Anti-Inflammatory Effects of GLP-1R Activation in the Retina.


Journal

International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791

Informations de publication

Date de publication:
17 Oct 2022
Historique:
received: 06 09 2022
revised: 12 10 2022
accepted: 13 10 2022
entrez: 27 10 2022
pubmed: 28 10 2022
medline: 29 10 2022
Statut: epublish

Résumé

Glucagon-like peptide-1 (GLP-1) is an incretin hormone, mainly produced by enteroendocrine L cells, which participates in the regulation of glucose homeostasis, and in reduction in body weight by promoting satiety. Actions of GLP-1 are mediated by activation of its receptor GLP-1R, which is widely expressed in several tissues including the retina. The effects of GLP-1R activation are useful in the management of type 2 diabetes mellitus (T2DM). In addition, the activation of GLP-1R has anti-inflammatory effects in several organs, suggesting that it may be also useful in the treatment of inflammatory diseases. Inflammation is a common element in the pathogenesis of several ocular diseases, and the protective effects of treatment with GLP-1 emerged also in retinal diseases. In this review we highlight the anti-inflammatory effects of GLP-1R activation in the retina. Firstly, we summarized the pathogenic role of inflammation in ocular diseases. Then, we described the pleiotropic effects of GLP-1R activation on the cellular components of the retina which are mainly involved in the pathogenesis of inflammatory retinal diseases: the retinal ganglion cells, retinal pigment epithelial cells and endothelial cells.

Identifiants

pubmed: 36293281
pii: ijms232012428
doi: 10.3390/ijms232012428
pmc: PMC9604172
pii:
doi:

Substances chimiques

Glucagon-Like Peptide-1 Receptor 0
Incretins 0
Hypoglycemic Agents 0
Glucagon-Like Peptide 1 89750-14-1
Glucose IY9XDZ35W2
Anti-Inflammatory Agents 0

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Déclaration de conflit d'intérêts

The authors declare no conflict of interest.

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Auteurs

Alessandra Puddu (A)

Department of Internal Medicine and Medical Specialties, University of Genoa, 16132 Genoa, Italy.

Davide Maggi (D)

Department of Internal Medicine and Medical Specialties, University of Genoa, 16132 Genoa, Italy.

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Classifications MeSH