Autoimmune glial fibrillary acidic protein astrocytopathy associated with breast cancer: a case report.


Journal

BMC neurology
ISSN: 1471-2377
Titre abrégé: BMC Neurol
Pays: England
ID NLM: 100968555

Informations de publication

Date de publication:
04 Apr 2023
Historique:
received: 10 12 2022
accepted: 30 03 2023
medline: 6 4 2023
entrez: 4 4 2023
pubmed: 5 4 2023
Statut: epublish

Résumé

Autoimmune glial fibrillary acidic protein (GFAP) astrocytopathy (GFAP-A) is an autoimmune inflammatory central nervous system disorder characterized by the detection of autoantibodies that recognize GFAP in CSF. The pathogenesis of GFAP-A is poorly understood. Some patients had a neoplasm detected and GFAP expressed by neoplasms is plausible as immunogen triggering paraneoplastic neurological autoimmunity. We report a case of 76-year-old female patient with GFAP-A complicated with breast cancer. She presented with altered consciousness, nuchal rigidity, speech disturbances, and weakness. Her clinical symptoms were improved by immunotherapy and cancer treatments. Immunohistochemical analysis showed that the restricted tumor expressed GFAP. The infiltration of CD3 + T cells were observed in the peritumoral and intratumoral areas. The most common infiltrating lymphocytes were CD8 + T cells. CD4 + T cells and CD20 + B cells were also observed in the predominant peritumoral area. These results suggest that GFAP-A may occur in a paraneoplastic neurological syndrome associated with breast cancer.

Sections du résumé

BACKGROUND BACKGROUND
Autoimmune glial fibrillary acidic protein (GFAP) astrocytopathy (GFAP-A) is an autoimmune inflammatory central nervous system disorder characterized by the detection of autoantibodies that recognize GFAP in CSF. The pathogenesis of GFAP-A is poorly understood. Some patients had a neoplasm detected and GFAP expressed by neoplasms is plausible as immunogen triggering paraneoplastic neurological autoimmunity.
CASE PRESENTATION METHODS
We report a case of 76-year-old female patient with GFAP-A complicated with breast cancer. She presented with altered consciousness, nuchal rigidity, speech disturbances, and weakness. Her clinical symptoms were improved by immunotherapy and cancer treatments. Immunohistochemical analysis showed that the restricted tumor expressed GFAP. The infiltration of CD3 + T cells were observed in the peritumoral and intratumoral areas. The most common infiltrating lymphocytes were CD8 + T cells. CD4 + T cells and CD20 + B cells were also observed in the predominant peritumoral area.
CONCLUSIONS CONCLUSIONS
These results suggest that GFAP-A may occur in a paraneoplastic neurological syndrome associated with breast cancer.

Identifiants

pubmed: 37016352
doi: 10.1186/s12883-023-03194-7
pii: 10.1186/s12883-023-03194-7
pmc: PMC10071775
doi:

Substances chimiques

Glial Fibrillary Acidic Protein 0
Autoantibodies 0

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

145

Subventions

Organisme : JSPS KAKENHI
ID : JP21K07457

Informations de copyright

© 2023. The Author(s).

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Auteurs

Tomonori Yaguchi (T)

Department of Neurology, Gifu Prefectural General Medical Center, Gifu, Japan.

Akio Kimura (A)

Department of Neurology, Gifu University Graduate School of Medicine, 1-1 Yanagido, Gifu, 501-1194, Japan.

Akira Takekoshi (A)

Department of Neurology, Gifu University Graduate School of Medicine, 1-1 Yanagido, Gifu, 501-1194, Japan.

Mikiko Matsuo (M)

Department of Tumor Pathology, Gifu University Graduate School of Medicine, Gifu, Japan.

Hiroyuki Tomita (H)

Department of Tumor Pathology, Gifu University Graduate School of Medicine, Gifu, Japan.

Takayoshi Shimohata (T)

Department of Neurology, Gifu University Graduate School of Medicine, 1-1 Yanagido, Gifu, 501-1194, Japan. shimohata@gmail.com.

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Classifications MeSH