Serum thymidine kinase activity in patients with HR-positive/HER2-negative advanced breast cancer treated with ribociclib plus letrozole: Results from the prospective BioItaLEE trial.


Journal

European journal of cancer (Oxford, England : 1990)
ISSN: 1879-0852
Titre abrégé: Eur J Cancer
Pays: England
ID NLM: 9005373

Informations de publication

Date de publication:
06 2023
Historique:
received: 02 12 2022
revised: 28 02 2023
accepted: 01 03 2023
medline: 19 5 2023
pubmed: 2 4 2023
entrez: 1 4 2023
Statut: ppublish

Résumé

Thymidine kinase 1 (TK1) is an enzyme downstream of the CDK4/6 pathway, with a critical role in DNA synthesis; serum TK1 activity (sTKa) is a novel liquid biopsy biomarker of tumour cell proliferation. The phase IIIb, BioItaLEE trial (NCT03439046) collected sera from postmenopausal patients with hormone receptor-positive (HR+), HER2-negative (HER2-) advanced breast cancer (ABC) treated with first-line ribociclib plus letrozole at baseline, day 15 of cycle 1 (C1D15), day 1 of cycle 2 (C2D1), and at first imaging. Associations between sTKa assessed at different time points or sTKa dynamic patterns, and progression-free survival (PFS) were evaluated using multivariate Cox models. Overall, 287 patients were enroled. Median follow-up was 26.9 months. High sTKa (>median) at baseline was associated with higher risk of progression (hazard ratio [HR], 2.21; 95% confidence interval [95% CI], 1.45, 3.37; P = 0.0002); similar results were observed for patients with high sTKa levels at C1D15 and C2D1. Early sTKa dynamic patterns were strongly predictive of PFS. The pattern with high sTKa levels at C2D1 following initial decrease at C1D15 was associated with higher risk of progression versus the pattern with low sTKa levels at both time points (HR, 2.89; 95% CI, 1.57, 5.31; P = 0.0006), while the pattern with high sTKa levels at C1D15 was associated with the shortest PFS (HR, 5.65; CI: 2.84, 11.2; P < 0.0001). Baseline and dynamic sTKa changes provided independent information. sTKa appears to be a new promising prognostic and pharmacodynamic biomarker in patients with HR+/HER2- ABC treated with ribociclib plus letrozole as first-line therapy.

Sections du résumé

BACKGROUND
Thymidine kinase 1 (TK1) is an enzyme downstream of the CDK4/6 pathway, with a critical role in DNA synthesis; serum TK1 activity (sTKa) is a novel liquid biopsy biomarker of tumour cell proliferation.
METHODS
The phase IIIb, BioItaLEE trial (NCT03439046) collected sera from postmenopausal patients with hormone receptor-positive (HR+), HER2-negative (HER2-) advanced breast cancer (ABC) treated with first-line ribociclib plus letrozole at baseline, day 15 of cycle 1 (C1D15), day 1 of cycle 2 (C2D1), and at first imaging. Associations between sTKa assessed at different time points or sTKa dynamic patterns, and progression-free survival (PFS) were evaluated using multivariate Cox models.
RESULTS
Overall, 287 patients were enroled. Median follow-up was 26.9 months. High sTKa (>median) at baseline was associated with higher risk of progression (hazard ratio [HR], 2.21; 95% confidence interval [95% CI], 1.45, 3.37; P = 0.0002); similar results were observed for patients with high sTKa levels at C1D15 and C2D1. Early sTKa dynamic patterns were strongly predictive of PFS. The pattern with high sTKa levels at C2D1 following initial decrease at C1D15 was associated with higher risk of progression versus the pattern with low sTKa levels at both time points (HR, 2.89; 95% CI, 1.57, 5.31; P = 0.0006), while the pattern with high sTKa levels at C1D15 was associated with the shortest PFS (HR, 5.65; CI: 2.84, 11.2; P < 0.0001). Baseline and dynamic sTKa changes provided independent information.
CONCLUSIONS
sTKa appears to be a new promising prognostic and pharmacodynamic biomarker in patients with HR+/HER2- ABC treated with ribociclib plus letrozole as first-line therapy.

Identifiants

pubmed: 37003098
pii: S0959-8049(23)00119-3
doi: 10.1016/j.ejca.2023.03.001
pii:
doi:

Substances chimiques

Letrozole 7LKK855W8I
ribociclib TK8ERE8P56
Thymidine Kinase EC 2.7.1.21
Aminopyridines 0
Aromatase Inhibitors 0
Biomarkers 0
Receptor, ErbB-2 EC 2.7.10.1

Types de publication

Clinical Trial, Phase III Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1-11

Informations de copyright

Copyright © 2023 The Authors. Published by Elsevier Ltd.. All rights reserved.

Auteurs

Luca Malorni (L)

Department of Oncology and Translational Research Unit "Sandro Pitigliani", Ospedale di Prato, Azienda USL Toscana Centro, Italy. Electronic address: Luca.malorni@uslcentro.toscana.it.

Giampaolo Bianchini (G)

Department of Oncology, Ospedale San Raffaele, Milano, Italy.

Roberta Caputo (R)

Department of Breast and Thoracic Oncology, IRCCS Istituto Nazionale dei Tumori Fondazione G Pascale, Napoli, Italy.

Alberto Zambelli (A)

Medical Oncology Unit, IRCCS Humanitas Research Hospital and Department of Biomedical Sciences - Humanitas University, Milano, Italy.

Fabio Puglisi (F)

Department of Medical Oncology, IRCCS, Centro di Riferimento Oncologico,Aviano, Italy; Department of Medicine, University of Udine, Italy.

Giulia V Bianchi (GV)

SC Oncologia Medica 1, Fondazione IRCCS Istituto Nazionale dei Tumori, Milan, Italy.

Lucia Del Mastro (L)

U.O.S.D. Breast Unit, IRCCS Ospedale Policlinico San Martino, Genoa, Italy.

Ida Paris (I)

Department of Woman and Child Sciences, Fondazione Policlinico Universitario Agostino Gemelli, IRCCS, Rome, Italy.

Filippo Montemurro (F)

Candiolo Cancer Institute, FPO-IRCCS, Candiolo, Torino, Italy.

Giacomo Allegrini (G)

U.O.C. Oncologia Medica, Presidio Ospedaliero Livorno, Italy.

Marco Colleoni (M)

Division of Medical Senology, Istituto Europeo di Oncologia (IEO), IRCCS, Milano, Italy.

Stefano Tamberi (S)

U.O. Oncologia, P.O. Ospedale degli Infermi - AUSL, Ravenna, Italy.

Claudio Zamagni (C)

IRCCS Azienda ospedaliero-universitaria di Bologna, Bologna, Italy.

Marina E Cazzaniga (ME)

Phase 1 Research Unit & Oncology Unit, Azienda Socio Sanitaria Territoriale Monza & Milano Bicocca School of Medicine and Surgery, Monza, Italy.

Michele Orditura (M)

U.O.C. Oncologia Medica e Ematologia, A.O.U. Università degli Studi L. Vanvitelli, Napoli, Italy.

Valentina Guarneri (V)

Deparment of Surgery, Oncology and Gastroenterology, University of Padova, Italy; Oncologia 2, Istituto Oncologico Veneto (IOV) IRCCS, Padova, Italy.

Daniela Castelletti (D)

Oncology, Novartis Farma SpA, Origgio, Italy.

Matteo Benelli (M)

Department of Oncology and Bioinformatics Unit, Ospedale di Prato, Azienda USL Toscana Centro, Italy.

Mariacristina Di Marino (M)

Oncology, Novartis Farma SpA, Origgio, Italy.

Grazia Arpino (G)

Department of Medical Clinics and Surgery, Università Federico II, Napoli, Italy.

Michelino De Laurentiis (M)

Department of Breast and Thoracic Oncology, IRCCS Istituto Nazionale dei Tumori Fondazione G Pascale, Napoli, Italy.

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