Tumor microenvironmental signals reshape chromatin landscapes to limit the functional potential of exhausted T cells.


Journal

Science immunology
ISSN: 2470-9468
Titre abrégé: Sci Immunol
Pays: United States
ID NLM: 101688624

Informations de publication

Date de publication:
05 08 2022
Historique:
entrez: 5 8 2022
pubmed: 6 8 2022
medline: 10 8 2022
Statut: ppublish

Résumé

Response rates to immunotherapy in solid tumors remain low due in part to the elevated prevalence of terminally exhausted T cells, a hypofunctional differentiation state induced through persistent antigen and stress signaling. However, the mechanisms promoting progression to terminal exhaustion in the tumor remain undefined. Using the low-input chromatin immunoprecipitation sequencing method CUT&RUN, we profiled the histone modification landscape of tumor-infiltrating CD8

Identifiants

pubmed: 35930654
doi: 10.1126/sciimmunol.abj9123
pmc: PMC9851604
mid: NIHMS1841161
doi:

Substances chimiques

Chromatin 0
Histones 0
Jumonji Domain-Containing Histone Demethylases EC 1.14.11.-
KDM6B protein, human EC 1.14.11.-

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

eabj9123

Subventions

Organisme : NIAID NIH HHS
ID : DP2 AI136598
Pays : United States
Organisme : NCI NIH HHS
ID : T32 CA082084
Pays : United States
Organisme : NIAID NIH HHS
ID : R21 AI135027
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA047904
Pays : United States
Organisme : NCI NIH HHS
ID : P50 CA121973
Pays : United States
Organisme : NCI NIH HHS
ID : K00 CA222711
Pays : United States
Organisme : NCI NIH HHS
ID : F99 CA222711
Pays : United States
Organisme : NCI NIH HHS
ID : F30 CA247034
Pays : United States
Organisme : NIAID NIH HHS
ID : R21 AI135367
Pays : United States
Organisme : NCI NIH HHS
ID : P50 CA097190
Pays : United States
Organisme : NIAID NIH HHS
ID : R01 AI153104
Pays : United States
Organisme : NIAID NIH HHS
ID : T32 AI089443
Pays : United States
Organisme : NIGMS NIH HHS
ID : T32 GM008208
Pays : United States

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Auteurs

B Rhodes Ford (BR)

Division of Pediatric Rheumatology, Department of Pediatrics, University of Pittsburgh, Pittsburgh, PA 15260, USA.
Department of Immunology, University of Pittsburgh, Pittsburgh, PA 15260, USA.

Paolo D A Vignali (PDA)

Department of Immunology, University of Pittsburgh, Pittsburgh, PA 15260, USA.
Tumor Microenvironment Center, UPMC Hillman Cancer Center, University of Pittsburgh, Pittsburgh, PA 15260, USA.

Natalie L Rittenhouse (NL)

Division of Pediatric Rheumatology, Department of Pediatrics, University of Pittsburgh, Pittsburgh, PA 15260, USA.

Nicole E Scharping (NE)

Department of Immunology, University of Pittsburgh, Pittsburgh, PA 15260, USA.
Tumor Microenvironment Center, UPMC Hillman Cancer Center, University of Pittsburgh, Pittsburgh, PA 15260, USA.

Ronal Peralta (R)

Department of Immunology, University of Pittsburgh, Pittsburgh, PA 15260, USA.
Tumor Microenvironment Center, UPMC Hillman Cancer Center, University of Pittsburgh, Pittsburgh, PA 15260, USA.

Konstantinos Lontos (K)

Tumor Microenvironment Center, UPMC Hillman Cancer Center, University of Pittsburgh, Pittsburgh, PA 15260, USA.
Department of Oncology, University of Pittsburgh Medical Center, Pittsburgh, PA 15260, USA.

Andrew T Frisch (AT)

Department of Immunology, University of Pittsburgh, Pittsburgh, PA 15260, USA.
Tumor Microenvironment Center, UPMC Hillman Cancer Center, University of Pittsburgh, Pittsburgh, PA 15260, USA.

Greg M Delgoffe (GM)

Department of Immunology, University of Pittsburgh, Pittsburgh, PA 15260, USA.
Tumor Microenvironment Center, UPMC Hillman Cancer Center, University of Pittsburgh, Pittsburgh, PA 15260, USA.

Amanda C Poholek (AC)

Division of Pediatric Rheumatology, Department of Pediatrics, University of Pittsburgh, Pittsburgh, PA 15260, USA.
Department of Immunology, University of Pittsburgh, Pittsburgh, PA 15260, USA.

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