Germline findings in patients with advanced malignancies screened with paired blood-tumour testing for personalised treatment approaches.


Journal

European journal of cancer (Oxford, England : 1990)
ISSN: 1879-0852
Titre abrégé: Eur J Cancer
Pays: England
ID NLM: 9005373

Informations de publication

Date de publication:
01 2023
Historique:
received: 01 07 2022
revised: 18 10 2022
accepted: 02 11 2022
pubmed: 11 12 2022
medline: 4 1 2023
entrez: 10 12 2022
Statut: ppublish

Résumé

Sequencing of tumour tissue with comprehensive gene panels is increasingly used to guide treatment in precision oncology. Analysis of tumour-normal pairs allows in contrast to tumour-only assessment direct discrimination between somatic and germline alterations, which might have important implications not only for the patients but also their families. We performed tumour normal sequencing with a large gene panel in 1048 patients with advanced cancer to support treatment decision. Sequencing results were correlated with clinical and family data. We identified 156 likely pathogenic or pathogenic (LP/P) germline variants in cancer predisposition genes (CPGs) in 144 cases (13.7%). Of all patients, 8.8% had a LP/P variant in autosomal-dominant cancer predisposition genes (AD-CPGs), most of them being genes with high or moderate penetrance (ATM, BRCA2, CHEK2 and BRCA1). In 48 cases, the P/LP variant matched the expected tumour spectrum. A second variant in tumour tissue was found in 31 patients with AD-CPG variants. Low frequency mutations in either TP53, ATM or DNMT3A in the normal sample indicated clonal haematopoiesis in five cases. Tumour-normal testing for personalised treatment identifies germline LP/P variants in a relevant proportion of patients with cancer. The majority of them would not have been referred to genetic counselling based on family history. Indirect functional readouts of tumour-normal sequencing can provide novel links between CPGs and unexpected cancers. The interpretation of increasingly complex datasets in precision oncology is challenging and concepts of interdisciplinary personalised cancer prevention are needed to support patients and their families.

Sections du résumé

BACKGROUND
Sequencing of tumour tissue with comprehensive gene panels is increasingly used to guide treatment in precision oncology. Analysis of tumour-normal pairs allows in contrast to tumour-only assessment direct discrimination between somatic and germline alterations, which might have important implications not only for the patients but also their families.
METHODS
We performed tumour normal sequencing with a large gene panel in 1048 patients with advanced cancer to support treatment decision. Sequencing results were correlated with clinical and family data.
RESULTS
We identified 156 likely pathogenic or pathogenic (LP/P) germline variants in cancer predisposition genes (CPGs) in 144 cases (13.7%). Of all patients, 8.8% had a LP/P variant in autosomal-dominant cancer predisposition genes (AD-CPGs), most of them being genes with high or moderate penetrance (ATM, BRCA2, CHEK2 and BRCA1). In 48 cases, the P/LP variant matched the expected tumour spectrum. A second variant in tumour tissue was found in 31 patients with AD-CPG variants. Low frequency mutations in either TP53, ATM or DNMT3A in the normal sample indicated clonal haematopoiesis in five cases.
CONCLUSIONS
Tumour-normal testing for personalised treatment identifies germline LP/P variants in a relevant proportion of patients with cancer. The majority of them would not have been referred to genetic counselling based on family history. Indirect functional readouts of tumour-normal sequencing can provide novel links between CPGs and unexpected cancers. The interpretation of increasingly complex datasets in precision oncology is challenging and concepts of interdisciplinary personalised cancer prevention are needed to support patients and their families.

Identifiants

pubmed: 36495689
pii: S0959-8049(22)01338-7
doi: 10.1016/j.ejca.2022.11.003
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

48-55

Informations de copyright

Copyright © 2022 Elsevier Ltd. All rights reserved.

Auteurs

Cristiana Roggia (C)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany. Electronic address: cristiana.roggia@med.uni-tuebingen.de.

Sorin Armeanu-Ebinger (S)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany.

Axel Gschwind (A)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany.

Olga Seibel-Kelemen (O)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany.

Sonja Hertler (S)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany.

Ulrike Faust (U)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany.

Alexandra Liebmann (A)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany.

Tobias B Haack (TB)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany.

Manuela Neumann (M)

Institute of Pathology and Neuropathology, University Hospital Tübingen, Tübingen, Germany.

Irina Bonzheim (I)

Institute of Pathology and Neuropathology, University Hospital Tübingen, Tübingen, Germany.

Andrea Forschner (A)

Department of Dermatology, University Hospital Tübingen, Tübingen, Germany.

Hans-Georg Kopp (HG)

Robert Bosch Center for Tumor Diseases (RBCT) Robert Bosch Cancer Center, Stuttgart, Germany.

Franziska Herster (F)

Robert Bosch Center for Tumor Diseases (RBCT) Robert Bosch Cancer Center, Stuttgart, Germany.

Andreas Hartkopf (A)

Department of Gynecology, University Hospital Tübingen, Tübingen, Germany.

Michael Bitzer (M)

Department of Internal Medicine, University Hospital Tübingen, Tübingen, Germany.

Nisar P Malek (NP)

Department of Internal Medicine, University Hospital Tübingen, Tübingen, Germany.

Ines B Brecht (IB)

Department of General Pediatrics, Pediatric Hematology and Oncology, University Hospital Tübingen, Tübingen, Germany.

Kristina Ruhm (K)

Center for Personalized Medicine, University of Tübingen, Tübingen, Germany.

Yvonne Möller (Y)

Center for Personalized Medicine, University of Tübingen, Tübingen, Germany.

Hubert Löwenheim (H)

Department of Otolaryngology-Head & Neck Surgery, University Hospital Tübingen, Tübingen, Germany.

Stephan Ossowski (S)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany.

Olaf H Rieß (OH)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany.

Christopher Schroeder (C)

Institute of Medical Genetics and Applied Genomics, University Hospital and Medical Faculty Tübingen, Tübingen, Germany.

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