A PHD inhibitor prevents changes in the phosphoproteome and capillary rarefaction by CsA: treatment option for CKD?


Journal

Kidney international
ISSN: 1523-1755
Titre abrégé: Kidney Int
Pays: United States
ID NLM: 0323470

Informations de publication

Date de publication:
10 2022
Historique:
received: 23 05 2022
revised: 08 06 2022
accepted: 10 06 2022
entrez: 23 9 2022
pubmed: 24 9 2022
medline: 28 9 2022
Statut: ppublish

Résumé

Labes et al. analyze the phosphoproteome in a mouse model of chronic cyclosporine A nephrotoxicity and detect significant changes in the angiogenic pathway. Furthermore, they observe reduced hemoglobin levels and capillary rarefaction in the kidney. The authors show that coadministration of the hypoxia-inducible factor prolyl hydroxylase inhibitor daprodustat almost completely prevents changes of the phosphoproteome and capillary rarefaction, suggesting that prolyl hydroxylase domain enzyme inhibitors may preserve microvasculature of the kidney, which is commonly impaired in chronic kidney disease.

Identifiants

pubmed: 36150758
pii: S0085-2538(22)00532-4
doi: 10.1016/j.kint.2022.06.020
pii:
doi:

Substances chimiques

Hemoglobins 0
Prolyl-Hydroxylase Inhibitors 0
Cyclosporine 83HN0GTJ6D
Prolyl Hydroxylases EC 1.14.11.-
Hypoxia-Inducible Factor-Proline Dioxygenases EC 1.14.11.29

Types de publication

Journal Article Comment

Langues

eng

Sous-ensembles de citation

IM

Pagination

686-688

Commentaires et corrections

Type : CommentOn

Informations de copyright

Copyright © 2022 International Society of Nephrology. Published by Elsevier Inc. All rights reserved.

Auteurs

Gunnar Schley (G)

Department of Nephrology and Hypertension, University Hospital Erlangen, Friedrich-Alexander-Universität Erlangen-Nürnberg, Erlangen, Germany.

Margarete Goppelt-Struebe (M)

Department of Nephrology and Hypertension, University Hospital Erlangen, Friedrich-Alexander-Universität Erlangen-Nürnberg, Erlangen, Germany. Electronic address: margarete.goppelt-struebe@fau.de.

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Classifications MeSH