Combined Blockade Of PD-1 and TIGIT is not Sufficient to Improve the Function Of CD8+ T-Cells in Chronic Lymphocytic Leukemia.


Journal

Asian Pacific journal of cancer prevention : APJCP
ISSN: 2476-762X
Titre abrégé: Asian Pac J Cancer Prev
Pays: Thailand
ID NLM: 101130625

Informations de publication

Date de publication:
01 Jul 2022
Historique:
received: 13 03 2021
entrez: 28 7 2022
pubmed: 29 7 2022
medline: 2 8 2022
Statut: epublish

Résumé

Blockade of immune checkpoint receptors in the treatment of cancers has been mentioned in several studies. Here, we investigated the efficacy of combined blockade of two inhibitory receptors, PD-1 and TIGIT, in restoring functional features of CD8+ T-cells in CLL. CD8+ T-cells were separated from the peripheral blood of 11 CLL patients and targeted with malignant B-cells isolated from the same patients. Cells were then stimulated with anti-CD3/CD28 and PMA/ionomycin to assess their proliferative response and cytotoxic activity using MTT and CD107a degranulation assays, respectively. Cytokine production of isolated CD8+ T-cells was also determined using ELISA. There were no significant differences in proliferation and cytotoxic activity of CD8+ T-cells co-blocked with anti-PD-1/TIGIT compared to those single blocked with anti-PD-1, anti-TIGIT, or the control antibody. There was no significant difference in cytokine production of mentioned groups, either. Collectively, combined blockade of PD-1 and TIGIT failed to restore the proliferation and function of CD8+ T-cells isolated from CLL patients.

Sections du résumé

BACKGROUND AND OBJECTIVE OBJECTIVE
Blockade of immune checkpoint receptors in the treatment of cancers has been mentioned in several studies. Here, we investigated the efficacy of combined blockade of two inhibitory receptors, PD-1 and TIGIT, in restoring functional features of CD8+ T-cells in CLL.
METHODS METHODS
CD8+ T-cells were separated from the peripheral blood of 11 CLL patients and targeted with malignant B-cells isolated from the same patients. Cells were then stimulated with anti-CD3/CD28 and PMA/ionomycin to assess their proliferative response and cytotoxic activity using MTT and CD107a degranulation assays, respectively. Cytokine production of isolated CD8+ T-cells was also determined using ELISA.
RESULTS RESULTS
There were no significant differences in proliferation and cytotoxic activity of CD8+ T-cells co-blocked with anti-PD-1/TIGIT compared to those single blocked with anti-PD-1, anti-TIGIT, or the control antibody. There was no significant difference in cytokine production of mentioned groups, either.
CONCLUSIONS CONCLUSIONS
Collectively, combined blockade of PD-1 and TIGIT failed to restore the proliferation and function of CD8+ T-cells isolated from CLL patients.

Identifiants

pubmed: 35901326
doi: 10.31557/APJCP.2022.23.7.2225
pmc: PMC9727349
pii:
doi:

Substances chimiques

Cytokines 0
PDCD1 protein, human 0
Programmed Cell Death 1 Receptor 0
Receptors, Immunologic 0
TIGIT protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2225-2231

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Auteurs

Fatemeh Hatefi (F)

Department of Immunology, School of Medicine, Mazandaran University of Medical Sciences, Sari, Iran.

Hossein Asgarian-Omran (H)

Department of Immunology, School of Medicine, Mazandaran University of Medical Sciences, Sari, Iran.
Gastrointestinal Cancer Research Center, Mazandaran University of Medical Sciences, Sari, Iran.

Hadi Hossein-Nataj (H)

Department of Immunology, School of Medicine, Mazandaran University of Medical Sciences, Sari, Iran.

Armin Akbar (A)

Department of Immunology, School of Medicine, Mazandaran University of Medical Sciences, Sari, Iran.

Ramin Shekarriz (R)

Gastrointestinal Cancer Research Center, Mazandaran University of Medical Sciences, Sari, Iran.
Department of Hematology and Oncology, Imam Khomeini hospital, Mazandaran university of Medical sciences, Sari, Iran.

Ehsan Zaboli (E)

Gastrointestinal Cancer Research Center, Mazandaran University of Medical Sciences, Sari, Iran.
Department of Hematology and Oncology, Imam Khomeini hospital, Mazandaran university of Medical sciences, Sari, Iran.

Ghasem Janbabai (G)

Gastrointestinal Cancer Research Center, Mazandaran University of Medical Sciences, Sari, Iran.

Mohsen Tehrani (M)

Department of Immunology, School of Medicine, Mazandaran University of Medical Sciences, Sari, Iran.
Gastrointestinal Cancer Research Center, Mazandaran University of Medical Sciences, Sari, Iran.

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