The physiologic response to epinephrine and pediatric cardiopulmonary resuscitation outcomes.


Journal

Critical care (London, England)
ISSN: 1466-609X
Titre abrégé: Crit Care
Pays: England
ID NLM: 9801902

Informations de publication

Date de publication:
13 03 2023
Historique:
received: 28 12 2022
accepted: 08 03 2023
entrez: 14 3 2023
pubmed: 15 3 2023
medline: 16 3 2023
Statut: epublish

Résumé

Epinephrine is provided during cardiopulmonary resuscitation (CPR) to increase systemic vascular resistance and generate higher diastolic blood pressure (DBP) to improve coronary perfusion and attain return of spontaneous circulation (ROSC). The DBP response to epinephrine during pediatric CPR and its association with outcomes have not been well described. Thus, the objective of this study was to measure the association between change in DBP after epinephrine administration during CPR and ROSC. This was a prospective multicenter study of children receiving ≥ 1 min of CPR with ≥ 1 dose of epinephrine and evaluable invasive arterial BP data in the 18 ICUs of the ICU-RESUS trial (NCT02837497). Blood pressure waveforms underwent compression-by-compression quantitative analysis. The mean DBP before first epinephrine dose was compared to mean DBP two minutes post-epinephrine. Patients with ≥ 5 mmHg increase in DBP were characterized as "responders." Among 147 patients meeting inclusion criteria, 66 (45%) were characterized as responders and 81 (55%) were non-responders. The mean increase in DBP with epinephrine was 4.4 [- 1.9, 11.5] mmHg (responders: 13.6 [7.5, 29.3] mmHg versus non-responders: - 1.5 [- 5.0, 1.5] mmHg; p < 0.001). After controlling for a priori selected covariates, epinephrine response was associated with ROSC (aRR 1.60 [1.21, 2.12]; p = 0.001). Sensitivity analyses identified similar associations between DBP response thresholds of ≥ 10, 15, and 20 mmHg and ROSC; DBP responses of ≥ 10 and ≥ 15 mmHg were associated with higher aRR of survival to hospital discharge and survival with favorable neurologic outcome (Pediatric Cerebral Performance Category score of 1-3 or no worsening from baseline). The change in DBP following epinephrine administration during pediatric in-hospital CPR was associated with return of spontaneous circulation.

Sections du résumé

BACKGROUND
Epinephrine is provided during cardiopulmonary resuscitation (CPR) to increase systemic vascular resistance and generate higher diastolic blood pressure (DBP) to improve coronary perfusion and attain return of spontaneous circulation (ROSC). The DBP response to epinephrine during pediatric CPR and its association with outcomes have not been well described. Thus, the objective of this study was to measure the association between change in DBP after epinephrine administration during CPR and ROSC.
METHODS
This was a prospective multicenter study of children receiving ≥ 1 min of CPR with ≥ 1 dose of epinephrine and evaluable invasive arterial BP data in the 18 ICUs of the ICU-RESUS trial (NCT02837497). Blood pressure waveforms underwent compression-by-compression quantitative analysis. The mean DBP before first epinephrine dose was compared to mean DBP two minutes post-epinephrine. Patients with ≥ 5 mmHg increase in DBP were characterized as "responders."
RESULTS
Among 147 patients meeting inclusion criteria, 66 (45%) were characterized as responders and 81 (55%) were non-responders. The mean increase in DBP with epinephrine was 4.4 [- 1.9, 11.5] mmHg (responders: 13.6 [7.5, 29.3] mmHg versus non-responders: - 1.5 [- 5.0, 1.5] mmHg; p < 0.001). After controlling for a priori selected covariates, epinephrine response was associated with ROSC (aRR 1.60 [1.21, 2.12]; p = 0.001). Sensitivity analyses identified similar associations between DBP response thresholds of ≥ 10, 15, and 20 mmHg and ROSC; DBP responses of ≥ 10 and ≥ 15 mmHg were associated with higher aRR of survival to hospital discharge and survival with favorable neurologic outcome (Pediatric Cerebral Performance Category score of 1-3 or no worsening from baseline).
CONCLUSIONS
The change in DBP following epinephrine administration during pediatric in-hospital CPR was associated with return of spontaneous circulation.

Identifiants

pubmed: 36915182
doi: 10.1186/s13054-023-04399-5
pii: 10.1186/s13054-023-04399-5
pmc: PMC10012560
doi:

Substances chimiques

Epinephrine YKH834O4BH

Types de publication

Multicenter Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

105

Subventions

Organisme : NHLBI NIH HHS
ID : R01 HL131544
Pays : United States
Organisme : NICHD NIH HHS
ID : RL1 HD107777
Pays : United States

Investigateurs

Tageldin Ahmed (T)
Michael J Bell (MJ)
Robert Bishop (R)
Matthew Bochkoris (M)
Candice Burns (C)
Joseph A Carcillo (JA)
J Michael Dean (JM)
J Wesley Diddle (JW)
Myke Federman (M)
Richard Fernandez (R)
Ericka L Fink (EL)
Stuart H Friess (SH)
Mark Hall (M)
David A Hehir (DA)
Christopher M Horvat (CM)
Leanna L Huard (LL)
Tensing Maa (T)
Arushi Manga (A)
Patrick S McQuillen (PS)
Peter M Mourani (PM)
Daniel Notterman (D)
Murray M Pollack (MM)
Anil Sapru (A)
Carleen Schneiter (C)
Matthew P Sharron (MP)
Neeraj Srivastava (N)
Sarah Tabbutt (S)
Shirley Viteri (S)
David Wessel (D)
Andrew R Yates (AR)
Athena F Zuppa (AF)

Informations de copyright

© 2023. The Author(s).

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Auteurs

Ryan W Morgan (RW)

Department of Anesthesiology and Critical Care Medicine, The Children's Hospital of Philadelphia, University of Pennsylvania, 3401 Civic Center Boulevard, Wood Building - 6104, Philadelphia, PA, 19104, USA. morganR1@chop.edu.

Robert A Berg (RA)

Department of Anesthesiology and Critical Care Medicine, The Children's Hospital of Philadelphia, University of Pennsylvania, 3401 Civic Center Boulevard, Wood Building - 6104, Philadelphia, PA, 19104, USA.

Ron W Reeder (RW)

Department of Pediatrics, University of Utah, Salt Lake City, UT, USA.

Todd C Carpenter (TC)

Department of Pediatrics, University of Colorado School of Medicine and Children's Hospital Colorado, Aurora, CO, USA.

Deborah Franzon (D)

Department of Pediatrics, Benioff Children's Hospital, University of California, San Francisco, San Francisco, CA, USA.

Aisha H Frazier (AH)

Nemours Cardiac Center, Nemours/Alfred I. duPont Hospital for Children, Wilmington, DE, USA.
Department of Pediatrics, Sidney Kimmel Medical College, Thomas Jefferson University, Philadelphia, PA, USA.

Kathryn Graham (K)

Department of Anesthesiology and Critical Care Medicine, The Children's Hospital of Philadelphia, University of Pennsylvania, 3401 Civic Center Boulevard, Wood Building - 6104, Philadelphia, PA, 19104, USA.

Kathleen L Meert (KL)

Department of Pediatrics, Children's Hospital of Michigan, Central Michigan University, Detroit, MI, USA.

Vinay M Nadkarni (VM)

Department of Anesthesiology and Critical Care Medicine, The Children's Hospital of Philadelphia, University of Pennsylvania, 3401 Civic Center Boulevard, Wood Building - 6104, Philadelphia, PA, 19104, USA.

Maryam Y Naim (MY)

Department of Anesthesiology and Critical Care Medicine, The Children's Hospital of Philadelphia, University of Pennsylvania, 3401 Civic Center Boulevard, Wood Building - 6104, Philadelphia, PA, 19104, USA.

Bradley Tilford (B)

Department of Pediatrics, Children's Hospital of Michigan, Central Michigan University, Detroit, MI, USA.

Heather A Wolfe (HA)

Department of Anesthesiology and Critical Care Medicine, The Children's Hospital of Philadelphia, University of Pennsylvania, 3401 Civic Center Boulevard, Wood Building - 6104, Philadelphia, PA, 19104, USA.

Andrew R Yates (AR)

Department of Pediatrics, Nationwide Children's Hospital, The Ohio State University, Columbus, OH, USA.

Robert M Sutton (RM)

Department of Anesthesiology and Critical Care Medicine, The Children's Hospital of Philadelphia, University of Pennsylvania, 3401 Civic Center Boulevard, Wood Building - 6104, Philadelphia, PA, 19104, USA.

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