Co-transport of the nuclear-encoded Cox7c mRNA with mitochondria along axons occurs through a coding-region-dependent mechanism.


Journal

Journal of cell science
ISSN: 1477-9137
Titre abrégé: J Cell Sci
Pays: England
ID NLM: 0052457

Informations de publication

Date de publication:
15 08 2022
Historique:
received: 20 10 2021
accepted: 07 07 2022
pubmed: 15 7 2022
medline: 19 8 2022
entrez: 14 7 2022
Statut: ppublish

Résumé

Nuclear-encoded mitochondrial protein mRNAs have been found to be localized and locally translated within neuronal processes. However, the mechanism of transport for those mRNAs to distal locations is not fully understood. Here, we describe axonal co-transport of Cox7c with mitochondria. Fractionation analysis and single-molecule fluorescence in situ hybridization (smFISH) assay revealed that endogenous mRNA encoding Cox7c was preferentially associated with mitochondria in a mouse neuronal cell line and within mouse primary motor neuron axons, whereas other mRNAs that do not encode mitochondrial protein were much less associated. Live-cell imaging of MS2-tagged Cox7c mRNA further confirmed the preferential colocalization and co-transport of Cox7c mRNA with mitochondria in motor neuron axons. Intriguingly, the coding region, rather than the 3' untranslated region (UTR), was the key domain for the co-transport. Our results reveal that Cox7c mRNA can be transported with mitochondria along significant distances and that its coding region is a major recognition feature. This is consistent with the idea that mitochondria can play a vital role in spatial regulation of the axonal transcriptome at distant neuronal sites.

Identifiants

pubmed: 35833493
pii: 276008
doi: 10.1242/jcs.259436
pmc: PMC9481926
pii:
doi:

Substances chimiques

3' Untranslated Regions 0
Mitochondrial Proteins 0
RNA, Messenger 0
Electron Transport Complex IV EC 1.9.3.1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Israel Science Foundation
ID : 1096/13
Organisme : Adelis Brain research
ID : 2023478
Organisme : Adelis Brain Research
ID : 2023478
Organisme : Technion-Israel Institute of Technology

Informations de copyright

© 2022. Published by The Company of Biologists Ltd.

Déclaration de conflit d'intérêts

Competing interests The authors declare no competing or financial interests.

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Auteurs

Bar Cohen (B)

Faculty of Biology, Technion - Israel Institute of Technology, Haifa 3200003, Israel.

Topaz Altman (T)

Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv 69978, Israel.

Adi Golani-Armon (A)

Faculty of Biology, Technion - Israel Institute of Technology, Haifa 3200003, Israel.
Faculty of Nanosciences and Nanoengineering, Technion - Israel Institute of Technology, Haifa 3200003, Israel.

Anca F Savulescu (AF)

Division of Chemical, Systems & Synthetic Biology, Department of Integrative Biomedical Sciences, Faculty of Health Sciences, Institute of Infectious Disease & Molecular Medicine, University of Cape Town, Cape Town 7925, South Africa.

Amjd Ibraheem (A)

Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv 69978, Israel.

Musa M Mhlanga (MM)

Radboud Institute for Molecular Life Sciences (RIMLS), Radboud University Medical Center, 6525 GA Nijmegen, The Netherlands.
Epigenomics & Single Cell Biophysics Group, Department of Cell Biology, FNWI, Radboud University, 6525 GA Nijmegen, The Netherlands.
Department of Human Genetics, Radboud University Medical Center, 6525 GA Nijmegen, The Netherlands.

Eran Perlson (E)

Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv 69978, Israel.

Yoav S Arava (YS)

Faculty of Biology, Technion - Israel Institute of Technology, Haifa 3200003, Israel.

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Classifications MeSH