A longitudinal circulating tumor DNA-based model associated with survival in metastatic non-small-cell lung cancer.


Journal

Nature medicine
ISSN: 1546-170X
Titre abrégé: Nat Med
Pays: United States
ID NLM: 9502015

Informations de publication

Date de publication:
04 2023
Historique:
received: 29 06 2022
accepted: 23 01 2023
medline: 21 4 2023
pubmed: 18 3 2023
entrez: 17 3 2023
Statut: ppublish

Résumé

One of the great challenges in therapeutic oncology is determining who might achieve survival benefits from a particular therapy. Studies on longitudinal circulating tumor DNA (ctDNA) dynamics for the prediction of survival have generally been small or nonrandomized. We assessed ctDNA across 5 time points in 466 non-small-cell lung cancer (NSCLC) patients from the randomized phase 3 IMpower150 study comparing chemotherapy-immune checkpoint inhibitor (chemo-ICI) combinations and used machine learning to jointly model multiple ctDNA metrics to predict overall survival (OS). ctDNA assessments through cycle 3 day 1 of treatment enabled risk stratification of patients with stable disease (hazard ratio (HR) = 3.2 (2.0-5.3), P < 0.001; median 7.1 versus 22.3 months for high- versus low-intermediate risk) and with partial response (HR = 3.3 (1.7-6.4), P < 0.001; median 8.8 versus 28.6 months). The model also identified high-risk patients in an external validation cohort from the randomized phase 3 OAK study of ICI versus chemo in NSCLC (OS HR = 3.73 (1.83-7.60), P = 0.00012). Simulations of clinical trial scenarios employing our ctDNA model suggested that early ctDNA testing outperforms early radiographic imaging for predicting trial outcomes. Overall, measuring ctDNA dynamics during treatment can improve patient risk stratification and may allow early differentiation between competing therapies during clinical trials.

Identifiants

pubmed: 36928816
doi: 10.1038/s41591-023-02226-6
pii: 10.1038/s41591-023-02226-6
pmc: PMC10115641
doi:

Substances chimiques

Circulating Tumor DNA 0
Biomarkers, Tumor 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

859-868

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2023. The Author(s).

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Auteurs

Zoe June F Assaf (ZJF)

Genentech Inc., South San Francisco, CA, USA. assaf.zoe-june@gene.com.

Wei Zou (W)

Genentech Inc., South San Francisco, CA, USA.

Alexander D Fine (AD)

Foundation Medicine Inc., Cambridge, MA, USA.

Mark A Socinski (MA)

AdventHealth Cancer Institute, Orlando, FL, USA.

Amanda Young (A)

Foundation Medicine Inc., Cambridge, MA, USA.

Doron Lipson (D)

Foundation Medicine Inc., Cambridge, MA, USA.

Jonathan F Freidin (JF)

Foundation Medicine Inc., Cambridge, MA, USA.

Mark Kennedy (M)

Foundation Medicine Inc., Cambridge, MA, USA.

Eliana Polisecki (E)

Foundation Medicine Inc., Cambridge, MA, USA.

Makoto Nishio (M)

The Cancer Institute Hospital, Japanese Foundation for Cancer Research, Tokyo, Japan.

David Fabrizio (D)

Foundation Medicine Inc., Cambridge, MA, USA.

Geoffrey R Oxnard (GR)

Foundation Medicine Inc., Cambridge, MA, USA.

Craig Cummings (C)

Genentech Inc., South San Francisco, CA, USA.

Anja Rode (A)

F. Hoffman-La Roche AG, Basel, Switzerland.

Martin Reck (M)

LungenClinic Grosshansdorf, Airway Research Center North, German Center for Lung Research, Grosshansdorf, Germany.

Namrata S Patil (NS)

Genentech Inc., South San Francisco, CA, USA.

Mark Lee (M)

Genentech Inc., South San Francisco, CA, USA.

David S Shames (DS)

Genentech Inc., South San Francisco, CA, USA. shames.david@gene.com.

Katja Schulze (K)

Genentech Inc., South San Francisco, CA, USA. schulze.katja.ks2@gene.com.

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