Therapeutic sensitivity to standard treatments in BRCA positive metastatic castration-resistant prostate cancer patients-a systematic review and meta-analysis.


Journal

Prostate cancer and prostatic diseases
ISSN: 1476-5608
Titre abrégé: Prostate Cancer Prostatic Dis
Pays: England
ID NLM: 9815755

Informations de publication

Date de publication:
Dec 2023
Historique:
received: 06 09 2022
accepted: 29 11 2022
revised: 22 11 2022
medline: 13 11 2023
pubmed: 13 12 2022
entrez: 12 12 2022
Statut: ppublish

Résumé

Recent oncology guidelines recommend BRCA1/2 testing for a wide range of prostate cancer (PCa) patients. In addition, PARP inhibitors are available for mutation-positive metastatic castration-resistant PCa (mCRPC) patients following prior treatment with abiraterone, enzalutamide or docetaxel. However, the question of which of these standard treatments is the most effective for BRCA1/2 positive mCRPC patients remains to be answered. The aim of this meta-analysis was to assess the efficacy of abiraterone, enzalutamide and docetaxel in BRCA1/2 mutation-positive mCRPC patients in terms of PSA-response (PSA50), progression-free survival (PFS) and overall survival (OS). As no interventional trials are available on this topic, we performed the data synthesis of BRCA1/2 positive mCRPC patients by using both proportional and individual patient data. For PSA50 evaluation, we pooled event rates with 95% confidence intervals (CI), while for time-to-event (PFS, OS) analyses we used individual patient data with random effect Cox regression calculations. Our meta-analysis included 16 eligible studies with 348 BRCA1/2 positive mCRPC patients. In the first treatment line, response rates for abiraterone, enzalutamide and docetaxel were 52% (CI: 25-79%), 64% (CI: 43-80%) and 55% (CI: 36-73%), respectively. Analyses of individual patient data revealed a PFS (HR: 0.47, CI: 0.26-0.83, p = 0.010) but no OS (HR: 1.41, CI: 0.82-2.42, p = 0.210) benefit for enzalutamide compared to abiraterone-treated patients. Our PSA50 analyses revealed that all the three first-line treatments have therapeutic effect in BRCA1/2 positive mCRPC; although, based on the results of PSA50 and PFS analyses, BRCA positive mCRPC patients might better respond to enzalutamide treatment. However, molecular marker-driven interventional studies directly comparing these agents are crucial for providing higher-level evidence.

Sections du résumé

BACKGROUND BACKGROUND
Recent oncology guidelines recommend BRCA1/2 testing for a wide range of prostate cancer (PCa) patients. In addition, PARP inhibitors are available for mutation-positive metastatic castration-resistant PCa (mCRPC) patients following prior treatment with abiraterone, enzalutamide or docetaxel. However, the question of which of these standard treatments is the most effective for BRCA1/2 positive mCRPC patients remains to be answered. The aim of this meta-analysis was to assess the efficacy of abiraterone, enzalutamide and docetaxel in BRCA1/2 mutation-positive mCRPC patients in terms of PSA-response (PSA50), progression-free survival (PFS) and overall survival (OS).
METHODS METHODS
As no interventional trials are available on this topic, we performed the data synthesis of BRCA1/2 positive mCRPC patients by using both proportional and individual patient data. For PSA50 evaluation, we pooled event rates with 95% confidence intervals (CI), while for time-to-event (PFS, OS) analyses we used individual patient data with random effect Cox regression calculations.
RESULTS RESULTS
Our meta-analysis included 16 eligible studies with 348 BRCA1/2 positive mCRPC patients. In the first treatment line, response rates for abiraterone, enzalutamide and docetaxel were 52% (CI: 25-79%), 64% (CI: 43-80%) and 55% (CI: 36-73%), respectively. Analyses of individual patient data revealed a PFS (HR: 0.47, CI: 0.26-0.83, p = 0.010) but no OS (HR: 1.41, CI: 0.82-2.42, p = 0.210) benefit for enzalutamide compared to abiraterone-treated patients.
CONCLUSIONS CONCLUSIONS
Our PSA50 analyses revealed that all the three first-line treatments have therapeutic effect in BRCA1/2 positive mCRPC; although, based on the results of PSA50 and PFS analyses, BRCA positive mCRPC patients might better respond to enzalutamide treatment. However, molecular marker-driven interventional studies directly comparing these agents are crucial for providing higher-level evidence.

Identifiants

pubmed: 36509931
doi: 10.1038/s41391-022-00626-2
pii: 10.1038/s41391-022-00626-2
pmc: PMC10638083
doi:

Substances chimiques

Docetaxel 15H5577CQD
enzalutamide 93T0T9GKNU
BRCA1 protein, human 0
BRCA1 Protein 0
BRCA2 protein, human 0
BRCA2 Protein 0
Nitriles 0

Types de publication

Meta-Analysis Systematic Review Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

665-672

Subventions

Organisme : Magyar Tudományos Akadémia (Hungarian Academy of Sciences)
ID : BO/00451/20/5

Informations de copyright

© 2022. The Author(s).

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Auteurs

Tamás Fazekas (T)

Department of Urology, Semmelweis University, Budapest, Hungary.
Centre for Translational Medicine, Semmelweis University, Budapest, Hungary.

Ádám D Széles (ÁD)

Department of Urology, Semmelweis University, Budapest, Hungary.
Centre for Translational Medicine, Semmelweis University, Budapest, Hungary.

Brigitta Teutsch (B)

Centre for Translational Medicine, Semmelweis University, Budapest, Hungary.
Institute for Translational Medicine, Medical School, University of Pécs, Pécs, Hungary.

Anita Csizmarik (A)

Department of Urology, Semmelweis University, Budapest, Hungary.

Bálint Vékony (B)

Department of Urology, Semmelweis University, Budapest, Hungary.

Alex Váradi (A)

Centre for Translational Medicine, Semmelweis University, Budapest, Hungary.

Tamás Kói (T)

Centre for Translational Medicine, Semmelweis University, Budapest, Hungary.
Department of Stochastics, Institute of Mathematics, Budapest University of Technology and Economics, Budapest, Hungary.

Zsolt Lang (Z)

Centre for Translational Medicine, Semmelweis University, Budapest, Hungary.
Department of Biostatistics, University of Veterinary Medicine Budapest, Budapest, Hungary.

Nándor Ács (N)

Centre for Translational Medicine, Semmelweis University, Budapest, Hungary.
Department of Obstetrics and Gynecology, Semmelweis University, Budapest, Hungary.

Zsolt Kopa (Z)

Department of Urology, Semmelweis University, Budapest, Hungary.
Centre for Translational Medicine, Semmelweis University, Budapest, Hungary.

Péter Hegyi (P)

Centre for Translational Medicine, Semmelweis University, Budapest, Hungary.
Institute for Translational Medicine, Medical School, University of Pécs, Pécs, Hungary.
Division of Pancreatic Diseases, Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Boris Hadaschik (B)

Department of Urology, University of Duisburg-Essen and German Cancer Consortium (DKTK)-University Hospital Essen, Essen, Germany.

Viktor Grünwald (V)

Department of Urology, University of Duisburg-Essen and German Cancer Consortium (DKTK)-University Hospital Essen, Essen, Germany.

Péter Nyirády (P)

Department of Urology, Semmelweis University, Budapest, Hungary.
Centre for Translational Medicine, Semmelweis University, Budapest, Hungary.

Tibor Szarvas (T)

Department of Urology, Semmelweis University, Budapest, Hungary. szarvas.tibor@med.semmelweis-univ.hu.
Centre for Translational Medicine, Semmelweis University, Budapest, Hungary. szarvas.tibor@med.semmelweis-univ.hu.
Department of Urology, University of Duisburg-Essen and German Cancer Consortium (DKTK)-University Hospital Essen, Essen, Germany. szarvas.tibor@med.semmelweis-univ.hu.

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