An atypical MAPK regulates translocation of a GATA transcription factor in response to chemoattractant stimulation.

Dictyostelium Chemotaxis Folate GATA transcription factor Kinase translocation reporter MAPK Nuclear-to-cytoplasmic translocation Transcription factor

Journal

Journal of cell science
ISSN: 1477-9137
Titre abrégé: J Cell Sci
Pays: England
ID NLM: 0052457

Informations de publication

Date de publication:
15 08 2022
Historique:
received: 20 04 2022
accepted: 25 07 2022
pubmed: 3 8 2022
medline: 23 8 2022
entrez: 2 8 2022
Statut: ppublish

Résumé

The Dictyostelium atypical mitogen-activated protein kinase (MAPK) Erk2 is required for chemotactic responses to cAMP as amoeba undergo multicellular development. In this study, Erk2 was found to be essential for the cAMP-stimulated translocation of the GATA transcription factor GtaC as indicated by the distribution of a GFP-GtaC reporter. Erk2 was also found to be essential for the translocation of GtaC in response to external folate, a foraging signal that directs the chemotaxis of amoeba to bacteria. Erk1, the only other Dictyostelium MAPK, was not required for the GtaC translocation to either chemoattractant, indicating that GFP-GtaC is a kinase translocation reporter specific for atypical MAPKs. The translocation of GFP-GtaC in response to folate was absent in mutants lacking the folate receptor Far1 or the coupled G-protein subunit Gα4. Loss of GtaC function resulted in enhanced chemotactic movement to folate, suggesting that GtaC suppresses responses to folate. The alteration of four Erk2-preferred phosphorylation sites in GtaC impacted the translocation of GFP-GtaC in response to folate and the GFP-GtaC-mediated rescue of aggregation and development of gtaC- cells. The ability of different chemoattractants to stimulate Erk2-regulated GtaC translocation suggests that atypical MAPK-mediated regulation of transcription factors can contribute to different cell fates.

Identifiants

pubmed: 35916164
pii: 276183
doi: 10.1242/jcs.260148
pmc: PMC9481928
pii:
doi:

Substances chimiques

Chemotactic Factors 0
GATA Transcription Factors 0
Folic Acid 935E97BOY8
Mitogen-Activated Protein Kinases EC 2.7.11.24

Types de publication

Journal Article Research Support, Non-U.S. Gov't Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : NIGMS NIH HHS
ID : R15 GM131269
Pays : United States
Organisme : NIGMS NIH HHS
ID : T32 GM140953
Pays : United States
Organisme : National Natural Science Foundation of China
ID : 32170701

Informations de copyright

© 2022. Published by The Company of Biologists Ltd.

Déclaration de conflit d'intérêts

Competing interests The authors declare no competing or financial interests.

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Auteurs

Jeffrey A Hadwiger (JA)

Department of Microbiology and Molecular Genetics, Oklahoma State University, Stillwater, OK 74078-3020, USA.

Huaqing Cai (H)

National Laboratory of Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Chaoyang District, Beijing 100101, China.

Ramee G Aranda (RG)

Department of Microbiology and Molecular Genetics, Oklahoma State University, Stillwater, OK 74078-3020, USA.

Saher Fatima (S)

Department of Microbiology and Molecular Genetics, Oklahoma State University, Stillwater, OK 74078-3020, USA.

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Classifications MeSH