VSSP-activated macrophages mediate senescence and tumor inhibition in a preclinical model of advanced prostate cancer.


Journal

Cell communication and signaling : CCS
ISSN: 1478-811X
Titre abrégé: Cell Commun Signal
Pays: England
ID NLM: 101170464

Informations de publication

Date de publication:
13 04 2023
Historique:
received: 18 11 2022
accepted: 07 03 2023
medline: 17 4 2023
entrez: 13 4 2023
pubmed: 14 4 2023
Statut: epublish

Résumé

Androgen deprivation therapy (ADT) is a standard therapy for prostate cancer (PCa). Though disseminated disease is initially sensitive to ADT, an important fraction of the patients progresses to castration-resistant prostate cancer (CRPC). For this reason, the identification of novel effective therapies for treating CRPC is needed. Immunotherapeutic strategies focused on macrophages as antitumor effectors, directly enhancing their tumoricidal potential at the tumor microenvironment or their adoptive transfer after ex vivo activation, have arisen as promising therapies in several cancer types. Despite several approaches centered on the activation of tumor-associated macrophages (TAMs) in PCa are under investigation, to date there is no evidence of clinical benefit in patients. In addition, the evidence of the effectiveness of macrophage adoptive transfer on PCa is poor. Here we find that VSSP, an immunomodulator of the myeloid system, decreases TAMs and inhibits prostatic tumor growth when administered to castrated Pten-deficient prostate tumor-bearing mice. In mice bearing castration-resistant Pten

Identifiants

pubmed: 37055829
doi: 10.1186/s12964-023-01095-3
pii: 10.1186/s12964-023-01095-3
pmc: PMC10100133
doi:

Substances chimiques

Androgen Antagonists 0

Types de publication

Video-Audio Media Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

76

Informations de copyright

© 2023. The Author(s).

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Auteurs

Rydell Alvarez-Arzola (R)

Department of Immunoregulation, Immunology and Immunotherapy Direction, Center of Molecular Immunology, Havana, Cuba. rydell@cim.sld.cu.

Nicoló Bancaro (N)

Department of Molecular Oncology, Institute of Oncology Research (IOR), 6500, Bellinzona, Switzerland.

Ping Lai (P)

Department of Molecular Oncology, Institute of Oncology Research (IOR), 6500, Bellinzona, Switzerland.

Giuseppe Attanasio (G)

Department of Molecular Oncology, Institute of Oncology Research (IOR), 6500, Bellinzona, Switzerland.

Laura Pellegrini (L)

Department of Molecular Oncology, Institute of Oncology Research (IOR), 6500, Bellinzona, Switzerland.

Martina Troiani (M)

Department of Molecular Oncology, Institute of Oncology Research (IOR), 6500, Bellinzona, Switzerland.

Manuel Colucci (M)

Department of Molecular Oncology, Institute of Oncology Research (IOR), 6500, Bellinzona, Switzerland.

Simone Mosole (S)

Department of Molecular Oncology, Institute of Oncology Research (IOR), 6500, Bellinzona, Switzerland.

Emiliano Pasquini (E)

Department of Molecular Oncology, Institute of Oncology Research (IOR), 6500, Bellinzona, Switzerland.

Andrea Alimonti (A)

Department of Molecular Oncology, Institute of Oncology Research (IOR), 6500, Bellinzona, Switzerland.
Faculty of Medicine, Università della Svizzera Italiana, 1011, Lugano, Switzerland.
Department of Medicine, University of Padua, 35131, Padua, Italy.
Medical Oncology, Oncology Institute of Southern Switzerland, 6500, Bellinzona, Switzerland.

Circe Mesa (C)

Innovative Immunotherapy Alliance S.A., Mariel, Artemisa, Cuba.

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Classifications MeSH