English version of Japanese guidance for the use of oral Janus kinase inhibitors (JAK1 and TYK2 inhibitors) in the treatments of psoriasis.


Journal

The Journal of dermatology
ISSN: 1346-8138
Titre abrégé: J Dermatol
Pays: England
ID NLM: 7600545

Informations de publication

Date de publication:
May 2023
Historique:
revised: 08 03 2023
received: 01 03 2023
accepted: 12 03 2023
medline: 4 5 2023
pubmed: 3 5 2023
entrez: 3 5 2023
Statut: ppublish

Résumé

This is the English version of Japanese guidance for the use of oral Janus kinase (JAK) inhibitors (JAK1 and tyrosine kinase 2 [TYK2] inhibitors) in the treatments of psoriasis. Several cytokines, such as interleukin (IL)-6, IL-7, IL-12, IL-21, IL-22, IL-23, interferon (IFN)-α, and IFN-γ, are involved in the pathogenesis of psoriasis (including psoriatic arthritis). As oral JAK inhibitors hinder the JAK-signal transducers and activators of transcription signal transduction routes involved in the signal transduction of these cytokines, they may be effective for the treatment of psoriasis. JAK has four types: JAK1, JAK2, JAK3, and TYK2. Regarding the use of oral JAK inhibitors for the treatment of psoriasis in Japan, indications of the JAK1 inhibitor upadacitinib were extended also to psoriatic arthritis in 2021, and the use of the TYK2 inhibitor deucravacitinib for plaque-type psoriasis, pustular psoriasis, and erythrodermic psoriasis became covered by health insurance in 2022. This guidance was developed for board-certified dermatologists who specialize in the treatment of psoriasis and to promote the proper use of oral JAK inhibitors. In the package inserts and guides for appropriate use, upadacitinib and deucravacitinib are classified as a "JAK inhibitor" and a "TYK2 inhibitor", respectively, and it is possible that there may be differences in safety between the two drugs. The safety of these drugs will be evaluated for the future by the postmarketing surveillance for molecularly targeted drugs for psoriasis of the Japanese Dermatological Association.

Identifiants

pubmed: 37132187
doi: 10.1111/1346-8138.16797
doi:

Substances chimiques

Cytokines 0
Interleukin-6 0
JAK1 protein, human EC 2.7.10.2
Janus Kinase 1 EC 2.7.10.2
Janus Kinase 2 EC 2.7.10.2
Janus Kinase Inhibitors 0
TYK2 Kinase EC 2.7.10.2
TYK2 protein, human EC 2.7.10.2

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e138-e150

Informations de copyright

© 2023 Japanese Dermatological Association.

Références

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Auteurs

Hidehisa Saeki (H)

Department of Dermatology, Nippon Medical School, Tokyo, Japan.

Tomotaka Mabuchi (T)

Department of Dermatology, Tokai University School of Medicine, Isehara, Japan.

Akihiko Asahina (A)

Department of Dermatology, The Jikei University School of Medicine, Tokyo, Japan.

Masatoshi Abe (M)

Kojinkai Sapporo Skin Clinic, Sapporo, Japan.

Atsuyuki Igarashi (A)

Department of Dermatology, NTT Medical Center Tokyo, Tokyo, Japan.

Shinichi Imafuku (S)

Department of Dermatology, Faculty of Medicine, Fukuoka University, Fukuoka, Japan.

Yukari Okubo (Y)

Department of Dermatology, Tokyo Medical University, Tokyo, Japan.

Mayumi Komine (M)

Department of Dermatology, Jichi Medical University, Shimotsuke, Japan.

Kenzo Takahashi (K)

Department of Dermatology, University of the Ryukyus Graduate School of Medicine, Okinawa, Japan.

Hideshi Torii (H)

Division of Dermatology, Tokyo Yamate Medical Center, Tokyo, Japan.

Akimichi Morita (A)

Department of Geriatric and Environmental Dermatology, Nagoya City University Graduate School of Medical Sciences, Nagoya, Japan.

Hiroshi Yotsuyanagi (H)

Division of Infectious Diseases, Advanced Clinical Research Center, Institute of Medical Science, The University of Tokyo, Tokyo, Japan.

Akira Watanabe (A)

Research Division for Development of Anti-Infective Agents, Faculty of Medical Science and Welfare, Tohoku Bunka Gakuen University, Sendai, Japan.

Mamitaro Ohtsuki (M)

Department of Dermatology, Jichi Medical University, Shimotsuke, Japan.

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