The impact of organ motion and the appliance of mitigation strategies on the effectiveness of hypoxia-guided proton therapy for non-small cell lung cancer.


Journal

Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology
ISSN: 1879-0887
Titre abrégé: Radiother Oncol
Pays: Ireland
ID NLM: 8407192

Informations de publication

Date de publication:
11 2022
Historique:
received: 25 04 2022
revised: 26 09 2022
accepted: 26 09 2022
pubmed: 14 10 2022
medline: 2 12 2022
entrez: 13 10 2022
Statut: ppublish

Résumé

To investigate the impact of organ motion on hypoxia-guided proton therapy treatments for non-small cell lung cancer (NSCLC) patients. Hypoxia PET and 4D imaging data of six NSCLC patients were used to simulate hypoxia-guided proton therapy with different motion mitigation strategies including rescanning, breath-hold, respiratory gating and tumour tracking. Motion-induced dose degradation was estimated for treatment plans with dose painting of hypoxic tumour sub-volumes at escalated dose levels. Tumour control probability (TCP) and dosimetry indices were assessed to weigh the clinical benefit of dose escalation and motion mitigation. In addition, the difference in normal tissue complication probability (NTCP) between escalated proton and photon VMAT treatments has been assessed. Motion-induced dose degradation was found for target coverage (CTV V NSCLC affected by hypoxia appears to be a prime target for proton therapy which, by dose-escalation, allows to mitigate hypoxia-induced radio-resistance despite the sensitivity to organ motion. Furthermore, substantial reduction in normal tissue toxicity can be expected compared to conventional VMAT. Accessibility and standardization of hypoxia imaging and clinical trials are necessary to confirm these findings in a clinical setting.

Sections du résumé

BACKGROUND AND PURPOSE
To investigate the impact of organ motion on hypoxia-guided proton therapy treatments for non-small cell lung cancer (NSCLC) patients.
MATERIALS AND METHODS
Hypoxia PET and 4D imaging data of six NSCLC patients were used to simulate hypoxia-guided proton therapy with different motion mitigation strategies including rescanning, breath-hold, respiratory gating and tumour tracking. Motion-induced dose degradation was estimated for treatment plans with dose painting of hypoxic tumour sub-volumes at escalated dose levels. Tumour control probability (TCP) and dosimetry indices were assessed to weigh the clinical benefit of dose escalation and motion mitigation. In addition, the difference in normal tissue complication probability (NTCP) between escalated proton and photon VMAT treatments has been assessed.
RESULTS
Motion-induced dose degradation was found for target coverage (CTV V
CONCLUSION
NSCLC affected by hypoxia appears to be a prime target for proton therapy which, by dose-escalation, allows to mitigate hypoxia-induced radio-resistance despite the sensitivity to organ motion. Furthermore, substantial reduction in normal tissue toxicity can be expected compared to conventional VMAT. Accessibility and standardization of hypoxia imaging and clinical trials are necessary to confirm these findings in a clinical setting.

Identifiants

pubmed: 36228759
pii: S0167-8140(22)04491-7
doi: 10.1016/j.radonc.2022.09.021
pii:
doi:

Substances chimiques

Protons 0

Types de publication

Clinical Trial Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

208-214

Informations de copyright

Copyright © 2022 The Author(s). Published by Elsevier B.V. All rights reserved.

Auteurs

Andreas Köthe (A)

Center for Proton Therapy, Paul Scherrer Institute, Villigen, Switzerland; Department of Physics, ETH-Hönggerberg, Zurich, Switzerland. Electronic address: andreas.koethe@psi.ch.

Antony John Lomax (AJ)

Center for Proton Therapy, Paul Scherrer Institute, Villigen, Switzerland; Department of Physics, ETH-Hönggerberg, Zurich, Switzerland.

Anna Chiara Giovannelli (AC)

Center for Proton Therapy, Paul Scherrer Institute, Villigen, Switzerland; Department of Physics, ETH-Hönggerberg, Zurich, Switzerland.

Sairos Safai (S)

Center for Proton Therapy, Paul Scherrer Institute, Villigen, Switzerland.

Nicola Bizzocchi (N)

Center for Proton Therapy, Paul Scherrer Institute, Villigen, Switzerland.

Erik Roelofs (E)

Department of Radiation Oncology (Maastro), Maastricht University Medical Center+, GROW-School for Oncology and Reproduction, Maastricht, The Netherlands.

Aniek J G Even (AJG)

Department of Radiation Oncology (Maastro), Maastricht University Medical Center+, GROW-School for Oncology and Reproduction, Maastricht, The Netherlands.

Damien Charles Weber (DC)

Center for Proton Therapy, Paul Scherrer Institute, Villigen, Switzerland; Department of Radiation Oncology, Inselspital, Bern University Hospital, University of Bern, Switzerland; Radiation Oncology Department, University Hospital of Zurich, Zurich, Switzerland.

Giovanni Fattori (G)

Center for Proton Therapy, Paul Scherrer Institute, Villigen, Switzerland.

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