Gut microbiome and breast-feeding: Implications for early immune development.

Human milk IgA breast-feeding human milk oligosaccharides infant immunity microbiome

Journal

The Journal of allergy and clinical immunology
ISSN: 1097-6825
Titre abrégé: J Allergy Clin Immunol
Pays: United States
ID NLM: 1275002

Informations de publication

Date de publication:
09 2022
Historique:
received: 11 06 2022
revised: 21 07 2022
accepted: 21 07 2022
entrez: 8 9 2022
pubmed: 9 9 2022
medline: 14 9 2022
Statut: ppublish

Résumé

Establishment of the gut microbiome during early life is a complex process with lasting implications for an individual's health. Several factors influence microbial assembly; however, breast-feeding is recognized as one of the most influential drivers of gut microbiome composition during infancy, with potential implications for function. Differences in gut microbial communities between breast-fed and formula-fed infants have been consistently observed and are hypothesized to partially mediate the relationships between breast-feeding and decreased risk for numerous communicable and noncommunicable diseases in early life. Despite decades of research on the gut microbiome of breast-fed infants, there are large scientific gaps in understanding how human milk has evolved to support microbial and immune development. This review will summarize the evidence on how breast-feeding broadly affects the composition and function of the early-life gut microbiome and discuss mechanisms by which specific human milk components shape intestinal bacterial colonization, succession, and function.

Identifiants

pubmed: 36075638
pii: S0091-6749(22)00992-7
doi: 10.1016/j.jaci.2022.07.014
pmc: PMC9463492
mid: NIHMS1828112
pii:
doi:

Types de publication

Journal Article Review Research Support, N.I.H., Extramural Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

523-534

Subventions

Organisme : NICHD NIH HHS
ID : R01 HD106554
Pays : United States
Organisme : NIAID NIH HHS
ID : R21 AI154680
Pays : United States
Organisme : NIAID NIH HHS
ID : U01 AI131344
Pays : United States

Informations de copyright

Copyright © 2022 American Academy of Allergy, Asthma & Immunology. Published by Elsevier Inc. All rights reserved.

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Auteurs

Erin C Davis (EC)

Division of Allergy and Immunology, Center for Food Allergy, Department of Pediatrics, University of Rochester School of Medicine and Dentistry, Golisano Children's Hospital, Rochester, NY.

Vanessa P Castagna (VP)

TurtleTree LLC, Woodland, Calif.

David A Sela (DA)

Department of Food Science, University of Massachusetts Amherst, Amherst, Mass; Department of Microbiology and Physiological Systems, University of Massachusetts Chan Medical School, Worcester, Mass; Organismic and Evolutionary Biology Graduate Program, University of Massachusetts Amherst, Amherst, Mass.

Margaret A Hillard (MA)

Department of Food Science, University of Massachusetts Amherst, Amherst, Mass; Organismic and Evolutionary Biology Graduate Program, University of Massachusetts Amherst, Amherst, Mass.

Samantha Lindberg (S)

Department of Biomedical Sciences, University of Albany, Rensselaer, NY.

Nicholas J Mantis (NJ)

Division of Infectious Diseases, New York State Department of Health, Albany, NY.

Antti E Seppo (AE)

Division of Allergy and Immunology, Center for Food Allergy, Department of Pediatrics, University of Rochester School of Medicine and Dentistry, Golisano Children's Hospital, Rochester, NY.

Kirsi M Järvinen (KM)

Division of Allergy and Immunology, Center for Food Allergy, Department of Pediatrics, University of Rochester School of Medicine and Dentistry, Golisano Children's Hospital, Rochester, NY; Division of Allergy, Immunology, and Rheumatology, Department of Medicine, University of Rochester School of Medicine and Dentistry, Rochester, NY; Department of Microbiology and Immunology, University of Rochester School of Medicine and Dentistry, Rochester, NY. Electronic address: kirsi_jarvinen-seppo@URMC.Rochester.edu.

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