Staphylococcal Sensitization: A Correlate of Type 2-High Inflammation in Children With Severe Asthma.


Journal

The journal of allergy and clinical immunology. In practice
ISSN: 2213-2201
Titre abrégé: J Allergy Clin Immunol Pract
Pays: United States
ID NLM: 101597220

Informations de publication

Date de publication:
02 2023
Historique:
received: 25 05 2022
revised: 14 10 2022
accepted: 20 10 2022
medline: 1 5 2023
pubmed: 28 4 2023
entrez: 28 4 2023
Statut: ppublish

Résumé

Sensitization to Staphylococcus aureus enterotoxin (SE) has been identified to be a risk factor for asthma, but its determinants remain unclear. To determine the significance of SE sensitization in children with moderate to severe asthma. This was an observational cross-sectional analysis performed from 2011 to 2015 including children from the prospective Severe Asthma Molecular Phenotype cohort: school-age children with severe and moderate asthma or preschool-age children with severe and moderate recurrent wheeze. We evaluated sensitization to four SEs (Staphylococcus enterotoxin A, Staphylococcus enterotoxin B, Staphylococcus enterotoxin C, and toxic shock staphylococcic toxin). We analyzed data from 377 children: 233 of preschool age and 144 of school age. Among them, 26 (11.2%) and 59 (41.0%) children, respectively, had sensitization to at least one SE. The burden of sensitization was higher in older children in terms of both specific IgE levels and the number of sensitizations. In multivariable analysis, SE sensitization was associated with elevated total IgE in both populations (odds ratio [OR] = 9.35, P = .01; and OR = 8.06, P < .01), and with bronchoalveolar lavage eosinophilia in both preschool and school-age children (OR = 3.95, P = .03; and OR = 4.11, P = .03, respectively). Classification and regression trees showed an association of SE sensitization with age and with total IgE in the entire population, and with total IgE, bronchoalveolar lavage eosinophilia, and blood eosinophilia in school-age children. Staphylococcal enterotoxin sensitization was correlated with type 2-high inflammation (eosinophilic inflammation and elevated total IgE count) in this population of moderate to severe asthmatic children.

Sections du résumé

BACKGROUND
Sensitization to Staphylococcus aureus enterotoxin (SE) has been identified to be a risk factor for asthma, but its determinants remain unclear.
OBJECTIVE
To determine the significance of SE sensitization in children with moderate to severe asthma.
METHODS
This was an observational cross-sectional analysis performed from 2011 to 2015 including children from the prospective Severe Asthma Molecular Phenotype cohort: school-age children with severe and moderate asthma or preschool-age children with severe and moderate recurrent wheeze. We evaluated sensitization to four SEs (Staphylococcus enterotoxin A, Staphylococcus enterotoxin B, Staphylococcus enterotoxin C, and toxic shock staphylococcic toxin).
RESULTS
We analyzed data from 377 children: 233 of preschool age and 144 of school age. Among them, 26 (11.2%) and 59 (41.0%) children, respectively, had sensitization to at least one SE. The burden of sensitization was higher in older children in terms of both specific IgE levels and the number of sensitizations. In multivariable analysis, SE sensitization was associated with elevated total IgE in both populations (odds ratio [OR] = 9.35, P = .01; and OR = 8.06, P < .01), and with bronchoalveolar lavage eosinophilia in both preschool and school-age children (OR = 3.95, P = .03; and OR = 4.11, P = .03, respectively). Classification and regression trees showed an association of SE sensitization with age and with total IgE in the entire population, and with total IgE, bronchoalveolar lavage eosinophilia, and blood eosinophilia in school-age children.
CONCLUSIONS
Staphylococcal enterotoxin sensitization was correlated with type 2-high inflammation (eosinophilic inflammation and elevated total IgE count) in this population of moderate to severe asthmatic children.

Identifiants

pubmed: 37113036
pii: S2213-2198(22)01126-6
doi: 10.1016/j.jaip.2022.10.026
pii:
doi:

Substances chimiques

Immunoglobulin E 37341-29-0
Enterotoxins 0

Types de publication

Observational Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

564-571.e1

Informations de copyright

Copyright © 2022 American Academy of Allergy, Asthma & Immunology. Published by Elsevier Inc. All rights reserved.

Auteurs

Mélisande Bourgoin-Heck (M)

Allergology Department, Hospital A. Trousseau, Sorbonne Université, Assistance Publique-Hôpitaux de Paris, Paris, France; CRESS, Inserm, INRAE, HERA Team, Université Paris Cité, Paris, France.

Marion Duféal (M)

Allergology Department, Hospital A. Trousseau, Sorbonne Université, Assistance Publique-Hôpitaux de Paris, Paris, France.

Sarah Saf (S)

Allergology Department, Hospital A. Trousseau, Sorbonne Université, Assistance Publique-Hôpitaux de Paris, Paris, France.

Fanny Rancière (F)

CRESS, Inserm, INRAE, HERA Team, Université Paris Cité, Paris, France; Faculté de Pharmacie de Paris, Université Paris Cité, Paris, France.

Yannick Chantran (Y)

CRESS, Inserm, INRAE, HERA Team, Université Paris Cité, Paris, France; PharmD.Immunology Department, Biological Allergology Unit, Hôpital A. Trousseau, Sorbonne Université, Assistance Publique-Hôpitaux de Paris, Paris, France.

Isabelle Momas (I)

CRESS, Inserm, INRAE, HERA Team, Université Paris Cité, Paris, France; Faculté de Pharmacie de Paris, Université Paris Cité, Paris, France.

Stéphanie Wanin (S)

Allergology Department, Hospital A. Trousseau, Sorbonne Université, Assistance Publique-Hôpitaux de Paris, Paris, France; CRESS, Inserm, INRAE, HERA Team, Université Paris Cité, Paris, France.

Philippe Saint-Pierre (P)

Institut de Mathématiques de Toulouse, UMR 5219, Université de Toulouse, CNRS, Toulouse, France.

Jocelyne Just (J)

Allergology Department, Hospital A. Trousseau, Sorbonne Université, Assistance Publique-Hôpitaux de Paris, Paris, France; CRESS, Inserm, INRAE, HERA Team, Université Paris Cité, Paris, France; Unité d'Allergologie, Hôpital Américain de Paris, Neuilly sur Seine, France. Electronic address: jocelyne.just@ahparis.org.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH