Efficacy and safety of antiseizure medication in post-stroke epilepsy.


Journal

Seizure
ISSN: 1532-2688
Titre abrégé: Seizure
Pays: England
ID NLM: 9306979

Informations de publication

Date de publication:
Aug 2022
Historique:
received: 29 03 2022
revised: 03 07 2022
accepted: 05 07 2022
pubmed: 15 7 2022
medline: 4 8 2022
entrez: 14 7 2022
Statut: ppublish

Résumé

Specific antiseizure medications (ASM) would improve the outcome in post-stroke epilepsy (PSE). The aim of this multicenter observational study was to compare different antiseizure monotherapies in PSE. We collected the data from 207 patients with PSE who did not change their initial antiseizure monotherapy during the period of 12 months. Efficacy was assessed by a standardized three month seizure frequency and seizure freedom. Safety was estimated by the reported side effects. The mean three month seizure frequency was 1.9 ± 3.1 on eslicarbazepine, 2.1 ± 3.2 on lacosamide, 3.4 ± 4.4 on levetiracetam, 4.3 ± 6.8 on lamotrigine, and 5.1 ± 7.3 on valproate (p < 0.05 for eslicarbazepine or lacosamide in comparison with levetiracetam, lamotrigine and valproate, respectively). The lowest seizure frequency and the highest seizure freedom was observed on ASMs acting via the slow inactivation of sodium channels in comparison to other mechanisms of action (0.7 ± 0.9 vs 2.2 ± 2.4, p < 0.01). Among side effects, the most frequently reported were vertigo (25%) and tiredness (15.9%). They were similar in all investigated groups of ASM. The independent factors increasing seizure frequency that were identified in multiple regression analyses were increased size of infarction, cortical involvement, hemorrhagic transformation, neurological deficits at admission and functional impairment. Administration of ASM with the mechanism of action via the slow inactivation of sodium channels was an independent factor decreasing the seizure frequency. Our data show that antiseizure medications acting via the slow inactivation of sodium channels, such as lacosamide and eslicarbazepine, are well tolerated and might be associated with better seizure control in PSE.

Sections du résumé

BACKGROUND BACKGROUND
Specific antiseizure medications (ASM) would improve the outcome in post-stroke epilepsy (PSE). The aim of this multicenter observational study was to compare different antiseizure monotherapies in PSE.
METHODS METHODS
We collected the data from 207 patients with PSE who did not change their initial antiseizure monotherapy during the period of 12 months. Efficacy was assessed by a standardized three month seizure frequency and seizure freedom. Safety was estimated by the reported side effects.
RESULTS RESULTS
The mean three month seizure frequency was 1.9 ± 3.1 on eslicarbazepine, 2.1 ± 3.2 on lacosamide, 3.4 ± 4.4 on levetiracetam, 4.3 ± 6.8 on lamotrigine, and 5.1 ± 7.3 on valproate (p < 0.05 for eslicarbazepine or lacosamide in comparison with levetiracetam, lamotrigine and valproate, respectively). The lowest seizure frequency and the highest seizure freedom was observed on ASMs acting via the slow inactivation of sodium channels in comparison to other mechanisms of action (0.7 ± 0.9 vs 2.2 ± 2.4, p < 0.01). Among side effects, the most frequently reported were vertigo (25%) and tiredness (15.9%). They were similar in all investigated groups of ASM. The independent factors increasing seizure frequency that were identified in multiple regression analyses were increased size of infarction, cortical involvement, hemorrhagic transformation, neurological deficits at admission and functional impairment. Administration of ASM with the mechanism of action via the slow inactivation of sodium channels was an independent factor decreasing the seizure frequency.
CONCLUSION CONCLUSIONS
Our data show that antiseizure medications acting via the slow inactivation of sodium channels, such as lacosamide and eslicarbazepine, are well tolerated and might be associated with better seizure control in PSE.

Identifiants

pubmed: 35834881
pii: S1059-1311(22)00156-X
doi: 10.1016/j.seizure.2022.07.003
pii:
doi:

Substances chimiques

Anticonvulsants 0
Sodium Channels 0
Levetiracetam 44YRR34555
Lacosamide 563KS2PQY5
Valproic Acid 614OI1Z5WI
Lamotrigine U3H27498KS

Types de publication

Journal Article Multicenter Study Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

109-114

Informations de copyright

Copyright © 2022 British Epilepsy Association. Published by Elsevier Ltd. All rights reserved.

Auteurs

Yaroslav Winter (Y)

Mainz Comprehensive Epilepsy and Sleep Medicine Center, Department of Neurology, University Medical Center of the Johannes Gutenberg University Mainz, Langenbeckstr.1, Mainz 55131, Germany; Department of Neurology, Philipps-University Marburg, Baldingerstr, Marburg 35043, Germany. Electronic address: yaroslav.winter@unimedizin-mainz.de.

Timo Uphaus (T)

Department of Neurology, Focus Program Translational Neuroscience (FTN), Rhine Main Neuroscience Network, University Medical Center of the Johannes Gutenberg University Mainz, Langenbeckstr.1, Mainz 55131, Germany.

Katharina Sandner (K)

Mainz Comprehensive Epilepsy and Sleep Medicine Center, Department of Neurology, University Medical Center of the Johannes Gutenberg University Mainz, Langenbeckstr.1, Mainz 55131, Germany.

Sven Klimpe (S)

Specialized Epilepsy Practice, Ringstraße 6, Nierstein 55283, Germany.

Sebastian von Stuckrad-Barre (SV)

Specialized Epilepsy Practice, Bahnhofstraße 26A, Ingelheim am Rhein 55218, Germany.

Sergiu Groppa (S)

Department of Neurology, Focus Program Translational Neuroscience (FTN), Rhine Main Neuroscience Network, University Medical Center of the Johannes Gutenberg University Mainz, Langenbeckstr.1, Mainz 55131, Germany.

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Classifications MeSH