Spironolactone effect on circulating procollagen type I carboxy-terminal propeptide: Pooled analysis of three randomized trials.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
15 04 2023
Historique:
received: 28 11 2022
revised: 24 01 2023
accepted: 31 01 2023
pubmed: 5 2 2023
medline: 16 3 2023
entrez: 4 2 2023
Statut: ppublish

Résumé

Spironolactone might improve the prognosis of patients with heart failure with preserved left ventricular ejection fraction (HFpEF), but the mechanisms by which it acts are uncertain. Serum concentrations of procollagen type I carboxy-terminal propeptide (PICP) reflect the synthesis of type I collagen and correlate well with histologically proven cardiac fibrosis. To investigate the effect of spironolactone on serum PICP concentration in patients with stage B and C HFpEF across three trials (HOMAGE, ALDO-DHF, and TOPCAT) for which measurements of serum PICP were available. Random-effects meta-analysis. A total of 1038 patients with PICP measurements available both at baseline and 9-12 months were included in this analysis: 488 (47.0%) from HOMAGE, 386 (37.2%) from ALDO-DHF, and 164 (15.8%) from TOPCAT. The median (percentile Spironolactone reduced serum concentrations of PICP in patients with HFpEF with different severity and stages of disease. These findings are consistent with spironolactone having an anti-fibrotic effect.

Sections du résumé

BACKGROUND
Spironolactone might improve the prognosis of patients with heart failure with preserved left ventricular ejection fraction (HFpEF), but the mechanisms by which it acts are uncertain. Serum concentrations of procollagen type I carboxy-terminal propeptide (PICP) reflect the synthesis of type I collagen and correlate well with histologically proven cardiac fibrosis.
AIMS
To investigate the effect of spironolactone on serum PICP concentration in patients with stage B and C HFpEF across three trials (HOMAGE, ALDO-DHF, and TOPCAT) for which measurements of serum PICP were available.
METHODS
Random-effects meta-analysis.
RESULTS
A total of 1038 patients with PICP measurements available both at baseline and 9-12 months were included in this analysis: 488 (47.0%) from HOMAGE, 386 (37.2%) from ALDO-DHF, and 164 (15.8%) from TOPCAT. The median (percentile
CONCLUSIONS
Spironolactone reduced serum concentrations of PICP in patients with HFpEF with different severity and stages of disease. These findings are consistent with spironolactone having an anti-fibrotic effect.

Identifiants

pubmed: 36738846
pii: S0167-5273(23)00171-7
doi: 10.1016/j.ijcard.2023.01.088
pii:
doi:

Substances chimiques

Spironolactone 27O7W4T232
Procollagen Type I 0
Procollagen 0
Peptide Fragments 0

Types de publication

Meta-Analysis Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

86-88

Informations de copyright

Copyright © 2023 Elsevier B.V. All rights reserved.

Auteurs

João Pedro Ferreira (JP)

Cardiovascular R&D Centre - UnIC@RISE, Department of Physiology and Cardiothoracic Surgery, Faculty of Medicine of the University of Porto, Porto, Portugal & Internal Medicine Departament, Centro Hospitalar de Vila Nova de Gaia/Espinho, Vila Nova de Gaia, Portugal; Université de Lorraine, Inserm, Centre d'Investigation Clinique Plurithématique 1433, U1116, CHRU de Nancy, F-CRIN INI-CRCT, Nancy, France. Electronic address: jpferreira@med.up.pt.

John G Cleland (JG)

School of Cardiovascular and Metabolic Health, University of Glasgow, Glasgow, UK.

Nicolas Girerd (N)

Université de Lorraine, Inserm, Centre d'Investigation Clinique Plurithématique 1433, U1116, CHRU de Nancy, F-CRIN INI-CRCT, Nancy, France.

Patrick Rossignol (P)

Université de Lorraine, Inserm, Centre d'Investigation Clinique Plurithématique 1433, U1116, CHRU de Nancy, F-CRIN INI-CRCT, Nancy, France.

Pierpaolo Pellicori (P)

School of Cardiovascular and Metabolic Health, University of Glasgow, Glasgow, UK.

Franco Cosmi (F)

Department of Cardiology, Cortona Hospital, Arezzo, Italy.

Beatrice Mariottoni (B)

Department of Cardiology, Cortona Hospital, Arezzo, Italy.

Arantxa González (A)

Program of Cardiovascular Diseases, CIMA, Universidad de Navarra and IdiSNA, Pamplona, Spain; CIBERCV, Carlos III Institute of Health, Madrid, Spain.

Javier Diez (J)

Program of Cardiovascular Diseases, CIMA, Universidad de Navarra and IdiSNA, Pamplona, Spain; CIBERCV, Carlos III Institute of Health, Madrid, Spain.

Scott D Solomon (SD)

Division of Cardiovascular Medicine, Brigham and Women's Hospital, Boston, MA, USA.

Brian Claggett (B)

Division of Cardiovascular Medicine, Brigham and Women's Hospital, Boston, MA, USA.

Marc A Pfeffer (MA)

Division of Cardiovascular Medicine, Brigham and Women's Hospital, Boston, MA, USA.

Bertram Pitt (B)

Division of Cardiology, University of Michigan, Ann Arbor, MI, USA.

Johannes Petutschnigg (J)

Department of Internal Medicine and Cardiology, Campus Virchow Klinikum, Charité University Medicine Berlin, Berlin, Germany; German Centre for Cardiovascular Research (DZHK), Partner Site Berlin, Berlin, Germany.

Burkert Pieske (B)

Department of Internal Medicine and Cardiology, Campus Virchow Klinikum, Charité University Medicine Berlin, Berlin, Germany; German Centre for Cardiovascular Research (DZHK), Partner Site Berlin, Berlin, Germany.

Frank Edelmann (F)

Department of Internal Medicine and Cardiology, Campus Virchow Klinikum, Charité University Medicine Berlin, Berlin, Germany; German Centre for Cardiovascular Research (DZHK), Partner Site Berlin, Berlin, Germany.

Faiez Zannad (F)

Université de Lorraine, Inserm, Centre d'Investigation Clinique Plurithématique 1433, U1116, CHRU de Nancy, F-CRIN INI-CRCT, Nancy, France.

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Classifications MeSH