How does neurokinin 3 receptor agonism affect pathological and cognitive impairments in an Alzheimer's disease-like rat model?


Journal

Amino acids
ISSN: 1438-2199
Titre abrégé: Amino Acids
Pays: Austria
ID NLM: 9200312

Informations de publication

Date de publication:
Apr 2023
Historique:
received: 22 08 2022
accepted: 20 01 2023
medline: 1 5 2023
pubmed: 7 2 2023
entrez: 6 2 2023
Statut: ppublish

Résumé

Alzheimer's disease (AD) is accepted as a form of progressive dementia. Cholinergic systems are commonly affected in AD. Neurokinin 3 receptor (NK3R) is involved in learning memory-related processes. It is known that the activation of NK3R affects the release of many neurotransmitters. The aim of this project was to investigate the effects of NK3R agonist senktide administration on neurobehavioral mechanisms in the experimental AD-like rat model. 50 male Wistar albino rats were divided into Control (C), AD, Control + NK3R agonist (CS), AD + NK3R agonist (ADS), AD + NK3Ragonist + antagonist groups (ADSO). We designed AD-like model by intrahippocampal administration of Aβ1-42. After NK3R agonist + antagonist injections, open field (OF), Morris water maze (MWM) tests were applied. Cholinergic mechanism analysis from hippocampus-cortex tissues was performed by ELISA and catecholamine analysis from brain stem tissue were performed by HPLC method. The transitions from edge to center, rearing, grooming parameters were found to be reduced in final values of OF. While the group-time interaction was significant in the OF test findings, there was no significant difference between the groups. In MWM test, ADS group showed a learning level close to control group and animals in AD and ADSO groups could not learn target quadrant in MWM test. The brain stem NA and DA concentrations were not statistically significant. Hippocampal AChE-ChAT levels were supported by positive effects of senktide on learning via the cholinergic mechanisms. As a result, NK3R agonists were found to be effective in improving cognitive functions in rats with AD pathology. In the experimental AD model, positive effects of NK3R on learning memory may be mediated by cholinergic mechanisms.

Identifiants

pubmed: 36745246
doi: 10.1007/s00726-023-03241-0
pii: 10.1007/s00726-023-03241-0
doi:

Substances chimiques

Receptors, Neurokinin-3 0
Cholinergic Agents 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

481-498

Subventions

Organisme : Necmettin Erbakan Üniversitesi
ID : 161418005

Informations de copyright

© 2023. The Author(s), under exclusive licence to Springer-Verlag GmbH Austria, part of Springer Nature.

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Auteurs

Raviye Ozen Koca (RO)

Department of Physiology, Meram Faculty of Medicine, Necmettin Erbakan University, Konya, Turkey.

Z Isik Solak Gormus (ZIS)

Department of Physiology, Meram Faculty of Medicine, Necmettin Erbakan University, Konya, Turkey. igormus@gmail.com.

Hatice Solak (H)

Department of Physiology, Faculty of Medicine, Kutahya Health Science University, Kutahya, Turkey.

Aynur Koc (A)

Department of Physiology, Faculty of Medicine, Hitit University, Corum, Turkey.

İbrahim Kılınc (İ)

Department of Medical Education and Informatics, Meram Faculty of Medicine, Necmettin Erbakan University, Konya, Turkey.

Mehmet Sinan İyisoy (MS)

Department of Biochemistry, Meram Faculty of Medicine, Necmettin Erbakan University, Konya, Turkey.

Selim Kutlu (S)

Department of Physiology, Meram Faculty of Medicine, Necmettin Erbakan University, Konya, Turkey.

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