Becker Muscular Dystrophy Accompanied by Anti-HMGCR Antibody-positive Immune-mediated Necrotizing Myopathy.


Journal

Internal medicine (Tokyo, Japan)
ISSN: 1349-7235
Titre abrégé: Intern Med
Pays: Japan
ID NLM: 9204241

Informations de publication

Date de publication:
01 May 2023
Historique:
medline: 2 5 2023
pubmed: 22 9 2022
entrez: 21 9 2022
Statut: ppublish

Résumé

Becker muscular dystrophy (BMD) is an X-linked neuromuscular disease characterized by progressive muscle weakness that currently has no cure. Immune-mediated necrotizing myopathy (IMNM) is a type of autoimmune inflammatory myopathy characterized by proximal muscle weakness that is treated with immunosuppressive therapy. We herein report a patient diagnosed with BMD complicated with IMNM by a pathological analysis. Notably, the patient had an elevated serum anti-3-hydroxy-3-methylglutaryl-coenzyme A reductase antibody level. Oral glucocorticoid and methotrexate treatment partially improved the muscle weakness with decreased levels of serum creatine kinase. An accurate diagnosis is important for therapeutic decisions in these complicated cases.

Identifiants

pubmed: 36130887
doi: 10.2169/internalmedicine.0202-22
pmc: PMC10208778
doi:

Substances chimiques

Autoantibodies 0
HMGCR protein, human EC 1.1.1.-

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1345-1349

Références

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Auteurs

Asami Munekane (A)

Department of Neurology, Kawasaki Medical School, Japan.

Yutaka Ohsawa (Y)

Department of Neurology, Kawasaki Medical School, Japan.

Mariko Okubo (M)

Department of Neuromuscular Research, National Institute of Neuroscience (NCNP), Japan.

Taiji Nagai (T)

Department of Neurology, Kawasaki Medical School, Japan.

Hirotake Nishimura (H)

Department of Pathology, Kawasaki Medical School, Japan.

Ichizo Nishino (I)

Department of Neuromuscular Research, National Institute of Neuroscience (NCNP), Japan.

Yoshihide Sunada (Y)

Department of Neurology, Kawasaki Medical School, Japan.

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Classifications MeSH