The multi-kinase inhibitor CG-806 exerts anti-cancer activity against acute myeloid leukemia by co-targeting FLT3, BTK, and Aurora kinases.

CG-806 FLT3 Multi-kinase inhibitor acute myeloid leukemia

Journal

Research square
Titre abrégé: Res Sq
Pays: United States
ID NLM: 101768035

Informations de publication

Date de publication:
22 Feb 2023
Historique:
pubmed: 4 3 2023
medline: 4 3 2023
entrez: 3 3 2023
Statut: epublish

Résumé

Despite the development of several FLT3 inhibitors that have improved outcomes in patients with FLT3-mutant acute myeloid leukemias (AML), drug resistance is frequently observed, which may be associated with the activation of additional pro-survival pathways such as those regulated by BTK, aurora kinases, and potentially others in addition to acquired tyrosine kinase domains (TKD) mutations of To evaluate the anti-leukemia efficacy of the novel multi-kinase inhibitor CG-806, which targets FLT3 and other kinases, in order to circumvent drug resistance and target The anti-leukemia activity of CG-806 was investigated by measuring apoptosis induction and analyzing cell cycle with flow cytometry CG-806 demonstrated superior anti-leukemia efficacy compared to commercially available FLT3 inhibitors, both The results of this study suggest that CG-806 is a promising multi-kinase inhibitor with anti-leukemia efficacy, regardless of

Sections du résumé

Background UNASSIGNED
Despite the development of several FLT3 inhibitors that have improved outcomes in patients with FLT3-mutant acute myeloid leukemias (AML), drug resistance is frequently observed, which may be associated with the activation of additional pro-survival pathways such as those regulated by BTK, aurora kinases, and potentially others in addition to acquired tyrosine kinase domains (TKD) mutations of
Objective UNASSIGNED
To evaluate the anti-leukemia efficacy of the novel multi-kinase inhibitor CG-806, which targets FLT3 and other kinases, in order to circumvent drug resistance and target
Methods UNASSIGNED
The anti-leukemia activity of CG-806 was investigated by measuring apoptosis induction and analyzing cell cycle with flow cytometry
Results UNASSIGNED
CG-806 demonstrated superior anti-leukemia efficacy compared to commercially available FLT3 inhibitors, both
Conclusion UNASSIGNED
The results of this study suggest that CG-806 is a promising multi-kinase inhibitor with anti-leukemia efficacy, regardless of

Identifiants

pubmed: 36865133
doi: 10.21203/rs.3.rs-2570204/v1
pmc: PMC9980215
pii:
doi:

Banques de données

ClinicalTrials.gov
['NCT04477291']

Types de publication

Preprint

Langues

eng

Déclaration de conflit d'intérêts

Disclosure of Conflicts of Interest: H.Z. and W.R. are employees of Aptose Biosciences; M.A. serves on the Aptose Biosciences Scientific Advisory Board.

Auteurs

Guopan Yu (G)

The University of Texas MD Anderson Cancer Center.

Weiguo Zhang (W)

University of Texas MD Anderson Cancer Center.

Hongying Zhang (H)

Aptose Biosciences.

Charlie Ly (C)

The University of Texas MD Anderson Cancer Center.

Mahesh Basyal (M)

The University of Texas MD Anderson Cancer Center.

William G Rice (WG)

Aptose Biosciences.

Michael Andreeff (M)

The University of Texas MD Anderson Cancer Center.

Classifications MeSH