The Effect of Angiotensin Converting Enzyme (ACE) I/D Polymorphism on Atherosclerotic Cardiovascular Disease and Cardiovascular Mortality Risk in Non-Hemodialyzed Chronic Kidney Disease: The Mediating Role of Plasma ACE Level.


Journal

Genes
ISSN: 2073-4425
Titre abrégé: Genes (Basel)
Pays: Switzerland
ID NLM: 101551097

Informations de publication

Date de publication:
23 06 2022
Historique:
received: 14 05 2022
revised: 18 06 2022
accepted: 20 06 2022
entrez: 27 7 2022
pubmed: 28 7 2022
medline: 29 7 2022
Statut: epublish

Résumé

The association between angiotensin-converting enzyme insertion/deletion (ACE I/D) polymorphisms and plasma ACE levels may allow for the optimization of a preventive intervention to reduce cardiovascular morbidity and mortality in the chronic kidney disease (CKD) population. In this study, we aimed to analyze the association between ACE I/D polymorphism and cardiovascular mortality risk among non-hemodialyzed chronic kidney disease patients. This cross-sectional study examined 70 patients of Javanese ethnic origin with stable CKD who did not receive hemodialysis. ACE I/D polymorphisms, plasma ACE levels, atherosclerotic cardiovascular disease (ASCVD) risk, and cardiovascular mortality risk were investigated. As per our findings, the I allele was found to be more frequent (78.6) than the D allele (21.4), and the DD genotype was less frequent than the II genotype (4.3 vs. 61.4). The ACE I/D polymorphism had a significant direct positive effect on plasma ACE levels (path coefficient = 0.302,

Identifiants

pubmed: 35885904
pii: genes13071121
doi: 10.3390/genes13071121
pmc: PMC9318243
pii:
doi:

Substances chimiques

ACE protein, human EC 3.4.15.1
Peptidyl-Dipeptidase A EC 3.4.15.1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

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Auteurs

Hendri Susilo (H)

Doctoral Program of Medical Science, Faculty of Medicine, Universitas Airlangga, Surabaya 60132, Indonesia.
Department of Cardiology and Vascular Medicine, Universitas Airlangga Hospital, Surabaya 60115, Indonesia.

Budi Susetyo Pikir (BS)

Department of Cardiology and Vascular Medicine, Universitas Airlangga Hospital, Surabaya 60115, Indonesia.
Department of Cardiology and Vascular Medicine, Faculty of Medicine, Universitas Airlangga, Surabaya 60132, Indonesia.

Mochammad Thaha (M)

Department of Internal Medicine, Faculty of Medicine, Universitas Airlangga, Surabaya 60132, Indonesia.
Department of Internal Medicine, Universitas Airlangga Hospital, Surabaya 60115, Indonesia.

Mochamad Yusuf Alsagaff (MY)

Department of Cardiology and Vascular Medicine, Universitas Airlangga Hospital, Surabaya 60115, Indonesia.
Department of Cardiology and Vascular Medicine, Faculty of Medicine, Universitas Airlangga, Surabaya 60132, Indonesia.

Satriyo Dwi Suryantoro (SD)

Department of Internal Medicine, Faculty of Medicine, Universitas Airlangga, Surabaya 60132, Indonesia.
Department of Internal Medicine, Universitas Airlangga Hospital, Surabaya 60115, Indonesia.

Citrawati Dyah Kencono Wungu (CDK)

Department of Physiology and Medical Biochemistry, Faculty of Medicine, Universitas Airlangga, Surabaya 60132, Indonesia.
Institute of Tropical Disease, Universitas Airlangga, Surabaya 60286, Indonesia.

Ifan Ali Wafa (IA)

Faculty of Medicine, Universitas Airlangga, Surabaya 60132, Indonesia.

Cennikon Pakpahan (C)

Department of Biomedicine, Faculty of Medicine, Universitas Airlangga, Surabaya 60132, Indonesia.

Delvac Oceandy (D)

Division of Cardiovascular Science, Manchester Academic Health Science Centre, University of Manchester, Manchester M13 9PG, UK.

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