Heterozygous deletion of the VEGFC gene in 4q34.3 is associated with Milroy-like lymphedema: First prenatal case report.

4q34.3 microdeletion Milroy-like disease VEGFC haploinsufficiency prenatal diagnosis primary lymphedema

Journal

American journal of medical genetics. Part A
ISSN: 1552-4833
Titre abrégé: Am J Med Genet A
Pays: United States
ID NLM: 101235741

Informations de publication

Date de publication:
12 2022
Historique:
revised: 08 08 2022
received: 29 05 2022
accepted: 24 08 2022
pubmed: 22 9 2022
medline: 15 11 2022
entrez: 21 9 2022
Statut: ppublish

Résumé

Deleterious variants in the vascular endothelial growth factor C (VEGFC) gene have been recently associated with Milroy-like primary lymphedema, an autosomal dominant disorder, characterized mainly by swelling of the lower limbs due to functional impairment of the lymphatic vessels. To date, only 26 patients with congenital lymphedema harboring VEGFC pathogenic variants were documented. Here, we describe the first prenatal case of a fetus with Milroy-like disease. Fetal ultrasound showed bilateral foot swelling. Chromosomal microarray analysis revealed a 137-kb copy number loss in 4q34.3 including only VEGFC gene in the propositus fetus. Segregation analysis showed that the deletion was inherited from the affected mother and grandmother. Taken together, our study highlights the important role of microarray analysis to detect subtle chromosomal imbalances in the prenatal setting and contributes to delineate the fetal phenotype of VEGFC-related primary congenital lymphedema.

Identifiants

pubmed: 36129367
doi: 10.1002/ajmg.a.62973
doi:

Substances chimiques

Vascular Endothelial Growth Factor C 0
VEGFC protein, human 0

Types de publication

Case Reports

Langues

eng

Sous-ensembles de citation

IM

Pagination

3550-3554

Informations de copyright

© 2022 Wiley Periodicals LLC.

Références

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Auteurs

Minh-Tuan Huynh (MT)

Centre Hospitalier du Havre, Unité de Génétique Clinique, Montivilliers, France.
Laboratoire de Génétique Moléculaire, Pharmacogénétique et Hormonologie, Hôpital Bicêtre, APHP Université Paris Saclay, Le Kremlin-Bicêtre, France.
Inserm UMR_S 1185, Faculté de Médecine Paris Saclay, Université Paris Saclay, Le Kremlin-Bicêtre, France.

Sophie Degre (S)

Centre Hospitalier du Havre, Service de Gynécologie-Obstétrique, Montivilliers, France.

Géraldine Joly-Helas (G)

Centre Hospitalier Universitaire de Rouen, Service de Cytogénétique, Rouen, France.

Cathy Bréon (C)

Centre Hospitalier du Havre, Service de Gynécologie-Obstétrique, Montivilliers, France.

Stéphanie Potel (S)

Centre Hospitalier du Havre, Service de Gynécologie-Obstétrique, Montivilliers, France.

Pascal Chambon (P)

Centre Hospitalier Universitaire de Rouen, Service de Cytogénétique, Rouen, France.

Jérôme Bouligand (J)

Laboratoire de Génétique Moléculaire, Pharmacogénétique et Hormonologie, Hôpital Bicêtre, APHP Université Paris Saclay, Le Kremlin-Bicêtre, France.
Inserm UMR_S 1185, Faculté de Médecine Paris Saclay, Université Paris Saclay, Le Kremlin-Bicêtre, France.

Valérie Layet (V)

Centre Hospitalier du Havre, Unité de Génétique Clinique, Montivilliers, France.

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