CCR4


Journal

European journal of immunology
ISSN: 1521-4141
Titre abrégé: Eur J Immunol
Pays: Germany
ID NLM: 1273201

Informations de publication

Date de publication:
04 2023
Historique:
revised: 23 01 2023
received: 27 10 2021
accepted: 30 01 2023
medline: 12 4 2023
pubmed: 2 2 2023
entrez: 1 2 2023
Statut: ppublish

Résumé

Psoriasis is a chronic inflammatory skin disease with an autoimmune component and associated with joint inflammation in up to 30% of cases. To investigate autoreactive T cells, we developed an imiquimod-induced psoriasis-like inflammation model in K5-mOVA.tg C57BL/6 mice expressing ovalbumin (OVA) on the keratinocyte membrane, adoptively transferred with OT-I OVA-specific CD8

Identifiants

pubmed: 36722608
doi: 10.1002/eji.202149702
doi:

Substances chimiques

Imiquimod P1QW714R7M
Receptors, Antigen, T-Cell 0
CCR4 protein, human 0
Receptors, CCR4 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e2149702

Subventions

Organisme : The National Psoriasis Foundation-NPF
Organisme : University of Ferrara
ID : 2020-FAR.L-CF_003
Organisme : University of Ferrara
ID : 2021-FAR.L-CF_002

Informations de copyright

© 2023 The Authors. European Journal of Immunology published by Wiley-VCH GmbH.

Références

Boehncke, W. H. and Schon, M. P., Psoriasis. Lancet 2015. 386: 983-994.
Ceponis, A. and Kavanaugh, A., Use of methotrexate in patients with psoriatic arthritis. Clin. Exp. Rheumatol. 2010. 28: S132-S137.
Chandran, V. and Gladman, D. D., Update on biomarkers in psoriatic arthritis. Curr. Rheumatol. Rep. 2010. 12: 288-294.
Siegel, D., Devaraj, S., Mitra, A., Raychaudhuri, S. P., Raychaudhuri, S. K. and Jialal, I., Inflammation, atherosclerosis, and psoriasis. Clin. Rev. Allergy Immunol. 2013. 44: 194-204.
Ghoreschi, K., Balato, A., Enerback, C. and Sabat, R., Therapeutics targeting the IL-23 and IL-17 pathway in psoriasis. Lancet 2021. 397: 754-766.
Conrad, C., Domizio, J. D., Mylonas, A., Belkhodja, C., Demaria, O., Navarini, A. A., Lapointe, A. K. et al., TNF blockade induces a dysregulated type I interferon response without autoimmunity in paradoxical psoriasis. Nat. Commun. 2018. 9: 25.
Mylonas, A. and Conrad, C., Psoriasis: classical vs. paradoxical. The Yin-Yang of TNF and type I interferon. Front. Immunol. 2018. 9: 2746.
Bridgewood, C., Fearnley, G. W., Berekmeri, A., Laws, P., Macleod, T., Ponnambalam, S., Stacey, M. et al., IL-36gamma is a strong inducer of IL-23 in psoriatic cells and activates angiogenesis. Front. Immunol. 2018. 9: 200.
Glitzner, E., Korosec, A., Brunner, P. M., Drobits, B., Amberg, N., Schonthaler, H. B., Kopp, T. et al., Specific roles for dendritic cell subsets during initiation and progression of psoriasis. EMBO Mol. Med. 2014. 6: 1312-1327.
Bose, F., Petti, L., Diani, M., Moscheni, C., Molteni, S., Altomare, A., Rossi, R. L. et al., Inhibition of CCR7/CCL19 axis in lesional skin is a critical event for clinical remission induced by TNF blockade in patients with psoriasis. Am. J. Pathol. 2013. 183: 413-421.
Bergen, L. L. T., Petrovic, A., Aarebrot, A. K. and Appel, S., Current knowledge on autoantigens and autoantibodies in psoriasis. Scand. J. Immunol. 2020. 92: e12945.
Prinz, J. C., Human leukocyte antigen-class I alleles and the autoreactive T cell response in psoriasis pathogenesis. Front. Immunol. 2018. 9: 954.
Vural, S., Kerl, K., Dogan, P. E., Vollmer, S., Puchta, U., He, M., Arakawa, Y. et al., Lesional activation of T(c) 17 cells in Behcet disease and psoriasis supports HLA class I-mediated autoimmune responses. Br. J. Dermatol. 2021. 185: 1209-1220.
Arakawa, A., Siewert, K., Stohr, J., Besgen, P., Kim, S. M., Ruhl, G., Nickel, J. et al., Melanocyte antigen triggers autoimmunity in human psoriasis. J. Exp. Med. 2015. 212: 2203-2212.
Lande, R., Botti, E., Jandus, C., Dojcinovic, D., Fanelli, G., Conrad, C., Chamilos, G. et al., The antimicrobial peptide LL37 is a T-cell autoantigen in psoriasis. Nat. Commun. 2014. 5: 5621.
Sgambelluri, F., Diani, M., Altomare, A., Frigerio, E., Drago, L., Granucci, F., Banfi, G. et al., A role for CCR5(+)CD4 T cells in cutaneous psoriasis and for CD103(+) CCR4(+) CD8 Teff cells in the associated systemic inflammation. J. Autoimmun. 2016. 70: 80-90.
Bromley, S. K., Yan, S., Tomura, M., Kanagawa, O. and Luster, A. D., Recirculating memory T cells are a unique subset of CD4+ T cells with a distinct phenotype and migratory pattern. J. Immunol. 2013. 190: 970-976.
Diani, M., Casciano, F., Marongiu, L., Longhi, M., Altomare, A., Pigatto, P. D., Secchiero, P. et al., Increased frequency of activated CD8(+) T cell effectors in patients with psoriatic arthritis. Sci. Rep. 2019. 9: 10870.
Diani, M., Galasso, M., Cozzi, C., Sgambelluri, F., Altomare, A., Cigni, C., Frigerio, E. et al., Blood to skin recirculation of CD4(+) memory T cells associates with cutaneous and systemic manifestations of psoriatic disease. Clin. Immunol. 2017. 180: 84-94.
Penkava, F., Velasco-Herrera, M. D. C., Young, M. D., Yager, N., Nwosu, L. N., Pratt, A. G., Lara, A. L. et al., Single-cell sequencing reveals clonal expansions of pro-inflammatory synovial CD8 T cells expressing tissue-homing receptors in psoriatic arthritis. Nat. Commun. 2020. 11: 4767.
van der Fits, L., Mourits, S., Voerman, J. S., Kant, M., Boon, L., Laman, J. D., Cornelissen, F. et al., Imiquimod-induced psoriasis-like skin inflammation in mice is mediated via the IL-23/IL-17 axis. J. Immunol. 2009. 182: 5836-5845.
Azukizawa, H., Kosaka, H., Sano, S., Heath, W. R., Takahashi, I., Gao, X. H., Sumikawa, Y. et al., Induction of T-cell-mediated skin disease specific for antigen transgenically expressed in keratinocytes. Eur. J. Immunol. 2003. 33: 1879-1888.
Ramirez-Valle, F., Gray, E. E. and Cyster, J. G., Inflammation induces dermal Vgamma4+ gammadeltaT17 memory-like cells that travel to distant skin and accelerate secondary IL-17-driven responses. Proc. Natl. Acad. Sci. U. S. A. 2015. 112: 8046-8051.
Schaper, K., Dickhaut, J., Japtok, L., Kietzmann, M., Mischke, R., Kleuser, B. and Baumer, W., Sphingosine-1-phosphate exhibits anti-proliferative and anti-inflammatory effects in mouse models of psoriasis. J. Dermatol. Sci. 2013. 71: 29-36.
Cheuk, S., Wiken, M., Blomqvist, L., Nylen, S., Talme, T., Stahle, M. and Eidsmo, L., Epidermal Th22 and Tc17 cells form a localized disease memory in clinically healed psoriasis. J. Immunol. 2014. 192: 3111-3120.
Serezal, I. G., Hoffer, E., Ignatov, B., Martini, E., Zitti, B., Ehrstrom, M. and Eidsmo, L., A skewed pool of resident T cells triggers psoriasis-associated tissue responses in never-lesional skin from patients with psoriasis. J. Allergy Clin. Immunol. 2019. 143: 1444-1454.
Choi, J. W., Gardell, S. E., Herr, D. R., Rivera, R., Lee, C. W., Noguchi, K., Teo, S. T. et al., FTY720 (fingolimod) efficacy in an animal model of multiple sclerosis requires astrocyte sphingosine 1-phosphate receptor 1 (S1P1) modulation. Proc. Natl. Acad. Sci. U. S. A. 2011. 108: 751-756.
Casciano, F., Diani, M., Altomare, A., Granucci, F., Secchiero, P., Banfi, G. and Reali, E., CCR4(+) skin-tropic phenotype as a feature of central memory CD8(+) T cells in healthy subjects and psoriasis patients. Front. Immunol. 2020. 11: 529.
Nolz, J. C., Molecular mechanisms of CD8(+) T cell trafficking and localization. Cell. Mol. Life Sci. 2015. 72: 2461-2473.
Wirth, T. C., Xue, H. H., Rai, D., Sabel, J. T., Bair, T., Harty, J. T. and Badovinac, V. P., Repetitive antigen stimulation induces stepwise transcriptome diversification but preserves a core signature of memory CD8(+) T cell differentiation. Immunity 2010. 33: 128-140.
Slutter, B., Pewe, L. L., Kaech, S. M. and Harty, J. T., Lung airway-surveilling CXCR3(hi) memory CD8(+) T cells are critical for protection against influenza A virus. Immunity 2013. 39: 939-948.
Mueller, S. N., Gebhardt, T., Carbone, F. R. and Heath, W. R., Memory T cell subsets, migration patterns, and tissue residence. Annu. Rev. Immunol. 2013. 31: 137-161.
Klicznik, M. M., Morawski, P. A., Hollbacher, B., Varkhande, S. R., Motley, S. J., Kuri-Cervantes, L., Goodwin, E. et al., Human CD4(+)CD103(+) cutaneous resident memory T cells are found in the circulation of healthy individuals. Sci. Immunol. 2019. 4: eaav8995.
Matsuo, K., Kitahata, K., Kaibori, Y., Arima, Y., Iwama, A., Ito, M., Hara, Y. et al., CCR4 involvement in the expansion of T helper type 17 cells in a mouse model of psoriasis. J. Invest. Dermatol. 2021. 141: 1985-1994.
Bos, F., Capsoni, F., Molteni, S., Raeli, L., Diani, M., Altomare, A., Garavaglia, M. et al., Differential expression of interleukin-2 by anti-CD3-stimulated peripheral blood mononuclear cells in patients with psoriatic arthritis and patients with cutaneous psoriasis. Clin. Exp. Dermatol. 2014. 39: 385-390.
Maurice, N. J., McElrath, M. J., Andersen-Nissen, E., Frahm, N. and Prlic, M., CXCR3 enables recruitment and site-specific bystander activation of memory CD8(+) T cells. Nat. Commun. 2019. 10: 4987.
Li, Q., Yan, Y., Liu, J., Huang, X., Zhang, X., Kirschning, C., Xu, H. C. et al., Toll-like receptor 7 activation enhances CD8+ T cell effector functions by promoting cellular glycolysis. Front. Immunol. 2019. 10: 2191.
Fenix, K., Wijesundara, D. K., Cowin, A. J., Grubor-Bauk, B. and Kopecki, Z., Immunological memory in imiquimod-induced murine model of psoriasiform dermatitis. Int. J. Mol. Sci. 2020. 21: 7228.
Curran, S. A., FitzGerald, O. M., Costello, P. J., Selby, J. M., Kane, D. J., Bresnihan, B. and Winchester, R., Nucleotide sequencing of psoriatic arthritis tissue before and during methotrexate administration reveals a complex inflammatory T cell infiltrate with very few clones exhibiting features that suggest they drive the inflammatory process by recognizing autoantigens. J. Immunol. 2004. 172: 1935-1944.
Nelson, D., Bundell, C. and Robinson, B., In vivo cross-presentation of a soluble protein antigen: kinetics, distribution, and generation of effector CTL recognizing dominant and subdominant epitopes. J. Immunol. 2000. 165: 6123-6132.
Vanderlugt, C. L. and Miller, S. D., Epitope spreading in immune-mediated diseases: implications for immunotherapy. Nat. Rev. Immunol. 2002. 2: 85-95.
Hao, Y., Hao, S., Andersen-Nissen, E., Mauck, W. M., 3rd, Zheng, S., Butler, A., Lee, M. J. et al., Integrated analysis of multimodal single-cell data. Cell 2021. 184: 3573 e3529-3587 e3529.
Hafemeister, C. and Satija, R., Normalization and variance stabilization of single-cell RNA-seq data using regularized negative binomial regression. Genome Biol. 2019. 20: 296.
Korsunsky, I., Millard, N., Fan, J., Slowikowski, K., Zhang, F., Wei, K., Baglaenko, Y. et al., Fast, sensitive and accurate integration of single-cell data with Harmony. Nat. Methods 2019. 16: 1289-1296.
Aran, D., Looney, A. P., Liu, L., Wu, E., Fong, V., Hsu, A., Chak, S. et al., Reference-based analysis of lung single-cell sequencing reveals a transitional profibrotic macrophage. Nat. Immunol. 2019. 20: 163-172.
Martens, J. H. and Stunnenberg, H. G., BLUEPRINT: mapping human blood cell epigenomes. Haematologica. 2013. 98: 1487-1489.
Consortium, E. P., An integrated encyclopedia of DNA elements in the human genome. Nature 2012. 489: 57-74.
Borcherding, N., Bormann, N. L. and Kraus, G., scRepertoire: an R-based toolkit for single-cell immune receptor analysis. F1000Res. 2020. 9: 47.

Auteurs

Guendalina Montico (G)

IRCCS Istituto Ortopedico Galeazzi, Milan, Italy.

Francesca Mingozzi (F)

Department of Biotechnology and Biosciences, University of Milano-Bicocca, Milan, Italy.

Fabio Casciano (F)

Department of Translational Medicine and LTTA Centre, University of Ferrara, Ferrara, Italy.
Interdepartmental Research Center for the Study of Multiple Sclerosis and Inflammatory and Degenerative Diseases of the Nervous System, University of Ferrara, Ferrara, Italy.

Giulia Protti (G)

Department of Biotechnology and Biosciences, University of Milano-Bicocca, Milan, Italy.
National Institute of Molecular Genetics "Romeo ed Enrica Invernizzi" (INGM), Milan, Italy.

Laura Gornati (L)

Department of Biotechnology and Biosciences, University of Milano-Bicocca, Milan, Italy.

Erika Marzola (E)

Department of Chemical Pharmaceutical and Agricultural Sciences, University of Ferrara, Ferrara, Italy.

Giuseppe Banfi (G)

IRCCS Istituto Ortopedico Galeazzi, Milan, Italy.

Remo Guerrini (R)

Department of Chemical Pharmaceutical and Agricultural Sciences, University of Ferrara, Ferrara, Italy.

Paola Secchiero (P)

Department of Translational Medicine and LTTA Centre, University of Ferrara, Ferrara, Italy.

Stefano Volinia (S)

Department of Translational Medicine and LTTA Centre, University of Ferrara, Ferrara, Italy.
Biological and Chemical Research Centre (CNBCh UW), University of Warsaw, Warsaw, Poland.

Francesca Granucci (F)

Department of Biotechnology and Biosciences, University of Milano-Bicocca, Milan, Italy.

Eva Reali (E)

Department of Biotechnology and Biosciences, University of Milano-Bicocca, Milan, Italy.

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