Hyperactivity of the CD155 immune checkpoint suppresses anti-viral immunity in patients with coronary artery disease.


Journal

Nature cardiovascular research
ISSN: 2731-0590
Titre abrégé: Nat Cardiovasc Res
Pays: England
ID NLM: 9918284280206676

Informations de publication

Date de publication:
Jul 2022
Historique:
entrez: 2 3 2023
pubmed: 3 3 2023
medline: 3 3 2023
Statut: ppublish

Résumé

Pre-existent cardiovascular disease is a risk factor for weak anti-viral immunity, but underlying mechanisms remain undefined. Here, we report that patients with coronary artery disease (CAD) have macrophages (Mϕ) that actively suppress the induction of helper T cells reactive to two viral antigens: the SARS-CoV2 Spike protein and the Epstein-Barr virus (EBV) glycoprotein 350. CAD Mϕ overexpressed the methyltransferase METTL3, promoting the accumulation of N⁶-methyladenosine (m6A) in Poliovirus receptor (CD155) mRNA. m6A modifications of positions 1635 and 3103 in the 3'UTR of CD155 mRNA stabilized the transcript and enhanced CD155 surface expression. As a result, the patients' Mϕ abundantly expressed the immunoinhibitory ligand CD155 and delivered negative signals to CD4

Identifiants

pubmed: 36860353
doi: 10.1038/s44161-022-00096-8
pmc: PMC9974158
mid: NIHMS1825134
doi:

Types de publication

Journal Article

Langues

eng

Pagination

634-648

Subventions

Organisme : NIAID NIH HHS
ID : R01 AI129191
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL142068
Pays : United States
Organisme : NIAID NIH HHS
ID : R01 AI108906
Pays : United States
Organisme : NIAID NIH HHS
ID : R01 AI108891
Pays : United States
Organisme : NHLBI NIH HHS
ID : P01 HL129941
Pays : United States
Organisme : NIAID NIH HHS
ID : U19 AI057266
Pays : United States
Organisme : NIAMS NIH HHS
ID : R01 AR042527
Pays : United States
Organisme : NIA NIH HHS
ID : R01 AG045779
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL117913
Pays : United States

Déclaration de conflit d'intérêts

Declaration of Interests: The authors have declared that no conflict of interest exists.

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Auteurs

Tuantuan V Zhao (TV)

Department of Medicine, Mayo Clinic Alix School of Medicine, Rochester, MN 55901, USA.

Zhaolan Hu (Z)

Department of Medicine, Mayo Clinic Alix School of Medicine, Rochester, MN 55901, USA.

Shozo Ohtsuki (S)

Department of Medicine, Mayo Clinic Alix School of Medicine, Rochester, MN 55901, USA.

Ke Jin (K)

Department of Medicine, Mayo Clinic Alix School of Medicine, Rochester, MN 55901, USA.

Bowen Wu (B)

Department of Medicine, Mayo Clinic Alix School of Medicine, Rochester, MN 55901, USA.

Gerald J Berry (GJ)

Department of Pathology, Stanford University School of Medicine, Stanford, CA, 94305, USA.

Robert L Frye (RL)

Department of Medicine, Mayo Clinic Alix School of Medicine, Rochester, MN 55901, USA.

Jörg J Goronzy (JJ)

Department of Medicine, Mayo Clinic Alix School of Medicine, Rochester, MN 55901, USA.
Department of Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA.

Cornelia M Weyand (CM)

Department of Medicine, Mayo Clinic Alix School of Medicine, Rochester, MN 55901, USA.
Department of Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA.

Classifications MeSH