Diabetes mellitus and hemodynamics in advanced heart failure.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
15 05 2023
Historique:
received: 30 01 2023
revised: 24 02 2023
accepted: 05 03 2023
medline: 11 4 2023
pubmed: 13 3 2023
entrez: 12 3 2023
Statut: ppublish

Résumé

The presence of diabetes in patients with heart failure (HF) is associated with a worse prognosis. It is unclear if hemodynamics in HF patients with DM differ from those of non-diabetic patients and how this might influence outcome. This study aims to discover the impact of DM on hemodynamics in HF patients. Consecutive patients (n = 598) with HF and reduced ejection fraction (LVEF ≤40%) undergoing invasive hemodynamic evaluation were included (non-DM: n = 473, DM: n = 125). Hemodynamic parameters included pulmonary capillary wedge pressure (PCWP), central venous pressure (CVP), cardiac index (CI) and mean arterial pressure (MAP). Mean follow-up was 9.5 ± 5.1 years. Patients with DM (82.7% male, mean age 57.1 ± 10.1 years, mean HbA1c 60 ± 21 mmol/mol) had higher PCWP, mPAP, CVP and higher MAP. Adjusted analysis demonstrated that DM patients had higher PCWP and CVP. Increasing HbA1c-values were correlated with higher PCWP (p = 0.017) and CVP (p = 0.043). Patients with DM, especially those with poor glycemic control, have higher filling pressures. This may be a feature of diabetic cardiomyopathy, however, other unknown mechanisms beyond hemodynamic factors are likely to explain the increased mortality associated with diabetes in HF.

Sections du résumé

BACKGROUND
The presence of diabetes in patients with heart failure (HF) is associated with a worse prognosis. It is unclear if hemodynamics in HF patients with DM differ from those of non-diabetic patients and how this might influence outcome. This study aims to discover the impact of DM on hemodynamics in HF patients.
METHODS
Consecutive patients (n = 598) with HF and reduced ejection fraction (LVEF ≤40%) undergoing invasive hemodynamic evaluation were included (non-DM: n = 473, DM: n = 125). Hemodynamic parameters included pulmonary capillary wedge pressure (PCWP), central venous pressure (CVP), cardiac index (CI) and mean arterial pressure (MAP). Mean follow-up was 9.5 ± 5.1 years.
RESULTS
Patients with DM (82.7% male, mean age 57.1 ± 10.1 years, mean HbA1c 60 ± 21 mmol/mol) had higher PCWP, mPAP, CVP and higher MAP. Adjusted analysis demonstrated that DM patients had higher PCWP and CVP. Increasing HbA1c-values were correlated with higher PCWP (p = 0.017) and CVP (p = 0.043).
CONCLUSION
Patients with DM, especially those with poor glycemic control, have higher filling pressures. This may be a feature of diabetic cardiomyopathy, however, other unknown mechanisms beyond hemodynamic factors are likely to explain the increased mortality associated with diabetes in HF.

Identifiants

pubmed: 36907448
pii: S0167-5273(23)00341-8
doi: 10.1016/j.ijcard.2023.03.015
pii:
doi:

Substances chimiques

Glycated Hemoglobin 0

Types de publication

Journal Article Comment

Langues

eng

Sous-ensembles de citation

IM

Pagination

60-65

Commentaires et corrections

Type : CommentOn

Informations de copyright

Copyright © 2023 The Author(s). Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest BH and TD: Nothing to declare. KR: Advisor: Abbott, Boehringer Ingelheim and NovoNordisk. Speaker's fee: Astra-Zeneca, Orion Pharma, Novartis, Vifor Pharm. ME: Speaker's fee: Orion Pharma. CK: Advisor: Amicus, Sanofi Genozyme, Astra Zeneca, Boehringer Ingelheim. Speakers fee: Astra Zeneca, Amicus, Sanofi Genozyme, Boehringer Ingelheim, Novo Nordisk. FG: Advisor: Abbott, Ionis, Alnylam, Astra-Zeneca, Pfizer; Research grants. Pfizer. Speaker's fee: Novartis, Vifor Pharma

Auteurs

Benedicte Heegaard (B)

Department of Cardiology, Copenhagen University Hospital, Rigshospitalet, Denmark.

Tania Deis (T)

Department of Cardiology, Copenhagen University Hospital, Rigshospitalet, Denmark.

Kasper Rossing (K)

Department of Cardiology, Copenhagen University Hospital, Rigshospitalet, Denmark.

Mads Ersbøll (M)

Department of Cardiology, Copenhagen University Hospital, Rigshospitalet, Denmark.

Caroline Kistorp (C)

Department of Endocrinology, Copenhagen University Hospital, Rigshospitalet, Denmark; Department of Clinical Medicine, University of Copenhagen, Denmark.

Finn Gustafsson (F)

Department of Cardiology, Copenhagen University Hospital, Rigshospitalet, Denmark; Department of Clinical Medicine, University of Copenhagen, Denmark. Electronic address: finn.gustafsson@regionh.dk.

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