Melatonin Reverses the Warburg-Type Metabolism and Reduces Mitochondrial Membrane Potential of Ovarian Cancer Cells Independent of MT1 Receptor Activation.


Journal

Molecules (Basel, Switzerland)
ISSN: 1420-3049
Titre abrégé: Molecules
Pays: Switzerland
ID NLM: 100964009

Informations de publication

Date de publication:
07 Jul 2022
Historique:
received: 29 05 2022
revised: 29 06 2022
accepted: 01 07 2022
entrez: 27 7 2022
pubmed: 28 7 2022
medline: 29 7 2022
Statut: epublish

Résumé

Ovarian cancer (OC) is the most lethal gynecologic malignancy, and melatonin has shown various antitumor properties. Herein, we investigated the influence of melatonin therapy on energy metabolism and mitochondrial integrity in SKOV-3 cells and tested whether its effects depended on MT1 receptor activation. SKOV-3 cells were exposed to different melatonin concentrations, and experimental groups were divided as to the presence of MT1 receptors (melatonin groups) or receptor absence by RNAi silencing (siRNA MT1+melatonin). Intracellular melatonin levels increased after treatment with melatonin independent of the MT1. The mitochondrial membrane potential of SKOV-3 cells decreased in the group treated with the highest melatonin concentration. Melatonin reduced cellular glucose consumption, while MT1 knockdown increased its consumption. Interconversion of lactate to pyruvate increased after treatment with melatonin and was remarkable in siRNA MT1 groups. Moreover, lactate dehydrogenase activity decreased with melatonin and increased after MT1 silencing at all concentrations. The UCSC XenaBrowser tool showed a positive correlation between the human

Identifiants

pubmed: 35889222
pii: molecules27144350
doi: 10.3390/molecules27144350
pmc: PMC9321770
pii:
doi:

Substances chimiques

MTNR1A protein, human 0
Pyruvates 0
RNA, Small Interfering 0
Receptor, Melatonin, MT1 0
Melatonin JL5DK93RCL

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : São Paulo Research Foundation
ID : 2019/00906-6
Organisme : São Paulo Research Foundation
ID : 2018/15797-5
Organisme : National Council for Scientific and Technological Development
ID : 304108/2020-0
Organisme : Coordenação de Aperfeicoamento de Pessoal de Nível Superior
ID : 001

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Auteurs

Maira Smaniotto Cucielo (MS)

Department of Structural and Functional Biology, Institute of Biosciences, UNESP-São Paulo State University, Botucatu 18618-689, São Paulo, Brazil.

Roberta Carvalho Cesário (RC)

Department of Structural and Functional Biology, Institute of Biosciences, UNESP-São Paulo State University, Botucatu 18618-689, São Paulo, Brazil.

Henrique Spaulonci Silveira (HS)

Department of Structural and Functional Biology, Institute of Biosciences, UNESP-São Paulo State University, Botucatu 18618-689, São Paulo, Brazil.

Letícia Barbosa Gaiotte (LB)

Department of Structural and Functional Biology, Institute of Biosciences, UNESP-São Paulo State University, Botucatu 18618-689, São Paulo, Brazil.

Sérgio Alexandre Alcantara Dos Santos (SAA)

Department of Structural and Functional Biology, Institute of Biosciences, UNESP-São Paulo State University, Botucatu 18618-689, São Paulo, Brazil.

Debora Aparecida Pires de Campos Zuccari (DAP)

Cancer Molecular Research Laboratory (LIMC), FAMERP, Department of Molecular Biology, São José do Rio Preto 15090-000, São Paulo, Brazil.

Fábio Rodrigues Ferreira Seiva (FRF)

Biological Science Center, Department of Biology, Luiz Meneghel Campus, Universidade Estadual do Norte do Paraná-UENP, Bandeirantes 86360-000, Paraná, Brazil.

Russel J Reiter (RJ)

Department of Cell Systems and Anatomy, UT Health, San Antonio, TX 78229, USA.

Luiz Gustavo de Almeida Chuffa (LG)

Department of Structural and Functional Biology, Institute of Biosciences, UNESP-São Paulo State University, Botucatu 18618-689, São Paulo, Brazil.

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Classifications MeSH