P2Y12 reaction units and ischemic and bleeding events after neuro-endovascular treatment.


Journal

Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association
ISSN: 1532-8511
Titre abrégé: J Stroke Cerebrovasc Dis
Pays: United States
ID NLM: 9111633

Informations de publication

Date de publication:
Sep 2022
Historique:
received: 24 05 2022
revised: 20 06 2022
accepted: 30 06 2022
pubmed: 20 7 2022
medline: 25 8 2022
entrez: 19 7 2022
Statut: ppublish

Résumé

To investigate the associations of perioperative P2Y12 reaction units (PRU) measured using VerifyNow with ischemic and bleeding events, and to determine the PRU threshold in the setting of elective neuro-endovascular treatment (EVT) for intracranial/extracranial vascular disease in patients taking aspirin and clopidogrel. Of the patients undergoing elective neuro-EVT while taking aspirin and clopidogrel, those taking both antiplatelet agents for 7 days or more and whose PRU and aspirin reaction units (ARU) were measured were included. The primary and safety outcomes were defined as symptomatic ischemic and major bleeding events within 30 days after EVT. A total of 197 patients were available for the analyses. Higher PRU was associated with symptomatic ischemic events on multivariable logistic analysis (odds ratio per 10 increase 1.14 [95% confidence interval 1.03-1.27], p=0.011). Receiver operating characteristic curve analysis showed that PRU ≥212 was the threshold to predict symptomatic ischemic events (area under the curve=0.73; sensitivity, 62.5%; specificity, 82.0%). Lower PRU was also associated with major bleeding events (odds ratio per 10 increase 0.87 [0.78-0.96], p=0.004), and the threshold to predict major bleeding events was PRU ≤46 (area under the curve=0.76; sensitivity, 70.0%; specificity, 87.2%) CONCLUSIONS: The PRU value was associated with symptomatic ischemic and major bleeding events after elective neuro-EVT in patients taking aspirin and clopidogrel. PRU ≥212 and PRU ≤46 appeared to be the threshold values to predict symptomatic ischemic and major bleeding events, respectively.

Sections du résumé

BACKGROUND AND PURPOSE OBJECTIVE
To investigate the associations of perioperative P2Y12 reaction units (PRU) measured using VerifyNow with ischemic and bleeding events, and to determine the PRU threshold in the setting of elective neuro-endovascular treatment (EVT) for intracranial/extracranial vascular disease in patients taking aspirin and clopidogrel.
METHODS METHODS
Of the patients undergoing elective neuro-EVT while taking aspirin and clopidogrel, those taking both antiplatelet agents for 7 days or more and whose PRU and aspirin reaction units (ARU) were measured were included. The primary and safety outcomes were defined as symptomatic ischemic and major bleeding events within 30 days after EVT.
RESULTS RESULTS
A total of 197 patients were available for the analyses. Higher PRU was associated with symptomatic ischemic events on multivariable logistic analysis (odds ratio per 10 increase 1.14 [95% confidence interval 1.03-1.27], p=0.011). Receiver operating characteristic curve analysis showed that PRU ≥212 was the threshold to predict symptomatic ischemic events (area under the curve=0.73; sensitivity, 62.5%; specificity, 82.0%). Lower PRU was also associated with major bleeding events (odds ratio per 10 increase 0.87 [0.78-0.96], p=0.004), and the threshold to predict major bleeding events was PRU ≤46 (area under the curve=0.76; sensitivity, 70.0%; specificity, 87.2%) CONCLUSIONS: The PRU value was associated with symptomatic ischemic and major bleeding events after elective neuro-EVT in patients taking aspirin and clopidogrel. PRU ≥212 and PRU ≤46 appeared to be the threshold values to predict symptomatic ischemic and major bleeding events, respectively.

Identifiants

pubmed: 35849918
pii: S1052-3057(22)00325-1
doi: 10.1016/j.jstrokecerebrovasdis.2022.106631
pii:
doi:

Substances chimiques

Platelet Aggregation Inhibitors 0
Purinergic P2Y Receptor Antagonists 0
Clopidogrel A74586SNO7
Ticlopidine OM90ZUW7M1
Aspirin R16CO5Y76E

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

106631

Informations de copyright

Copyright © 2022 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest None declared.

Auteurs

Kyohei Fujita (K)

Department of Endovascular Surgery, Tokyo Medical and Dental University, Tokyo, Japan; Department of Neurology and Neurological Science, Tokyo Medical and Dental University, Tokyo, Japan.

Shoko Fujii (S)

Department of Endovascular Surgery, Tokyo Medical and Dental University, Tokyo, Japan. Electronic address: fujii.evs@tmd.ac.jp.

Sakyo Hirai (S)

Department of Endovascular Surgery, Tokyo Medical and Dental University, Tokyo, Japan. Electronic address: sakyevs@tmd.ac.jp.

Hiroto Yamaoka (H)

Department of Endovascular Surgery, Tokyo Medical and Dental University, Tokyo, Japan. Electronic address: yamaoka.evs@tmd.ac.jp.

Mariko Ishikawa (M)

Department of Endovascular Surgery, Tokyo Medical and Dental University, Tokyo, Japan. Electronic address: ishikawa.evs@tmd.ac.jp.

Jun Karakama (J)

Department of Neurosurgery, Tokyo Medical and Dental University, Tokyo, Japan. Electronic address: karakama.evs@tmd.ac.jp.

Kazunori Miki (K)

Department of Endovascular Surgery, Tokyo Medical and Dental University, Tokyo, Japan; Department of Endovascular Surgery, Tokyo Metropolitan Police Hospital, Tokyo, Japan.

Masataka Yoshimura (M)

Department of Neurosurgery, Tsuchiura Kyodo General Hospital, Ibaraki, Japan. Electronic address: mstkktsm@yahoo.co.jp.

Shigeru Nemoto (S)

Department of Endovascular Surgery, Tokyo Medical and Dental University, Tokyo, Japan; Department of Neurosurgery, Kanto Rosai Hospital, Kanagawa, Japan. Electronic address: nemoto@sa2.so-net.ne.jp.

Kazutaka Sumita (K)

Department of Endovascular Surgery, Tokyo Medical and Dental University, Tokyo, Japan. Electronic address: sumita.nsrg@tmd.ac.jp.

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Classifications MeSH