Antimicrobial effects of inhaled sphingosine against Pseudomonas aeruginosa in isolated ventilated and perfused pig lungs.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2022
Historique:
received: 28 02 2022
accepted: 04 07 2022
entrez: 21 7 2022
pubmed: 22 7 2022
medline: 26 7 2022
Statut: epublish

Résumé

Ex-vivo lung perfusion (EVLP) is a save way to verify performance of donor lungs prior to implantation. A major problem of lung transplantation is a donor-to-recipient-transmission of bacterial cultures. Thus, a broadspectrum anti-infective treatment with sphingosine in EVLP might be a novel way to prevent such infections. Sphingosine inhalation might provide a reliable anti-infective treatment option in EVLP. Here, antimicrobial potency of inhalative sphingosine in an infection EVLP model was tested. A 3-hour EVLP run using pig lungs was performed. Bacterial infection was initiated 1-hour before sphingosine inhalation. Biopsies were obtained 60 and 120 min after infection with Pseudomonas aeruginosa. Aliquots of broncho-alveolar lavage (BAL) before and after inhalation of sphingosine were plated and counted, tissue samples were fixed in paraformaldehyde, embedded in paraffin and sectioned. Immunostainings were performed. Sphingosine inhalation in the setting of EVLP rapidly resulted in a 6-fold decrease of P. aeruginosa CFU in the lung (p = 0.016). We did not observe any negative side effects of sphingosine. Inhalation of sphingosine induced a significant decrease of Pseudomonas aeruginosa at the epithelial layer of tracheal and bronchial cells. The inhalation has no local side effects in ex-vivo perfused and ventilated pig lungs.

Sections du résumé

BACKGROUND
Ex-vivo lung perfusion (EVLP) is a save way to verify performance of donor lungs prior to implantation. A major problem of lung transplantation is a donor-to-recipient-transmission of bacterial cultures. Thus, a broadspectrum anti-infective treatment with sphingosine in EVLP might be a novel way to prevent such infections. Sphingosine inhalation might provide a reliable anti-infective treatment option in EVLP. Here, antimicrobial potency of inhalative sphingosine in an infection EVLP model was tested.
METHODS
A 3-hour EVLP run using pig lungs was performed. Bacterial infection was initiated 1-hour before sphingosine inhalation. Biopsies were obtained 60 and 120 min after infection with Pseudomonas aeruginosa. Aliquots of broncho-alveolar lavage (BAL) before and after inhalation of sphingosine were plated and counted, tissue samples were fixed in paraformaldehyde, embedded in paraffin and sectioned. Immunostainings were performed.
RESULTS
Sphingosine inhalation in the setting of EVLP rapidly resulted in a 6-fold decrease of P. aeruginosa CFU in the lung (p = 0.016). We did not observe any negative side effects of sphingosine.
CONCLUSION
Inhalation of sphingosine induced a significant decrease of Pseudomonas aeruginosa at the epithelial layer of tracheal and bronchial cells. The inhalation has no local side effects in ex-vivo perfused and ventilated pig lungs.

Identifiants

pubmed: 35862397
doi: 10.1371/journal.pone.0271620
pii: PONE-D-22-06001
pmc: PMC9302828
doi:

Substances chimiques

Anti-Infective Agents 0
Sphingosine NGZ37HRE42

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0271620

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Henning Carstens (H)

Department of Thoracic and Cardiovascular Surgery, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.
Cardiac Surgery for Congenital Heart Disease, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.

Katharina Kalka (K)

Department of Thoracic and Cardiovascular Surgery, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.

Rabea Verhaegh (R)

Institute of Molecular Biology, University of Duisburg-Essen, Essen, Germany.

Fabian Schumacher (F)

Institute of Pharmacy, Freie Universität Berlin, Berlin, Germany.

Matthias Soddemann (M)

Institute of Molecular Biology, University of Duisburg-Essen, Essen, Germany.

Barbara Wilker (B)

Institute of Molecular Biology, University of Duisburg-Essen, Essen, Germany.

Simone Keitsch (S)

Institute of Molecular Biology, University of Duisburg-Essen, Essen, Germany.

Carolin Sehl (C)

Institute of Molecular Biology, University of Duisburg-Essen, Essen, Germany.

Burkhard Kleuser (B)

Institute of Pharmacy, Freie Universität Berlin, Berlin, Germany.

Michael Hübler (M)

Cardiac Surgery for Congenital Heart Disease, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.

Ursula Rauen (U)

Institute of Biochemistry, University of Duisburg-Essen, Essen, Germany.

Anne Katrin Becker (AK)

Institute of Molecular Biology, University of Duisburg-Essen, Essen, Germany.

Achim Koch (A)

Department of Thoracic and Cardiovascular Surgery, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.

Erich Gulbins (E)

Institute of Molecular Biology, University of Duisburg-Essen, Essen, Germany.
Department of Surgery, University of Cincinnati, Medical School, Cincinnati, OH, United States of America.

Markus Kamler (M)

Department of Thoracic and Cardiovascular Surgery, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.

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Classifications MeSH