ANTXR1 Regulates Erythroid Cell Proliferation and Differentiation through wnt/
Journal
Disease markers
ISSN: 1875-8630
Titre abrégé: Dis Markers
Pays: United States
ID NLM: 8604127
Informations de publication
Date de publication:
2022
2022
Historique:
received:
29
06
2022
revised:
21
07
2022
accepted:
29
07
2022
entrez:
6
9
2022
pubmed:
7
9
2022
medline:
8
9
2022
Statut:
epublish
Résumé
Erythropoiesis is a highly complex and sophisticated multistage process regulated by many transcription factors, as well as noncoding RNAs. Anthrax toxin receptor 1 (ANTXR1) is a type I transmembrane protein that binds the anthrax toxin ligands and mediates the entry of its toxic part into cells. It also functions as a receptor for the Protective antigen (PA) of anthrax toxin, and mediates the entry of Edema factor (EF) and Lethal factor (LF) into the cytoplasm of target cells and exerts their toxicity. Previous research has shown that ANTXR1 inhibits the expression of
Identifiants
pubmed: 36065334
doi: 10.1155/2022/1226697
pmc: PMC9440811
doi:
Substances chimiques
ANTXR1 protein, human
0
Cell Adhesion Molecules
0
Microfilament Proteins
0
Receptors, Cell Surface
0
5-Aminolevulinate Synthetase
EC 2.3.1.37
ALAS2 protein, human
EC 2.3.1.37
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
1226697Informations de copyright
Copyright © 2022 Tingting Jin et al.
Déclaration de conflit d'intérêts
The authors declare no competing financial interests.
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