Accrual of organ damage in Behçet's syndrome: trajectory, associated factors, and impact on patients' quality of life over a 2-year prospective follow-up study.


Journal

Arthritis research & therapy
ISSN: 1478-6362
Titre abrégé: Arthritis Res Ther
Pays: England
ID NLM: 101154438

Informations de publication

Date de publication:
17 11 2022
Historique:
received: 15 06 2022
accepted: 03 11 2022
entrez: 18 11 2022
pubmed: 19 11 2022
medline: 22 11 2022
Statut: epublish

Résumé

This study aimed to investigate the trajectory of damage accrual, associated factors, and impact on health-related quality of life (HR-QoL) in a multicenter cohort of patients with Behçet's syndrome (BS) over 2 years of follow-up. Patients recruited in the BS Overall Damage Index (BODI) validation study were prospectively monitored for 2 years and assessed for damage accrual, defined as an increase ≥1 in the BODI score, and HR-QoL was evaluated by the SF-36 questionnaire. Logistic and multiple linear regression models were built to determine factors associated with damage accrual and impairment in the different SF-36 domains. During follow-up, 36 out of 189 (19.0%) patients had an increase ≥1 in the BODI score with a mean (SD) difference of 1.7 (0.8) (p <0.001). The incidence rate of damage accrual was stable over time, regardless of the disease duration. Out of 61 new BODI items, 25 (41.0%) were considered related to glucocorticoid (GC) use. In multivariate analysis, duration of GC therapy (OR per 1-year 1.15, 95% CI 1.07-1.23; p <0.001) and occurrence of ≥1 disease relapse (OR 3.15, 95% CI 1.09-9.12; p 0.038) were identified as predictors of damage accrual, whereas the use of immunosuppressants showed a protective effect (OR 0.20, 95% CI 0.08-0.54, p<0.001). Damage accrual was independently associated with the impairment of different physical domains and, to a greater extent, in emotional domains of the SF-36 questionnaire. Female sex, higher disease activity, and fibromyalgia were also significantly associated with impairment in HR-QoL. In BS, organ damage accrues over time, also in long-standing disease, resulting in an impairment of the perceived physical and mental health. Adequate immunosuppressive treatment, preventing disease flares and minimizing exposure to GCs have a crucial role in lowering the risk of damage accrual.

Sections du résumé

BACKGROUND
This study aimed to investigate the trajectory of damage accrual, associated factors, and impact on health-related quality of life (HR-QoL) in a multicenter cohort of patients with Behçet's syndrome (BS) over 2 years of follow-up.
METHODS
Patients recruited in the BS Overall Damage Index (BODI) validation study were prospectively monitored for 2 years and assessed for damage accrual, defined as an increase ≥1 in the BODI score, and HR-QoL was evaluated by the SF-36 questionnaire. Logistic and multiple linear regression models were built to determine factors associated with damage accrual and impairment in the different SF-36 domains.
RESULTS
During follow-up, 36 out of 189 (19.0%) patients had an increase ≥1 in the BODI score with a mean (SD) difference of 1.7 (0.8) (p <0.001). The incidence rate of damage accrual was stable over time, regardless of the disease duration. Out of 61 new BODI items, 25 (41.0%) were considered related to glucocorticoid (GC) use. In multivariate analysis, duration of GC therapy (OR per 1-year 1.15, 95% CI 1.07-1.23; p <0.001) and occurrence of ≥1 disease relapse (OR 3.15, 95% CI 1.09-9.12; p 0.038) were identified as predictors of damage accrual, whereas the use of immunosuppressants showed a protective effect (OR 0.20, 95% CI 0.08-0.54, p<0.001). Damage accrual was independently associated with the impairment of different physical domains and, to a greater extent, in emotional domains of the SF-36 questionnaire. Female sex, higher disease activity, and fibromyalgia were also significantly associated with impairment in HR-QoL.
CONCLUSION
In BS, organ damage accrues over time, also in long-standing disease, resulting in an impairment of the perceived physical and mental health. Adequate immunosuppressive treatment, preventing disease flares and minimizing exposure to GCs have a crucial role in lowering the risk of damage accrual.

Identifiants

pubmed: 36397162
doi: 10.1186/s13075-022-02947-y
pii: 10.1186/s13075-022-02947-y
pmc: PMC9670626
doi:

Substances chimiques

Immunosuppressive Agents 0

Types de publication

Multicenter Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

253

Informations de copyright

© 2022. The Author(s).

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Auteurs

Alberto Floris (A)

Department of Medical Sciences and Public Health, University of Cagliari, SS 554, 09042, Monserrato, CA, Italy.
Rheumatology Unit, Azienda Ospedaliero-Universitaria of Cagliari, Monserrato, Italy.

Matteo Piga (M)

Department of Medical Sciences and Public Health, University of Cagliari, SS 554, 09042, Monserrato, CA, Italy. matteopiga@unica.it.
Rheumatology Unit, Azienda Ospedaliero-Universitaria of Cagliari, Monserrato, Italy. matteopiga@unica.it.

Riccardo Laconi (R)

Department of Medical Sciences and Public Health, University of Cagliari, SS 554, 09042, Monserrato, CA, Italy.
Rheumatology Unit, Azienda Ospedaliero-Universitaria of Cagliari, Monserrato, Italy.

Gerard Espinosa (G)

Department of Autoimmune Diseases, University of Barcelona, Hospital Clinic, Institut d'Investigacions Biomediques August Pi i Sunyer (IDIBAPS), Barcelona, Catalonia, Spain.

Giuseppe Lopalco (G)

University of Bari, Rheumatology Unit, Bari, Italy.

Luisa Serpa Pinto (L)

Hospital Santo Antonio Centro Hospitalar do Porto, Unidade de Imunologia Clinica, Porto, Portugal.

Nikolaos Kougkas (N)

Rheumatology, Clinical Immunology and Allergy Unit, University of Crete, Heraklion, Greece.
Fourth Department of Internal Medicine, School of Medicine, Hippokration Hospital, Thessaloniki, Greece.

Jurgen Sota (J)

Rheumatology Unit, University of Siena, Siena, Italy.

Andrea Lo Monaco (A)

Rheumatology Unit - AOU, S. Anna, Ferrara, University of Ferrara, Ferrara, Italy.

Marcello Govoni (M)

Rheumatology Unit - AOU, S. Anna, Ferrara, University of Ferrara, Ferrara, Italy.

Luca Cantarini (L)

Rheumatology Unit, University of Siena, Siena, Italy.

George Bertsias (G)

Rheumatology, Clinical Immunology and Allergy Unit, University of Crete, Heraklion, Greece.

João Correia (J)

Hospital Santo Antonio Centro Hospitalar do Porto, Unidade de Imunologia Clinica, Porto, Portugal.

Florenzo Iannone (F)

University of Bari, Rheumatology Unit, Bari, Italy.

Ricard Cervera (R)

Department of Autoimmune Diseases, University of Barcelona, Hospital Clinic, Institut d'Investigacions Biomediques August Pi i Sunyer (IDIBAPS), Barcelona, Catalonia, Spain.

Carlos Vasconcelos (C)

University of Porto, UMIB Abel Salazar Biomedical Sciences Institute, Porto, Portugal.

Alessandro Mathieu (A)

Department of Medical Sciences and Public Health, University of Cagliari, SS 554, 09042, Monserrato, CA, Italy.

Alberto Cauli (A)

Department of Medical Sciences and Public Health, University of Cagliari, SS 554, 09042, Monserrato, CA, Italy.
Rheumatology Unit, Azienda Ospedaliero-Universitaria of Cagliari, Monserrato, Italy.

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