A high-throughput electron tomography workflow reveals over-elongated centrioles in relapsed/refractory multiple myeloma.

bone marrow centriole fragmentation centriole over-elongation centrosome electron tomography high-throughput electron microscopy multiple myeloma plasma cell disorder

Journal

Cell reports methods
ISSN: 2667-2375
Titre abrégé: Cell Rep Methods
Pays: United States
ID NLM: 9918227360606676

Informations de publication

Date de publication:
21 11 2022
Historique:
received: 08 04 2022
revised: 24 08 2022
accepted: 06 10 2022
entrez: 1 12 2022
pubmed: 2 12 2022
medline: 2 12 2022
Statut: epublish

Résumé

Electron microscopy is the gold standard to characterize centrosomal ultrastructure. However, production of significant morphometrical data is highly limited by acquisition time. We therefore developed a generalizable, semi-automated high-throughput electron tomography strategy to study centrosome aberrations in sparse patient-derived cancer cells at nanoscale. As proof of principle, we present electron tomography data on 455 centrioles of CD138

Identifiants

pubmed: 36452870
doi: 10.1016/j.crmeth.2022.100322
pii: S2667-2375(22)00212-0
pmc: PMC9701608
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Pagination

100322

Informations de copyright

© 2022 The Author(s).

Déclaration de conflit d'intérêts

H.G. has research funding from Amgen, BMS, Celgene, Chugai, Janssen, Incyte, Molecular Partners, Merck Sharp and Dohme, Sanofi, Mundipharma GmbH, Takeda, Dietmar-Hopp-Foundation, Johns Hopkins University, and Novartis; served as an advisor for Adaptive Biotechnology, Amgen, BMS, Celgene, Janssen, Sanofi, and Takeda; and received honoraria from Amgen, BMS, Celgene, Chugai, GlaxoSmithKline, Janssen, Novartis, and Sanofi. C.M.-T. served as a consultant for Janssen and received research funding from Bioline, Pfizer, and Janssen. M.S.R. received research funding from Amgen, Sanofi, Novartis, and Heidelberg Pharma and served as a consultant for Amgen, Sanofi, Novartis, BMS, Janssen, AbbVie, Regeneron, Takeda, and GSK. U.H. received travel grants from Janssen, Prothena, and Pfizer; served on advisory boards for Pfizer, Janssen, and Prothena; received honoraria from Janssen, Pfizer, Alnylam, and Akcea; and received research funding from Janssen and Prothena. A.K. received honoraria from Hoffmann-La Roche, Bayer, Daiichi Sankyo, and AbbVie and research funding from Bayer and Merck and is a paid consultant for Hoffmann-La Roche, Daiichi Sankyo, Bristol Myers Squibb, and AbbVie.

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Auteurs

Tobias Dittrich (T)

Clinical Cooperation Unit Molecular Hematology/Oncology, German Cancer Research Center (DKFZ), and Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.
Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), 69117 Heidelberg, Germany.
Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.
Amyloidosis Center, University of Heidelberg, 69120 Heidelberg, Germany.

Sebastian Köhrer (S)

Clinical Cooperation Unit Molecular Hematology/Oncology, German Cancer Research Center (DKFZ), and Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.
Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), 69117 Heidelberg, Germany.

Martin Schorb (M)

Electron Microscopy Core Facility, European Molecular Biology Laboratory (EMBL), 69117 Heidelberg, Germany.

Isabella Haberbosch (I)

Clinical Cooperation Unit Molecular Hematology/Oncology, German Cancer Research Center (DKFZ), and Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.
Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.

Mandy Börmel (M)

Electron Microscopy Core Facility, European Molecular Biology Laboratory (EMBL), 69117 Heidelberg, Germany.

Hartmut Goldschmidt (H)

Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.
National Center for Tumor Diseases (NCT), University of Heidelberg, 69120 Heidelberg, Germany.

Gabor Pajor (G)

Clinical Cooperation Unit Molecular Hematology/Oncology, German Cancer Research Center (DKFZ), and Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.

Carsten Müller-Tidow (C)

Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.
National Center for Tumor Diseases (NCT), University of Heidelberg, 69120 Heidelberg, Germany.

Marc S Raab (MS)

Clinical Cooperation Unit Molecular Hematology/Oncology, German Cancer Research Center (DKFZ), and Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.
Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.

Ute Hegenbart (U)

Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.
Amyloidosis Center, University of Heidelberg, 69120 Heidelberg, Germany.

Stefan O Schönland (SO)

Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.
Amyloidosis Center, University of Heidelberg, 69120 Heidelberg, Germany.

Yannick Schwab (Y)

Cell Biology and Biophysics Unit, European Molecular Biology Laboratory (EMBL), 69117 Heidelberg, Germany.
Electron Microscopy Core Facility, European Molecular Biology Laboratory (EMBL), 69117 Heidelberg, Germany.

Alwin Krämer (A)

Clinical Cooperation Unit Molecular Hematology/Oncology, German Cancer Research Center (DKFZ), and Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.
Department of Internal Medicine V, University of Heidelberg, 69120 Heidelberg, Germany.

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Classifications MeSH