.H., Intramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

e3769-e3780

Informations de copyright

Published by Oxford University Press on behalf of the Endocrine Society 2022.

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Auteurs

Tim Hollstein (T)

Phoenix Epidemiology and Clinical Research Branch, Phoenix, AZ 85016, USA.
Institute of Diabetes and Clinical Metabolic Research, 24195 Kiel, Germany.

Alessio Basolo (A)

Phoenix Epidemiology and Clinical Research Branch, Phoenix, AZ 85016, USA.

Yigit Unlu (Y)

Phoenix Epidemiology and Clinical Research Branch, Phoenix, AZ 85016, USA.

Takafumi Ando (T)

Phoenix Epidemiology and Clinical Research Branch, Phoenix, AZ 85016, USA.

Mary Walter (M)

Clinical Core Lab, National Institute of Diabetes and Digestive and Kidney Diseases, Bethesda, MD 20892, USA.

Jonathan Krakoff (J)

Phoenix Epidemiology and Clinical Research Branch, Phoenix, AZ 85016, USA.

Paolo Piaggi (P)

Phoenix Epidemiology and Clinical Research Branch, Phoenix, AZ 85016, USA.
Department of Information Engineering, University of Pisa, Pisa, Italy.

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Classifications MeSH